Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer

<i>Background and Objectives</i>: Von Hippel–Lindau (VHL) disease is caused by mutations in the VHL gene and can develop various cancers. Hypoxia-inducible factors 1 and 2 alphas, regulated by the VHL gene, can increase the levels of vascular endothelial growth factor, thereby activating...

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Main Authors: Sang-Won Moon, Jun-Chae Lee, Jae-Ho Lee, Tae-Young Kim, Jong Ho Park
Format: Article
Language:English
Published: MDPI AG 2025-02-01
Series:Medicina
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Online Access:https://www.mdpi.com/1648-9144/61/2/306
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author Sang-Won Moon
Jun-Chae Lee
Jae-Ho Lee
Tae-Young Kim
Jong Ho Park
author_facet Sang-Won Moon
Jun-Chae Lee
Jae-Ho Lee
Tae-Young Kim
Jong Ho Park
author_sort Sang-Won Moon
collection DOAJ
description <i>Background and Objectives</i>: Von Hippel–Lindau (VHL) disease is caused by mutations in the VHL gene and can develop various cancers. Hypoxia-inducible factors 1 and 2 alphas, regulated by the VHL gene, can increase the levels of vascular endothelial growth factor, thereby activating cancer progression. Here, we demonstrated clinical and prognostic values of VHL expression in rectal cancer (RC). <i>Materials and Methods</i>: Von Hippel–Lindau mRNA expression was examined in 60 patients with RC. Furthermore, we evaluated survival to determine the prognostic significance of VHL mRNA expression levels in RC using the Cancer Genome Atlas (TCGA) data. <i>Results</i>: Lower VHL expression was correlated with the recurrence (<i>p</i> = 0.058) and lymphatic invasion (<i>p</i> = 0.078), although it was not statistically significant. In TCGA data, VHL expression level was correlated with the M stage (<i>p</i> = 0.044); however, it had a possible association with lymphatic invasion (<i>p</i> = 0.068) and N stage (<i>p</i> = 0.104). Survival analysis showed that lower VHL gene expression predicted poorer survival in both patients with RC and TCGA data. <i>Conclusions</i>: This study identified a significant correlation between VHL gene expression and RC for the first time using patient tissues and TCGA data, suggesting that the VHL gene expression level could be a potential biomarker or candidate for the treatment of RC. Further studies are required to identify the molecular pathogenesis and clinical characteristics of VHL disease in RC.
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spelling doaj-art-465f1388f5944ec89cb0803a6c6ec2bf2025-08-20T02:44:46ZengMDPI AGMedicina1010-660X1648-91442025-02-0161230610.3390/medicina61020306Clinical and Prognostic Value of VHL in Korean Patients with Rectal CancerSang-Won Moon0Jun-Chae Lee1Jae-Ho Lee2Tae-Young Kim3Jong Ho Park4Medical Course, School of Medicine, Keimyung University, Daegu 42601, Republic of KoreaMedical Course, School of Medicine, Keimyung University, Daegu 42601, Republic of KoreaDepartment of Anatomy, School of Medicine & Institute for Medical Science, Keimyung University, Daegu 42601, Republic of KoreaDepartment of Anatomy, School of Medicine & Institute for Medical Science, Keimyung University, Daegu 42601, Republic of KoreaDepartment of Anatomy, School of Medicine & Institute for Medical Science, Keimyung University, Daegu 42601, Republic of Korea<i>Background and Objectives</i>: Von Hippel–Lindau (VHL) disease is caused by mutations in the VHL gene and can develop various cancers. Hypoxia-inducible factors 1 and 2 alphas, regulated by the VHL gene, can increase the levels of vascular endothelial growth factor, thereby activating cancer progression. Here, we demonstrated clinical and prognostic values of VHL expression in rectal cancer (RC). <i>Materials and Methods</i>: Von Hippel–Lindau mRNA expression was examined in 60 patients with RC. Furthermore, we evaluated survival to determine the prognostic significance of VHL mRNA expression levels in RC using the Cancer Genome Atlas (TCGA) data. <i>Results</i>: Lower VHL expression was correlated with the recurrence (<i>p</i> = 0.058) and lymphatic invasion (<i>p</i> = 0.078), although it was not statistically significant. In TCGA data, VHL expression level was correlated with the M stage (<i>p</i> = 0.044); however, it had a possible association with lymphatic invasion (<i>p</i> = 0.068) and N stage (<i>p</i> = 0.104). Survival analysis showed that lower VHL gene expression predicted poorer survival in both patients with RC and TCGA data. <i>Conclusions</i>: This study identified a significant correlation between VHL gene expression and RC for the first time using patient tissues and TCGA data, suggesting that the VHL gene expression level could be a potential biomarker or candidate for the treatment of RC. Further studies are required to identify the molecular pathogenesis and clinical characteristics of VHL disease in RC.https://www.mdpi.com/1648-9144/61/2/306VHLTCGArectal cancer
spellingShingle Sang-Won Moon
Jun-Chae Lee
Jae-Ho Lee
Tae-Young Kim
Jong Ho Park
Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
Medicina
VHL
TCGA
rectal cancer
title Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
title_full Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
title_fullStr Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
title_full_unstemmed Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
title_short Clinical and Prognostic Value of VHL in Korean Patients with Rectal Cancer
title_sort clinical and prognostic value of vhl in korean patients with rectal cancer
topic VHL
TCGA
rectal cancer
url https://www.mdpi.com/1648-9144/61/2/306
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AT taeyoungkim clinicalandprognosticvalueofvhlinkoreanpatientswithrectalcancer
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