Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells

The proliferation of tumors is not merely self-regulated by the cancer cells but is also intrinsically connected to the tumor microenvironment (TME). Within this complex TME, cancer-associated fibroblasts (CAFs) are pivotal in the modulation of tumor onset and progression. Rich signaling interaction...

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Main Authors: Chenbo Yang, Jiao Shu, Yiwei Li, Na Zhao, Xiaonan Liu, Xiangyu Tian, Zexin Sun, Muhammad Saud Tabish, Yichen Hong, Kuisheng Chen, Miaomiao Sun
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-12-01
Series:Frontiers in Immunology
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Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2024.1469918/full
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author Chenbo Yang
Chenbo Yang
Jiao Shu
Jiao Shu
Yiwei Li
Yiwei Li
Yiwei Li
Na Zhao
Na Zhao
Xiaonan Liu
Xiaonan Liu
Xiangyu Tian
Xiangyu Tian
Zexin Sun
Zexin Sun
Zexin Sun
Muhammad Saud Tabish
Muhammad Saud Tabish
Yichen Hong
Yichen Hong
Yichen Hong
Kuisheng Chen
Kuisheng Chen
Miaomiao Sun
Miaomiao Sun
author_facet Chenbo Yang
Chenbo Yang
Jiao Shu
Jiao Shu
Yiwei Li
Yiwei Li
Yiwei Li
Na Zhao
Na Zhao
Xiaonan Liu
Xiaonan Liu
Xiangyu Tian
Xiangyu Tian
Zexin Sun
Zexin Sun
Zexin Sun
Muhammad Saud Tabish
Muhammad Saud Tabish
Yichen Hong
Yichen Hong
Yichen Hong
Kuisheng Chen
Kuisheng Chen
Miaomiao Sun
Miaomiao Sun
author_sort Chenbo Yang
collection DOAJ
description The proliferation of tumors is not merely self-regulated by the cancer cells but is also intrinsically connected to the tumor microenvironment (TME). Within this complex TME, cancer-associated fibroblasts (CAFs) are pivotal in the modulation of tumor onset and progression. Rich signaling interactions exist between CAFs and tumor cells, which are crucial for tumor regulation. Long non-coding RNAs (LncRNAs) emerge from cellular transcription as a class of functionally diverse RNA molecules. Recent studies have revealed that LncRNAs are integral to the crosstalk between CAFs and tumor cells, with the capacity to modify cellular transcriptional activity and secretion profiles, thus facilitating CAFs activation, tumor proliferation, metastasis, drug resistance, and other related functionalities. This comprehensive review revisits the latest research on LncRNA-mediated interactions between CAFs and tumor cells, encapsulates the biological roles of LncRNAs, and delves into the molecular pathways from a broader perspective, aspiring to offer novel perspectives for a deeper comprehension of the etiology of tumors and the enhancement of therapeutic approaches.
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publisher Frontiers Media S.A.
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series Frontiers in Immunology
spelling doaj-art-45bdca9de1f6496f96c076261aa6ae2b2025-08-20T02:30:38ZengFrontiers Media S.A.Frontiers in Immunology1664-32242024-12-011510.3389/fimmu.2024.14699181469918Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cellsChenbo Yang0Chenbo Yang1Jiao Shu2Jiao Shu3Yiwei Li4Yiwei Li5Yiwei Li6Na Zhao7Na Zhao8Xiaonan Liu9Xiaonan Liu10Xiangyu Tian11Xiangyu Tian12Zexin Sun13Zexin Sun14Zexin Sun15Muhammad Saud Tabish16Muhammad Saud Tabish17Yichen Hong18Yichen Hong19Yichen Hong20Kuisheng Chen21Kuisheng Chen22Miaomiao Sun23Miaomiao Sun24Department of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaHenan Institute of Medical and Pharmaceutical Sciences, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaHenan Institute of Medical and Pharmaceutical Sciences, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaHenan Institute of Medical and Pharmaceutical Sciences, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaDepartment of Pathology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, ChinaHenan Key Laboratory of Tumor Pathology, Zhengzhou University, Zhengzhou, ChinaThe proliferation of tumors is not merely self-regulated by the cancer cells but is also intrinsically connected to the tumor microenvironment (TME). Within this complex TME, cancer-associated fibroblasts (CAFs) are pivotal in the modulation of tumor onset and progression. Rich signaling interactions exist between CAFs and tumor cells, which are crucial for tumor regulation. Long non-coding RNAs (LncRNAs) emerge from cellular transcription as a class of functionally diverse RNA molecules. Recent studies have revealed that LncRNAs are integral to the crosstalk between CAFs and tumor cells, with the capacity to modify cellular transcriptional activity and secretion profiles, thus facilitating CAFs activation, tumor proliferation, metastasis, drug resistance, and other related functionalities. This comprehensive review revisits the latest research on LncRNA-mediated interactions between CAFs and tumor cells, encapsulates the biological roles of LncRNAs, and delves into the molecular pathways from a broader perspective, aspiring to offer novel perspectives for a deeper comprehension of the etiology of tumors and the enhancement of therapeutic approaches.https://www.frontiersin.org/articles/10.3389/fimmu.2024.1469918/fulllong non-coding RNAscancer-associated fibroblaststumor microenvironmentsignaling pathwaytumor growth
spellingShingle Chenbo Yang
Chenbo Yang
Jiao Shu
Jiao Shu
Yiwei Li
Yiwei Li
Yiwei Li
Na Zhao
Na Zhao
Xiaonan Liu
Xiaonan Liu
Xiangyu Tian
Xiangyu Tian
Zexin Sun
Zexin Sun
Zexin Sun
Muhammad Saud Tabish
Muhammad Saud Tabish
Yichen Hong
Yichen Hong
Yichen Hong
Kuisheng Chen
Kuisheng Chen
Miaomiao Sun
Miaomiao Sun
Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
Frontiers in Immunology
long non-coding RNAs
cancer-associated fibroblasts
tumor microenvironment
signaling pathway
tumor growth
title Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
title_full Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
title_fullStr Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
title_full_unstemmed Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
title_short Long non-coding RNAs are involved in the crosstalk between cancer-associated fibroblasts and tumor cells
title_sort long non coding rnas are involved in the crosstalk between cancer associated fibroblasts and tumor cells
topic long non-coding RNAs
cancer-associated fibroblasts
tumor microenvironment
signaling pathway
tumor growth
url https://www.frontiersin.org/articles/10.3389/fimmu.2024.1469918/full
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