The role of IL-17 family cytokines in cardiac fibrosis

Myocardial fibrosis is a common pathological feature in various cardiovascular diseases including myocardial infarction, heart failure, and myocarditis. Generally, persistent myocardial fibrosis correlates with poor prognosis and ranks among the leading causes of death globally. Currently, there is...

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Main Author: Liqing Huang
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-10-01
Series:Frontiers in Cardiovascular Medicine
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fcvm.2024.1470362/full
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author Liqing Huang
author_facet Liqing Huang
author_sort Liqing Huang
collection DOAJ
description Myocardial fibrosis is a common pathological feature in various cardiovascular diseases including myocardial infarction, heart failure, and myocarditis. Generally, persistent myocardial fibrosis correlates with poor prognosis and ranks among the leading causes of death globally. Currently, there is no effective treatment for myocardial fibrosis, partly due to its unclear pathogenic mechanism. Increasing studies have shown IL-17 family cytokines are strongly associated with the initiation and propagation of myocardial fibrosis. This review summarizes the expression, action, and signal transduction mechanisms of IL-17, focusing on its role in fibrosis associated with cardiovascular diseases such as myocardial infarction, heart failure, hypertension, diabetes, and myocarditis. It also discusses its potential as a therapeutic target, offering new insights for the clinical treatment of myocardial fibrosis.
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spelling doaj-art-4b2d0467fbf24cc891f8b34147c23c2c2025-08-20T02:08:46ZengFrontiers Media S.A.Frontiers in Cardiovascular Medicine2297-055X2024-10-011110.3389/fcvm.2024.14703621470362The role of IL-17 family cytokines in cardiac fibrosisLiqing HuangMyocardial fibrosis is a common pathological feature in various cardiovascular diseases including myocardial infarction, heart failure, and myocarditis. Generally, persistent myocardial fibrosis correlates with poor prognosis and ranks among the leading causes of death globally. Currently, there is no effective treatment for myocardial fibrosis, partly due to its unclear pathogenic mechanism. Increasing studies have shown IL-17 family cytokines are strongly associated with the initiation and propagation of myocardial fibrosis. This review summarizes the expression, action, and signal transduction mechanisms of IL-17, focusing on its role in fibrosis associated with cardiovascular diseases such as myocardial infarction, heart failure, hypertension, diabetes, and myocarditis. It also discusses its potential as a therapeutic target, offering new insights for the clinical treatment of myocardial fibrosis.https://www.frontiersin.org/articles/10.3389/fcvm.2024.1470362/fullIL-17cardiac fibrosisfibroblastmyofibroblastextracellular matrix
spellingShingle Liqing Huang
The role of IL-17 family cytokines in cardiac fibrosis
Frontiers in Cardiovascular Medicine
IL-17
cardiac fibrosis
fibroblast
myofibroblast
extracellular matrix
title The role of IL-17 family cytokines in cardiac fibrosis
title_full The role of IL-17 family cytokines in cardiac fibrosis
title_fullStr The role of IL-17 family cytokines in cardiac fibrosis
title_full_unstemmed The role of IL-17 family cytokines in cardiac fibrosis
title_short The role of IL-17 family cytokines in cardiac fibrosis
title_sort role of il 17 family cytokines in cardiac fibrosis
topic IL-17
cardiac fibrosis
fibroblast
myofibroblast
extracellular matrix
url https://www.frontiersin.org/articles/10.3389/fcvm.2024.1470362/full
work_keys_str_mv AT liqinghuang theroleofil17familycytokinesincardiacfibrosis
AT liqinghuang roleofil17familycytokinesincardiacfibrosis