Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease

Summary: Brain waste clearance from the interstitial fluid environment is challenging to measure, which has contributed to controversy regarding the significance of glymphatic transport impairment for neurodegenerative processes. Dynamic contrast enhanced MRI (DCE-MRI) with cerebrospinal fluid admin...

Full description

Saved in:
Bibliographic Details
Main Authors: Sunil Koundal, Xinan Chen, Zachary Gursky, Hedok Lee, Kaiming Xu, Feng Liang, Zhongcong Xie, Feng Xu, Hung-Mo Lin, William E. Van Nostrand, Xianfeng Gu, Rena Elkin, Allen Tannenbaum, Helene Benveniste
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:iScience
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S2589004224026907
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850121557659090944
author Sunil Koundal
Xinan Chen
Zachary Gursky
Hedok Lee
Kaiming Xu
Feng Liang
Zhongcong Xie
Feng Xu
Hung-Mo Lin
William E. Van Nostrand
Xianfeng Gu
Rena Elkin
Allen Tannenbaum
Helene Benveniste
author_facet Sunil Koundal
Xinan Chen
Zachary Gursky
Hedok Lee
Kaiming Xu
Feng Liang
Zhongcong Xie
Feng Xu
Hung-Mo Lin
William E. Van Nostrand
Xianfeng Gu
Rena Elkin
Allen Tannenbaum
Helene Benveniste
author_sort Sunil Koundal
collection DOAJ
description Summary: Brain waste clearance from the interstitial fluid environment is challenging to measure, which has contributed to controversy regarding the significance of glymphatic transport impairment for neurodegenerative processes. Dynamic contrast enhanced MRI (DCE-MRI) with cerebrospinal fluid administration of Gd-tagged tracers is often used to assess glymphatic system function. We previously quantified glymphatic transport from DCE-MRI data utilizing regularized optimal mass transport (rOMT) analysis, however, information specific to glymphatic clearance was not directly derived. To fill this knowledge gap, we here implemented unbalanced rOMT analysis which allows for assessment of both influx and clearance. Dynamic influx/clearance brain maps were derived from rTg-DI rats with cerebral amyloid angiopathy (CAA) and TgSD-AD rats with Alzheimer’s disease (AD). The rTg-DI rats with severe CAA disease exhibited abnormal influx/clearance kinetics, while TgSD-AD rats with a moderate Aβ plaque load exhibited normal transport suggesting that different Aβ lesions and their overall burden differentially impact glymphatic system function.
format Article
id doaj-art-4feca50205da422f86d5f2fe7ab9b078
institution OA Journals
issn 2589-0042
language English
publishDate 2024-12-01
publisher Elsevier
record_format Article
series iScience
spelling doaj-art-4feca50205da422f86d5f2fe7ab9b0782025-08-20T02:35:04ZengElsevieriScience2589-00422024-12-01271211146310.1016/j.isci.2024.111463Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s diseaseSunil Koundal0Xinan Chen1Zachary Gursky2Hedok Lee3Kaiming Xu4Feng Liang5Zhongcong Xie6Feng Xu7Hung-Mo Lin8William E. Van Nostrand9Xianfeng Gu10Rena Elkin11Allen Tannenbaum12Helene Benveniste13Department of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USADepartment of Medical Physics, Memorial Sloan Kettering Cancer Center, New York City, NY 10065, USADepartment of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USADepartment of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USADepartment of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USA; Department of Applied Mathematics & Statistics, Stony Brook University, Stony Brook, NY 11794, USADepartment of Anesthesia, Critical Care and Pain Medicine, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02114, USADepartment of Anesthesia, Critical Care and Pain Medicine, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02114, USAGeorge and Anne Ryan Institute for Neuroscience and the Department of Biomedical and Pharmaceutical Sciences, University of Rhode Island, Kingston, RI 02906, USADepartment of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USAGeorge and Anne Ryan Institute for Neuroscience and the Department of Biomedical and Pharmaceutical Sciences, University of Rhode Island, Kingston, RI 02906, USADepartment of Applied Mathematics & Statistics, Stony Brook University, Stony Brook, NY 11794, USA; Departments of Computer Science, Stony Brook University, Stony Brook, NY 11794, USADepartment of Medical Physics, Memorial Sloan Kettering Cancer Center, New York City, NY 10065, USADepartment of Applied Mathematics & Statistics, Stony Brook University, Stony Brook, NY 11794, USA; Departments of Computer Science, Stony Brook University, Stony Brook, NY 11794, USADepartment of Anesthesiology, Yale School of Medicine, New Haven, CT 06510, USA; Department of Biomedical Engineering, Yale School of Medicine New Haven, New Haven, CT 06510, USA; Corresponding authorSummary: Brain waste clearance from the interstitial fluid environment is challenging to measure, which has contributed to controversy regarding the significance of glymphatic transport impairment for neurodegenerative processes. Dynamic contrast enhanced MRI (DCE-MRI) with cerebrospinal fluid administration of Gd-tagged tracers is often used to assess glymphatic system function. We previously quantified glymphatic transport from DCE-MRI data utilizing regularized optimal mass transport (rOMT) analysis, however, information specific to glymphatic clearance was not directly derived. To fill this knowledge gap, we here implemented unbalanced rOMT analysis which allows for assessment of both influx and clearance. Dynamic influx/clearance brain maps were derived from rTg-DI rats with cerebral amyloid angiopathy (CAA) and TgSD-AD rats with Alzheimer’s disease (AD). The rTg-DI rats with severe CAA disease exhibited abnormal influx/clearance kinetics, while TgSD-AD rats with a moderate Aβ plaque load exhibited normal transport suggesting that different Aβ lesions and their overall burden differentially impact glymphatic system function.http://www.sciencedirect.com/science/article/pii/S2589004224026907Biological sciencesNeuroscience
spellingShingle Sunil Koundal
Xinan Chen
Zachary Gursky
Hedok Lee
Kaiming Xu
Feng Liang
Zhongcong Xie
Feng Xu
Hung-Mo Lin
William E. Van Nostrand
Xianfeng Gu
Rena Elkin
Allen Tannenbaum
Helene Benveniste
Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
iScience
Biological sciences
Neuroscience
title Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
title_full Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
title_fullStr Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
title_full_unstemmed Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
title_short Divergent brain solute clearance in rat models of cerebral amyloid angiopathy and Alzheimer’s disease
title_sort divergent brain solute clearance in rat models of cerebral amyloid angiopathy and alzheimer s disease
topic Biological sciences
Neuroscience
url http://www.sciencedirect.com/science/article/pii/S2589004224026907
work_keys_str_mv AT sunilkoundal divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT xinanchen divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT zacharygursky divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT hedoklee divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT kaimingxu divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT fengliang divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT zhongcongxie divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT fengxu divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT hungmolin divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT williamevannostrand divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT xianfenggu divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT renaelkin divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT allentannenbaum divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease
AT helenebenveniste divergentbrainsoluteclearanceinratmodelsofcerebralamyloidangiopathyandalzheimersdisease