Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix

The concept of using tissue density as a mechanism to diagnose a tumor has been around for centuries. However, this concept has not been sufficiently explored in a laboratory setting. Therefore, in this paper, we observed the effects of cell density and extracellular matrix (ECM) density on colon ca...

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Main Authors: Ramana Vishnubhotla, Shruthi Bharadwaj, Shan Sun, Vitali Metlushko, Sarah C. Glover
Format: Article
Language:English
Published: Wiley 2012-01-01
Series:International Journal of Cell Biology
Online Access:http://dx.doi.org/10.1155/2012/259142
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author Ramana Vishnubhotla
Shruthi Bharadwaj
Shan Sun
Vitali Metlushko
Sarah C. Glover
author_facet Ramana Vishnubhotla
Shruthi Bharadwaj
Shan Sun
Vitali Metlushko
Sarah C. Glover
author_sort Ramana Vishnubhotla
collection DOAJ
description The concept of using tissue density as a mechanism to diagnose a tumor has been around for centuries. However, this concept has not been sufficiently explored in a laboratory setting. Therefore, in this paper, we observed the effects of cell density and extracellular matrix (ECM) density on colon cancer invasion and proliferation using SW620 cells. We also attempted to inhibit ROCK-I to determine its effect on cell invasion and proliferation using standard molecular biology techniques and advanced imaging. Increasing cell seeding density resulted in a 2-fold increase in cell invasion as well as cell proliferation independent of treatment with Y-27632. Increasing collagen I scaffold density resulted in a 2.5-fold increase in cell proliferation while treatment with Y-27632 attenuated this effect although 1.5 fold increase in cell invasion was observed in ROCK inhibited samples. Intriguingly, ROCK inhibition also resulted in a 3.5-fold increase in cell invasion within 3D collagen scaffolds for cells seeded at lower densities. We show in this paper that ROCK-I inhibition leads to increased invasion within 3D collagen I microenvironments. This data suggests that although ROCK inhibitors have been used clinically to treat several medical conditions, its effect largely depends on the surrounding microenvironment.
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issn 1687-8876
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language English
publishDate 2012-01-01
publisher Wiley
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series International Journal of Cell Biology
spelling doaj-art-4c213da72ad7426f8acf2e9e896b2e2b2025-08-20T02:38:39ZengWileyInternational Journal of Cell Biology1687-88761687-88842012-01-01201210.1155/2012/259142259142Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 MatrixRamana Vishnubhotla0Shruthi Bharadwaj1Shan Sun2Vitali Metlushko3Sarah C. Glover4Department of Bioengineering, University of Illinois at Chicago, Chicago, IL 60612, USADepartment of Medicine, University of Florida, P.O. Box 100214, Gainesville, FL 32610, USADepartment of Bioengineering, University of Illinois at Chicago, Chicago, IL 60612, USADepartment of Electrical and Computer Engineering, University of Illinois at Chicago, Chicago, IL 60607, USADepartment of Medicine, University of Florida, P.O. Box 100214, Gainesville, FL 32610, USAThe concept of using tissue density as a mechanism to diagnose a tumor has been around for centuries. However, this concept has not been sufficiently explored in a laboratory setting. Therefore, in this paper, we observed the effects of cell density and extracellular matrix (ECM) density on colon cancer invasion and proliferation using SW620 cells. We also attempted to inhibit ROCK-I to determine its effect on cell invasion and proliferation using standard molecular biology techniques and advanced imaging. Increasing cell seeding density resulted in a 2-fold increase in cell invasion as well as cell proliferation independent of treatment with Y-27632. Increasing collagen I scaffold density resulted in a 2.5-fold increase in cell proliferation while treatment with Y-27632 attenuated this effect although 1.5 fold increase in cell invasion was observed in ROCK inhibited samples. Intriguingly, ROCK inhibition also resulted in a 3.5-fold increase in cell invasion within 3D collagen scaffolds for cells seeded at lower densities. We show in this paper that ROCK-I inhibition leads to increased invasion within 3D collagen I microenvironments. This data suggests that although ROCK inhibitors have been used clinically to treat several medical conditions, its effect largely depends on the surrounding microenvironment.http://dx.doi.org/10.1155/2012/259142
spellingShingle Ramana Vishnubhotla
Shruthi Bharadwaj
Shan Sun
Vitali Metlushko
Sarah C. Glover
Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
International Journal of Cell Biology
title Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
title_full Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
title_fullStr Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
title_full_unstemmed Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
title_short Treatment with Y-27632, a ROCK Inhibitor, Increases the Proinvasive Nature of SW620 Cells on 3D Collagen Type 1 Matrix
title_sort treatment with y 27632 a rock inhibitor increases the proinvasive nature of sw620 cells on 3d collagen type 1 matrix
url http://dx.doi.org/10.1155/2012/259142
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