Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene

POLD1 (DNA polymerase delta 1, catalytic subunit) is a protein-coding gene that encodes the large catalytic subunit (POLD1/p125) of the DNA polymerase delta (Polδ) complex. The consequence of missense or nonsynonymous SNPs (nsSNPs), which occur in the coding region of a specific gene, is the replace...

Full description

Saved in:
Bibliographic Details
Main Authors: Md. Nazmul Islam Bappy, Anindita Roy, Md Gulam Rabbany Rabbi, Nusrat Jahan, Fahmida Akther Chowdhury, Syeda Farjana Hoque, Emran Hossain Sajib, Parvez Khan, Ferdaus Mohd Altaf Hossain, Kazi Md. Ali Zinnah
Format: Article
Language:English
Published: Wiley 2022-01-01
Series:Genetics Research
Online Access:http://dx.doi.org/10.1155/2022/1740768
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849405378933030912
author Md. Nazmul Islam Bappy
Anindita Roy
Md Gulam Rabbany Rabbi
Nusrat Jahan
Fahmida Akther Chowdhury
Syeda Farjana Hoque
Emran Hossain Sajib
Parvez Khan
Ferdaus Mohd Altaf Hossain
Kazi Md. Ali Zinnah
author_facet Md. Nazmul Islam Bappy
Anindita Roy
Md Gulam Rabbany Rabbi
Nusrat Jahan
Fahmida Akther Chowdhury
Syeda Farjana Hoque
Emran Hossain Sajib
Parvez Khan
Ferdaus Mohd Altaf Hossain
Kazi Md. Ali Zinnah
author_sort Md. Nazmul Islam Bappy
collection DOAJ
description POLD1 (DNA polymerase delta 1, catalytic subunit) is a protein-coding gene that encodes the large catalytic subunit (POLD1/p125) of the DNA polymerase delta (Polδ) complex. The consequence of missense or nonsynonymous SNPs (nsSNPs), which occur in the coding region of a specific gene, is the replacement of single amino acid. It may also change the structure, stability, and/or functions of the protein. Mutation in the POLD1 gene is associated with autosomal dominant predisposition to colonic adenomatous polyps, colon cancer, endometrial cancer (EDMC), breast cancer, and brain tumors. These de novo mutations in the POLD1 gene also result in autosomal dominant MDPL syndrome (mandibular hypoplasia, deafness, progeroid features, and lipodystrophy). In this study, genetic variations of POLD1 which may affect the structure and/or function were analyzed using different types of bioinformatics tools. A total of 17038 nsSNPs for POLD1 were collected from the NCBI database, among which 1317 were missense variants. Out of all missense nsSNPs, 28 were found to be deleterious functionally and structurally. Among these deleterious nsSNPs, 23 showed a conservation scale of >5, 2 were predicted to be associated with binding site formation, and one acted as a posttranslational modification site. All of them were involved in coil, extracellular structures, or helix formation, and some cause the change in size, charge, and hydrophobicity.
format Article
id doaj-art-4e18f22601c648bdbbfa2dbeb6f47ce4
institution Kabale University
issn 1469-5073
language English
publishDate 2022-01-01
publisher Wiley
record_format Article
series Genetics Research
spelling doaj-art-4e18f22601c648bdbbfa2dbeb6f47ce42025-08-20T03:36:41ZengWileyGenetics Research1469-50732022-01-01202210.1155/2022/1740768Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 GeneMd. Nazmul Islam Bappy0Anindita Roy1Md Gulam Rabbany Rabbi2Nusrat Jahan3Fahmida Akther Chowdhury4Syeda Farjana Hoque5Emran Hossain Sajib6Parvez Khan7Ferdaus Mohd Altaf Hossain8Kazi Md. Ali Zinnah9Faculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringFaculty of Biotechnology and Genetic EngineeringDept. of Biochemistry & Molecular BiologyFaculty of VeterinaryFaculty of Biotechnology and Genetic EngineeringPOLD1 (DNA polymerase delta 1, catalytic subunit) is a protein-coding gene that encodes the large catalytic subunit (POLD1/p125) of the DNA polymerase delta (Polδ) complex. The consequence of missense or nonsynonymous SNPs (nsSNPs), which occur in the coding region of a specific gene, is the replacement of single amino acid. It may also change the structure, stability, and/or functions of the protein. Mutation in the POLD1 gene is associated with autosomal dominant predisposition to colonic adenomatous polyps, colon cancer, endometrial cancer (EDMC), breast cancer, and brain tumors. These de novo mutations in the POLD1 gene also result in autosomal dominant MDPL syndrome (mandibular hypoplasia, deafness, progeroid features, and lipodystrophy). In this study, genetic variations of POLD1 which may affect the structure and/or function were analyzed using different types of bioinformatics tools. A total of 17038 nsSNPs for POLD1 were collected from the NCBI database, among which 1317 were missense variants. Out of all missense nsSNPs, 28 were found to be deleterious functionally and structurally. Among these deleterious nsSNPs, 23 showed a conservation scale of >5, 2 were predicted to be associated with binding site formation, and one acted as a posttranslational modification site. All of them were involved in coil, extracellular structures, or helix formation, and some cause the change in size, charge, and hydrophobicity.http://dx.doi.org/10.1155/2022/1740768
spellingShingle Md. Nazmul Islam Bappy
Anindita Roy
Md Gulam Rabbany Rabbi
Nusrat Jahan
Fahmida Akther Chowdhury
Syeda Farjana Hoque
Emran Hossain Sajib
Parvez Khan
Ferdaus Mohd Altaf Hossain
Kazi Md. Ali Zinnah
Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
Genetics Research
title Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
title_full Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
title_fullStr Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
title_full_unstemmed Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
title_short Scrutinizing Deleterious Nonsynonymous SNPs and Their Effect on Human POLD1 Gene
title_sort scrutinizing deleterious nonsynonymous snps and their effect on human pold1 gene
url http://dx.doi.org/10.1155/2022/1740768
work_keys_str_mv AT mdnazmulislambappy scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT aninditaroy scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT mdgulamrabbanyrabbi scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT nusratjahan scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT fahmidaaktherchowdhury scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT syedafarjanahoque scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT emranhossainsajib scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT parvezkhan scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT ferdausmohdaltafhossain scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene
AT kazimdalizinnah scrutinizingdeleteriousnonsynonymoussnpsandtheireffectonhumanpold1gene