Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.

The mouse granular convoluted tubules (GCTs), which are only located in the submandibular gland (SMG) are known to develop and maintain their structure in an androgen-dependent manner. We previously demonstrated that the GCTs are involuted by the epithelial deletion of core binding factor β (CBFβ),...

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Main Authors: Hitomi Ono Minagi, Safiye Esra Sarper, Hiroshi Kurosaka, Koh-Ichi Kuremoto, Ichiro Taniuchi, Takayoshi Sakai, Takashi Yamashiro
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2017-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184395&type=printable
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author Hitomi Ono Minagi
Safiye Esra Sarper
Hiroshi Kurosaka
Koh-Ichi Kuremoto
Ichiro Taniuchi
Takayoshi Sakai
Takashi Yamashiro
author_facet Hitomi Ono Minagi
Safiye Esra Sarper
Hiroshi Kurosaka
Koh-Ichi Kuremoto
Ichiro Taniuchi
Takayoshi Sakai
Takashi Yamashiro
author_sort Hitomi Ono Minagi
collection DOAJ
description The mouse granular convoluted tubules (GCTs), which are only located in the submandibular gland (SMG) are known to develop and maintain their structure in an androgen-dependent manner. We previously demonstrated that the GCTs are involuted by the epithelial deletion of core binding factor β (CBFβ), a transcription factor that physically interacts with any of the Runt-related transcription factor (RUNX) proteins (RUNX1, 2 and 3). This result clearly demonstrates that the Runx /Cbfb signaling pathway is indispensable in the development of the GCTs. However, it is not clear which of the RUNX proteins plays useful role in the development of the GCTs by activating the Runx /Cbfb signaling pathway. Past studies have revealed that the Runx /Cbfb signaling pathway plays important roles in various aspects of development and homeostatic events. Moreover, the Runx genes have different temporospatial requirements depending on the biological situation. In the present study, the GCTs of the SMG showed a remarkable phenotype of, which phenocopied the epithelial deletion of Cbfb, in epithelial-specific Runx1 conditional knock-out (cKO) mice. The results indicate that Runx1 works as a partner of Cbfb during the development of the GCTs. We also discovered that the depletion of Runx1 resulted in the reduced secretion of saliva in male mice. Consistent with this finding, one of the water channels, Aquaporin-5 (AQP5) was mislocalized in the cytoplasm of the Runx1 mutants, suggesting a novel role of Runx1 in the membrane trafficking of AQP5. In summary, the present findings demonstrated that RUNX1 is essential for the development of the GCTs. Furthermore, RUNX1 could also be involved in the membrane trafficking of the AQP5 protein of the acinar cells in the SMG in order to allow for the proper secretion of saliva.
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spelling doaj-art-440deb4d0d52444c84a02164ef2c4e622025-08-20T02:03:58ZengPublic Library of Science (PLoS)PLoS ONE1932-62032017-01-01129e018439510.1371/journal.pone.0184395Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.Hitomi Ono MinagiSafiye Esra SarperHiroshi KurosakaKoh-Ichi KuremotoIchiro TaniuchiTakayoshi SakaiTakashi YamashiroThe mouse granular convoluted tubules (GCTs), which are only located in the submandibular gland (SMG) are known to develop and maintain their structure in an androgen-dependent manner. We previously demonstrated that the GCTs are involuted by the epithelial deletion of core binding factor β (CBFβ), a transcription factor that physically interacts with any of the Runt-related transcription factor (RUNX) proteins (RUNX1, 2 and 3). This result clearly demonstrates that the Runx /Cbfb signaling pathway is indispensable in the development of the GCTs. However, it is not clear which of the RUNX proteins plays useful role in the development of the GCTs by activating the Runx /Cbfb signaling pathway. Past studies have revealed that the Runx /Cbfb signaling pathway plays important roles in various aspects of development and homeostatic events. Moreover, the Runx genes have different temporospatial requirements depending on the biological situation. In the present study, the GCTs of the SMG showed a remarkable phenotype of, which phenocopied the epithelial deletion of Cbfb, in epithelial-specific Runx1 conditional knock-out (cKO) mice. The results indicate that Runx1 works as a partner of Cbfb during the development of the GCTs. We also discovered that the depletion of Runx1 resulted in the reduced secretion of saliva in male mice. Consistent with this finding, one of the water channels, Aquaporin-5 (AQP5) was mislocalized in the cytoplasm of the Runx1 mutants, suggesting a novel role of Runx1 in the membrane trafficking of AQP5. In summary, the present findings demonstrated that RUNX1 is essential for the development of the GCTs. Furthermore, RUNX1 could also be involved in the membrane trafficking of the AQP5 protein of the acinar cells in the SMG in order to allow for the proper secretion of saliva.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184395&type=printable
spellingShingle Hitomi Ono Minagi
Safiye Esra Sarper
Hiroshi Kurosaka
Koh-Ichi Kuremoto
Ichiro Taniuchi
Takayoshi Sakai
Takashi Yamashiro
Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
PLoS ONE
title Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
title_full Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
title_fullStr Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
title_full_unstemmed Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
title_short Runx1 mediates the development of the granular convoluted tubules in the submandibular glands.
title_sort runx1 mediates the development of the granular convoluted tubules in the submandibular glands
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184395&type=printable
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