Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2

Abstract Guanylate-binding proteins (GBPs) are interferon-inducible GTPases that confer protective immunity against a variety of intracellular pathogens. GBP2 is one of the two highly inducible GBPs, yet the precise mechanisms underlying the activation and regulation of GBP2, in particular the nucle...

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Main Authors: Sayantan Roy, Bing Wang, Krittika Roy, Yuan Tian, Madhurima Bhattacharya, Sarah Williams, Qian Yin
Format: Article
Language:English
Published: Nature Portfolio 2025-02-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-025-07727-3
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author Sayantan Roy
Bing Wang
Krittika Roy
Yuan Tian
Madhurima Bhattacharya
Sarah Williams
Qian Yin
author_facet Sayantan Roy
Bing Wang
Krittika Roy
Yuan Tian
Madhurima Bhattacharya
Sarah Williams
Qian Yin
author_sort Sayantan Roy
collection DOAJ
description Abstract Guanylate-binding proteins (GBPs) are interferon-inducible GTPases that confer protective immunity against a variety of intracellular pathogens. GBP2 is one of the two highly inducible GBPs, yet the precise mechanisms underlying the activation and regulation of GBP2, in particular the nucleotide-induced conformational changes in GBP2, remain poorly understood. In this study, we elucidate the structural plasticity of GBP2 upon nucleotide binding through crystallographic analysis. By determining the crystal structures of GBP2 G domain (GBP2GD) in complex with GDP and nucleotide-free full-length GBP2 with K51A mutation (GBP2K51A), we unveil distinct conformational states adopted by the nucleotide-binding pocket and distal regions of the protein. Comparison between the nucleotide-free full-length GBP2K51A structure with homologous structures reveals notable movement in the C-terminal helical region, along with conformational changes in the G domain. Through comparative analysis, we identify subtle but critical differences in the nucleotide-bound states of GBP2, providing insights into the molecular basis of its dimer-monomer transition and enzymatic activity. These findings pave the way for future investigations aimed at elucidating the precise molecular mechanisms underlying GBP2’s role in the immune response and open avenues for exploring how the unique functions of GBPs could be leveraged to combat pathogen invasion.
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spelling doaj-art-4963bcdf62b343ab90612c97c94ffcc22025-08-20T02:15:06ZengNature PortfolioCommunications Biology2399-36422025-02-018111210.1038/s42003-025-07727-3Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2Sayantan Roy0Bing Wang1Krittika Roy2Yuan Tian3Madhurima Bhattacharya4Sarah Williams5Qian Yin6Department of Biological Science, Florida State UniversityDepartment of Biological Science, Florida State UniversityInstitute of Molecular Biophysics, Florida State UniversityDepartment of Biological Science, Florida State UniversityInstitute of Molecular Biophysics, Florida State UniversityInstitute of Molecular Biophysics, Florida State UniversityDepartment of Biological Science, Florida State UniversityAbstract Guanylate-binding proteins (GBPs) are interferon-inducible GTPases that confer protective immunity against a variety of intracellular pathogens. GBP2 is one of the two highly inducible GBPs, yet the precise mechanisms underlying the activation and regulation of GBP2, in particular the nucleotide-induced conformational changes in GBP2, remain poorly understood. In this study, we elucidate the structural plasticity of GBP2 upon nucleotide binding through crystallographic analysis. By determining the crystal structures of GBP2 G domain (GBP2GD) in complex with GDP and nucleotide-free full-length GBP2 with K51A mutation (GBP2K51A), we unveil distinct conformational states adopted by the nucleotide-binding pocket and distal regions of the protein. Comparison between the nucleotide-free full-length GBP2K51A structure with homologous structures reveals notable movement in the C-terminal helical region, along with conformational changes in the G domain. Through comparative analysis, we identify subtle but critical differences in the nucleotide-bound states of GBP2, providing insights into the molecular basis of its dimer-monomer transition and enzymatic activity. These findings pave the way for future investigations aimed at elucidating the precise molecular mechanisms underlying GBP2’s role in the immune response and open avenues for exploring how the unique functions of GBPs could be leveraged to combat pathogen invasion.https://doi.org/10.1038/s42003-025-07727-3
spellingShingle Sayantan Roy
Bing Wang
Krittika Roy
Yuan Tian
Madhurima Bhattacharya
Sarah Williams
Qian Yin
Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
Communications Biology
title Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
title_full Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
title_fullStr Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
title_full_unstemmed Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
title_short Crystal structures reveal nucleotide-induced conformational changes in G motifs and distal regions in human guanylate-binding protein 2
title_sort crystal structures reveal nucleotide induced conformational changes in g motifs and distal regions in human guanylate binding protein 2
url https://doi.org/10.1038/s42003-025-07727-3
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