circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8

High circ_0020123 expression is associated with poor prognosis in patients with non-small cell lung cancer (NSCLC) as previously reported. Whether circ_0020123 also plays an oncogenic role in lung adenocarcinoma (LUAD) is still unknown. Additionally, circ_0020123 is derived from part of exon (1312–3...

Full description

Saved in:
Bibliographic Details
Main Authors: Wei Wei, Wang Changyong, Wang Lele, Zhang Jianfeng
Format: Article
Language:English
Published: De Gruyter 2022-03-01
Series:Open Medicine
Subjects:
Online Access:https://doi.org/10.1515/med-2022-0434
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849738360942231552
author Wei Wei
Wang Changyong
Wang Lele
Zhang Jianfeng
author_facet Wei Wei
Wang Changyong
Wang Lele
Zhang Jianfeng
author_sort Wei Wei
collection DOAJ
description High circ_0020123 expression is associated with poor prognosis in patients with non-small cell lung cancer (NSCLC) as previously reported. Whether circ_0020123 also plays an oncogenic role in lung adenocarcinoma (LUAD) is still unknown. Additionally, circ_0020123 is derived from part of exon (1312–3851) from its host gene PDZ domain-containing protein 8 (PDZD8). We hypothesized that circ_0020123 might affect malignant behaviors of LUAD cells by regulating PDZD8. Reverse transcription quantitative polymerase chain reaction revealed that PDZD8 was highly expressed in LUAD tissues and cells. PDZD8 knockdown suppressed LUAD cell proliferation and migration as shown by colony formation assays, Ethynyl deoxyuridine incorporation assays, Transwell assays, and wound healing assays. circ_0020123 was also found to be upregulated in LUAD tissues and cells. Moreover, circ_0020123 positively regulated PDZD8 expression in LUAD cells but exerted no significant effect on the transcriptional level of PDZD8. Mechanistically, circ_0020123 act as a competing endogenous RNA (ceRNA) to interact with miR-1283, thereby releasing the repression on PDZD8. Moreover, PDZD8 overexpression rescued the suppressive effect of circ_0020123 knockdown on LUAD cell proliferation and migration. In conclusion, circ_0020123 interacts with miR-1283 as a ceRNA to regulate PDZD8 expression, thus promoting the proliferation and migration of LUAD cells. The study might provide new biomarkers for future LUAD investigation.
format Article
id doaj-art-48e853454c9249d2b771ea253801322e
institution DOAJ
issn 2391-5463
language English
publishDate 2022-03-01
publisher De Gruyter
record_format Article
series Open Medicine
spelling doaj-art-48e853454c9249d2b771ea253801322e2025-08-20T03:06:37ZengDe GruyterOpen Medicine2391-54632022-03-0117153654910.1515/med-2022-0434circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8Wei Wei0Wang Changyong1Wang Lele2Zhang Jianfeng3Department of Cardiothoracic Surgery, Jinling Hospital, Nanjing University School of Medicine, Nanjing 210002, Jiangsu, ChinaDepartment of Cardiothoracic Surgery, Jinling Hospital, Nanjing University School of Medicine, Nanjing 210002, Jiangsu, ChinaDepartment of Cardiothoracic Surgery, Jinling Hospital, Nanjing University School of Medicine, Nanjing 210002, Jiangsu, ChinaDepartment of Cardiothoracic Surgery, Jinling Hospital, Nanjing University School of Medicine, 305 East Zhongshan Road, Nanjing 210002, Jiangsu, ChinaHigh circ_0020123 expression is associated with poor prognosis in patients with non-small cell lung cancer (NSCLC) as previously reported. Whether circ_0020123 also plays an oncogenic role in lung adenocarcinoma (LUAD) is still unknown. Additionally, circ_0020123 is derived from part of exon (1312–3851) from its host gene PDZ domain-containing protein 8 (PDZD8). We hypothesized that circ_0020123 might affect malignant behaviors of LUAD cells by regulating PDZD8. Reverse transcription quantitative polymerase chain reaction revealed that PDZD8 was highly expressed in LUAD tissues and cells. PDZD8 knockdown suppressed LUAD cell proliferation and migration as shown by colony formation assays, Ethynyl deoxyuridine incorporation assays, Transwell assays, and wound healing assays. circ_0020123 was also found to be upregulated in LUAD tissues and cells. Moreover, circ_0020123 positively regulated PDZD8 expression in LUAD cells but exerted no significant effect on the transcriptional level of PDZD8. Mechanistically, circ_0020123 act as a competing endogenous RNA (ceRNA) to interact with miR-1283, thereby releasing the repression on PDZD8. Moreover, PDZD8 overexpression rescued the suppressive effect of circ_0020123 knockdown on LUAD cell proliferation and migration. In conclusion, circ_0020123 interacts with miR-1283 as a ceRNA to regulate PDZD8 expression, thus promoting the proliferation and migration of LUAD cells. The study might provide new biomarkers for future LUAD investigation.https://doi.org/10.1515/med-2022-0434lung adenocarcinomapdzd8circ_0020123mir-1283cerna
spellingShingle Wei Wei
Wang Changyong
Wang Lele
Zhang Jianfeng
circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
Open Medicine
lung adenocarcinoma
pdzd8
circ_0020123
mir-1283
cerna
title circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
title_full circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
title_fullStr circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
title_full_unstemmed circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
title_short circ_0020123 promotes cell proliferation and migration in lung adenocarcinoma via PDZD8
title_sort circ 0020123 promotes cell proliferation and migration in lung adenocarcinoma via pdzd8
topic lung adenocarcinoma
pdzd8
circ_0020123
mir-1283
cerna
url https://doi.org/10.1515/med-2022-0434
work_keys_str_mv AT weiwei circ0020123promotescellproliferationandmigrationinlungadenocarcinomaviapdzd8
AT wangchangyong circ0020123promotescellproliferationandmigrationinlungadenocarcinomaviapdzd8
AT wanglele circ0020123promotescellproliferationandmigrationinlungadenocarcinomaviapdzd8
AT zhangjianfeng circ0020123promotescellproliferationandmigrationinlungadenocarcinomaviapdzd8