Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study

Abstract Background As neuropeptide Y is associated with endothelial dysfunction, this study explored the relationship between neuropeptide Y and acute myocardial infarction. Methods We included 128 acute myocardial infarction cases and 62 controls. Using the SYNTAX scoring system, the acute myocard...

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Main Authors: Yan-li Zheng, Hui-li Lin, Yue-ting Li, Mei-mei Li, Jing-ru Du, Wan-da Wang, Yao-guo Wang, Yin-lian Cai
Format: Article
Language:English
Published: BMC 2024-11-01
Series:BMC Cardiovascular Disorders
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Online Access:https://doi.org/10.1186/s12872-024-04373-1
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author Yan-li Zheng
Hui-li Lin
Yue-ting Li
Mei-mei Li
Jing-ru Du
Wan-da Wang
Yao-guo Wang
Yin-lian Cai
author_facet Yan-li Zheng
Hui-li Lin
Yue-ting Li
Mei-mei Li
Jing-ru Du
Wan-da Wang
Yao-guo Wang
Yin-lian Cai
author_sort Yan-li Zheng
collection DOAJ
description Abstract Background As neuropeptide Y is associated with endothelial dysfunction, this study explored the relationship between neuropeptide Y and acute myocardial infarction. Methods We included 128 acute myocardial infarction cases and 62 controls. Using the SYNTAX scoring system, the acute myocardial infarction group was sub-grouped into “SYNTAX ≤ 22,” “SYNTAX = 23–32,” and “SYNTAX ≥ 33.” Plasma neuropeptide Y, endothelin, endothelial nitric oxide synthase, and thromboxane A2 levels were measured. Results The acute myocardial infarction group had higher plasma neuropeptide Y, endothelin, and thromboxane A2 levels than controls ([58.76 ± 17.63 vs. 37.48 ± 11.36 ng/ml, P = 0.000], [36.16 ± 10.04 vs. 27.80 ± 7.18 pg/ml, P = 0.000], and [27.69 ± 6.91 vs. 24.32 ± 7.28 pg/ml, P = 0.002], respectively). The acute myocardial infarction group also had lower plasma endothelial nitric oxide synthase levels than controls (3.00 ± 0.94 vs. 4.05 ± 1.44 ng/ml, P = 0.000). Additionally, the receiver operating characteristic curve analysis showed that a neuropeptide Y value of 49.94 ng/ml could help diagnose acute myocardial infarction (sensitivity: 70.9%; specificity: 91.9%). The SYNTAX scores, smoking, plasma endothelin, thromboxane A2, and neuropeptide Y levels were positively correlated, whereas plasma endothelial nitric oxide synthase and neuropeptide Y levels were negatively correlated. Lastly, plasma neuropeptide Y levels were different among subgroups (P < 0.05); patients with higher SYNTAX scores had higher neuropeptide Y levels. Conclusions The levels of plasma NPY may be accociated with the AMI process.
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spelling doaj-art-4fb093fced4a4eae93eba7f3f35986932025-08-20T02:51:45ZengBMCBMC Cardiovascular Disorders1471-22612024-11-012411910.1186/s12872-024-04373-1Role of plasma neuropeptide Y in acute myocardial infarction: a case-control studyYan-li Zheng0Hui-li Lin1Yue-ting Li2Mei-mei Li3Jing-ru Du4Wan-da Wang5Yao-guo Wang6Yin-lian Cai7Department of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical University Department of Nephrology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Fujian Medical UniversityAbstract Background As neuropeptide Y is associated with endothelial dysfunction, this study explored the relationship between neuropeptide Y and acute myocardial infarction. Methods We included 128 acute myocardial infarction cases and 62 controls. Using the SYNTAX scoring system, the acute myocardial infarction group was sub-grouped into “SYNTAX ≤ 22,” “SYNTAX = 23–32,” and “SYNTAX ≥ 33.” Plasma neuropeptide Y, endothelin, endothelial nitric oxide synthase, and thromboxane A2 levels were measured. Results The acute myocardial infarction group had higher plasma neuropeptide Y, endothelin, and thromboxane A2 levels than controls ([58.76 ± 17.63 vs. 37.48 ± 11.36 ng/ml, P = 0.000], [36.16 ± 10.04 vs. 27.80 ± 7.18 pg/ml, P = 0.000], and [27.69 ± 6.91 vs. 24.32 ± 7.28 pg/ml, P = 0.002], respectively). The acute myocardial infarction group also had lower plasma endothelial nitric oxide synthase levels than controls (3.00 ± 0.94 vs. 4.05 ± 1.44 ng/ml, P = 0.000). Additionally, the receiver operating characteristic curve analysis showed that a neuropeptide Y value of 49.94 ng/ml could help diagnose acute myocardial infarction (sensitivity: 70.9%; specificity: 91.9%). The SYNTAX scores, smoking, plasma endothelin, thromboxane A2, and neuropeptide Y levels were positively correlated, whereas plasma endothelial nitric oxide synthase and neuropeptide Y levels were negatively correlated. Lastly, plasma neuropeptide Y levels were different among subgroups (P < 0.05); patients with higher SYNTAX scores had higher neuropeptide Y levels. Conclusions The levels of plasma NPY may be accociated with the AMI process.https://doi.org/10.1186/s12872-024-04373-1Acute myocardial infarctionEndothelial functionNeuropeptide Y
spellingShingle Yan-li Zheng
Hui-li Lin
Yue-ting Li
Mei-mei Li
Jing-ru Du
Wan-da Wang
Yao-guo Wang
Yin-lian Cai
Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
BMC Cardiovascular Disorders
Acute myocardial infarction
Endothelial function
Neuropeptide Y
title Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
title_full Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
title_fullStr Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
title_full_unstemmed Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
title_short Role of plasma neuropeptide Y in acute myocardial infarction: a case-control study
title_sort role of plasma neuropeptide y in acute myocardial infarction a case control study
topic Acute myocardial infarction
Endothelial function
Neuropeptide Y
url https://doi.org/10.1186/s12872-024-04373-1
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