The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice

Autism spectrum disorders (ASD), a group of neurodevelopmental disorders characterized by the core symptoms of impaired social communication and stereotyped behaviors, is strongly associated with dysregulated microbiota-gut-brain axis. Emerging evidence suggests that Faecalibacterium, which showed r...

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Main Authors: You Yu, Yujing Wang, Jie Zhang, Shucheng Li, Yulin Wang, Xin You, Xue Chen, Mengxuan Du, Lisheng Xie, Shuang-Jiang Liu
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-08-01
Series:Frontiers in Microbiology
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Online Access:https://www.frontiersin.org/articles/10.3389/fmicb.2025.1640149/full
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author You Yu
Yujing Wang
Yujing Wang
Jie Zhang
Shucheng Li
Yulin Wang
Xin You
Xue Chen
Mengxuan Du
Lisheng Xie
Shuang-Jiang Liu
Shuang-Jiang Liu
author_facet You Yu
Yujing Wang
Yujing Wang
Jie Zhang
Shucheng Li
Yulin Wang
Xin You
Xue Chen
Mengxuan Du
Lisheng Xie
Shuang-Jiang Liu
Shuang-Jiang Liu
author_sort You Yu
collection DOAJ
description Autism spectrum disorders (ASD), a group of neurodevelopmental disorders characterized by the core symptoms of impaired social communication and stereotyped behaviors, is strongly associated with dysregulated microbiota-gut-brain axis. Emerging evidence suggests that Faecalibacterium, which showed reduced abundance in ASD cohorts, holds therapeutic potential, though its interaction with host remain unexplored. Here, we investigated the efficacy and molecular basis of Faecalibacterium hominis 4P-15 (4P-15) in BTBR T+Itpr3tf/J (BTBR) mice, an idiopathic ASD mouse model. Oral administration of 4P-15 significantly reduced the intestinal levels of indole, indole-3-propionic acid (IPA), and indole-3-acetic acid (IAA), as well as the level of IPA in brain. Furthermore, the decreased levels of IPA in brain contributed to the attenuated aryl hydrocarbon receptor (AhR) signaling characterized by increased expression of downstream elements, including glutamate transporters and GABA receptors. Ultimately, this modulation led to the restoration of excitatory/inhibitory imbalance, a typical pathophysiological feature of ASD, and thereby alleviated ASD core behavioral symptoms. Our findings underscore Faecalibacterium-mediated AhR modulation as a promising therapeutic strategy for ASD, highlighting the dual potential of Faecalibacterium-based probiotics and targeted interventions against indole-AhR signaling to address neurodevelopmental disorders.
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spelling doaj-art-4a1c3e4506cc4d26959af694100c46f92025-08-22T05:26:45ZengFrontiers Media S.A.Frontiers in Microbiology1664-302X2025-08-011610.3389/fmicb.2025.16401491640149The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR miceYou Yu0Yujing Wang1Yujing Wang2Jie Zhang3Shucheng Li4Yulin Wang5Xin You6Xue Chen7Mengxuan Du8Lisheng Xie9Shuang-Jiang Liu10Shuang-Jiang Liu11State Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Biopharmaceutical Preparation and Delivery, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Technology, Shandong University, Qingdao, ChinaState Key Laboratory of Microbial Technology, Shandong University, Qingdao, ChinaDepartment of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medica Sciences & Peking Union Medical College, Beijing, ChinaState Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Technology, Shandong University, Qingdao, ChinaState Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaState Key Laboratory of Microbial Technology, Shandong University, Qingdao, ChinaAutism spectrum disorders (ASD), a group of neurodevelopmental disorders characterized by the core symptoms of impaired social communication and stereotyped behaviors, is strongly associated with dysregulated microbiota-gut-brain axis. Emerging evidence suggests that Faecalibacterium, which showed reduced abundance in ASD cohorts, holds therapeutic potential, though its interaction with host remain unexplored. Here, we investigated the efficacy and molecular basis of Faecalibacterium hominis 4P-15 (4P-15) in BTBR T+Itpr3tf/J (BTBR) mice, an idiopathic ASD mouse model. Oral administration of 4P-15 significantly reduced the intestinal levels of indole, indole-3-propionic acid (IPA), and indole-3-acetic acid (IAA), as well as the level of IPA in brain. Furthermore, the decreased levels of IPA in brain contributed to the attenuated aryl hydrocarbon receptor (AhR) signaling characterized by increased expression of downstream elements, including glutamate transporters and GABA receptors. Ultimately, this modulation led to the restoration of excitatory/inhibitory imbalance, a typical pathophysiological feature of ASD, and thereby alleviated ASD core behavioral symptoms. Our findings underscore Faecalibacterium-mediated AhR modulation as a promising therapeutic strategy for ASD, highlighting the dual potential of Faecalibacterium-based probiotics and targeted interventions against indole-AhR signaling to address neurodevelopmental disorders.https://www.frontiersin.org/articles/10.3389/fmicb.2025.1640149/fullthe gut-brain axisautism spectrum disorders (ASD)microbiomeindolearyl hydrocarbon receptor (AhR)BTBR
spellingShingle You Yu
Yujing Wang
Yujing Wang
Jie Zhang
Shucheng Li
Yulin Wang
Xin You
Xue Chen
Mengxuan Du
Lisheng Xie
Shuang-Jiang Liu
Shuang-Jiang Liu
The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
Frontiers in Microbiology
the gut-brain axis
autism spectrum disorders (ASD)
microbiome
indole
aryl hydrocarbon receptor (AhR)
BTBR
title The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
title_full The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
title_fullStr The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
title_full_unstemmed The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
title_short The gut commensal Faecalibacterium hominis attenuates indole—AhR signaling and restores ASD—like behaviors with BTBR mice
title_sort gut commensal faecalibacterium hominis attenuates indole ahr signaling and restores asd like behaviors with btbr mice
topic the gut-brain axis
autism spectrum disorders (ASD)
microbiome
indole
aryl hydrocarbon receptor (AhR)
BTBR
url https://www.frontiersin.org/articles/10.3389/fmicb.2025.1640149/full
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