The length of vesicular stomatitis virus particles dictates a need for actin assembly during clathrin-dependent endocytosis.
Microbial pathogens exploit the clathrin endocytic machinery to enter host cells. Vesicular stomatitis virus (VSV), an enveloped virus with bullet-shaped virions that measure 70 x 200 nm, enters cells by clathrin-dependent endocytosis. We showed previously that VSV particles exceed the capacity of t...
Saved in:
| Main Authors: | David K Cureton, Ramiro H Massol, Sean P J Whelan, Tomas Kirchhausen |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Public Library of Science (PLoS)
2010-09-01
|
| Series: | PLoS Pathogens |
| Online Access: | https://doi.org/10.1371/journal.ppat.1001127 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Vesicular stomatitis virus enters cells through vesicles incompletely coated with clathrin that depend upon actin for internalization.
by: David K Cureton, et al.
Published: (2009-04-01) -
A feedback loop between dynamin and actin recruitment during clathrin-mediated endocytosis.
by: Marcus J Taylor, et al.
Published: (2012-01-01) -
Pathogenic Huntingtin aggregates alter actin organization and cellular stiffness resulting in stalled clathrin-mediated endocytosis
by: Surya Bansi Singh, et al.
Published: (2024-10-01) -
A freeze frame view of vesicular stomatitis virus transcription defines a minimal length of RNA for 5' processing.
by: Gergely Tekes, et al.
Published: (2011-06-01) -
Revisiting Pathogen Exploitation of Clathrin-Independent Endocytosis: Mechanisms and Implications
by: Oliver Goldmann, et al.
Published: (2025-05-01)