Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis

Relapsing polychondritis (RPC) is an autoimmune disease characterized by the inflammation of cartilaginous tissues and other proteoglycan rich tissues. A concomitant disease, particularly myelodysplasia or systemic autoimmune disease can be detected in one-third of the patients with RPC. Unlike adul...

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Main Authors: E. G. Khaleva, G. A. Novik, F. V. Rokhlina
Format: Article
Language:English
Published: Siberian State Medical University (Tomsk) 2018-07-01
Series:Бюллетень сибирской медицины
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Online Access:https://bulletin.ssmu.ru/jour/article/view/1214
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author E. G. Khaleva
G. A. Novik
F. V. Rokhlina
author_facet E. G. Khaleva
G. A. Novik
F. V. Rokhlina
author_sort E. G. Khaleva
collection DOAJ
description Relapsing polychondritis (RPC) is an autoimmune disease characterized by the inflammation of cartilaginous tissues and other proteoglycan rich tissues. A concomitant disease, particularly myelodysplasia or systemic autoimmune disease can be detected in one-third of the patients with RPC. Unlike adults in children, RPC is less often associated with other autoimmune diseases. The diagnosis of RPC is established using the criteria of Mc Adam (1976) or Damiani (1979). The basis of the pathogenesis of RPC is an autoimmune reaction, which is initially directed against cartilage and then spreads to non-cartilaginous tissues. One of the elements in the pathogenesis of RPC is the mechanical trauma of cartilage, resulting in the release of pro-inflammatory cytokines (tumor necrosis factor alpha, interferon-γ, interleukin-8, and macrophage inflammatory protein 1) and local inflammation followed by the formation of autoantibodies in a patient with a genetic predisposition. In the treatment of RPC, steroids, non-steroidal anti-inflammatory drugs, colchicine are used and, if they are ineffective, immunosuppressants are prescribed. The most effective anti-cytokine drugs used in the treatment of RPC are tumor necrosis factor-alpha (TNF-α) inhibitors, IL-1 receptor antagonists, an inhibitor of the costimulatory pathway of T-lymphocyte activation, monoclonal antibodies against the IL-6 receptor. Given the fact that management of these patients is very complex, the aim of the study is to review available data on pathogenetic mechanisms of biological agents in managing of relapsing polychondritis.
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spelling doaj-art-43ecff4332ea42708973ed6c9d1c46062025-08-20T03:21:43ZengSiberian State Medical University (Tomsk)Бюллетень сибирской медицины1682-03631819-36842018-07-0117216717410.20538/1682-0363-2018-2-167-174800Pathogenetic mechanisms of biological agents in managing of relapsing polychondritisE. G. Khaleva0G. A. Novik1F. V. Rokhlina2Saint-Petersburg State Pediatric Medical UniversitySaint-Petersburg State Pediatric Medical UniversitySaint-Petersburg State Pediatric Medical University Research Сhildren’s Orthopedic Institute named after G.I. TurnerRelapsing polychondritis (RPC) is an autoimmune disease characterized by the inflammation of cartilaginous tissues and other proteoglycan rich tissues. A concomitant disease, particularly myelodysplasia or systemic autoimmune disease can be detected in one-third of the patients with RPC. Unlike adults in children, RPC is less often associated with other autoimmune diseases. The diagnosis of RPC is established using the criteria of Mc Adam (1976) or Damiani (1979). The basis of the pathogenesis of RPC is an autoimmune reaction, which is initially directed against cartilage and then spreads to non-cartilaginous tissues. One of the elements in the pathogenesis of RPC is the mechanical trauma of cartilage, resulting in the release of pro-inflammatory cytokines (tumor necrosis factor alpha, interferon-γ, interleukin-8, and macrophage inflammatory protein 1) and local inflammation followed by the formation of autoantibodies in a patient with a genetic predisposition. In the treatment of RPC, steroids, non-steroidal anti-inflammatory drugs, colchicine are used and, if they are ineffective, immunosuppressants are prescribed. The most effective anti-cytokine drugs used in the treatment of RPC are tumor necrosis factor-alpha (TNF-α) inhibitors, IL-1 receptor antagonists, an inhibitor of the costimulatory pathway of T-lymphocyte activation, monoclonal antibodies against the IL-6 receptor. Given the fact that management of these patients is very complex, the aim of the study is to review available data on pathogenetic mechanisms of biological agents in managing of relapsing polychondritis.https://bulletin.ssmu.ru/jour/article/view/1214relapsing polychondritisclinicsetiopathogenesisbiological agents
spellingShingle E. G. Khaleva
G. A. Novik
F. V. Rokhlina
Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
Бюллетень сибирской медицины
relapsing polychondritis
clinics
etiopathogenesis
biological agents
title Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
title_full Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
title_fullStr Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
title_full_unstemmed Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
title_short Pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
title_sort pathogenetic mechanisms of biological agents in managing of relapsing polychondritis
topic relapsing polychondritis
clinics
etiopathogenesis
biological agents
url https://bulletin.ssmu.ru/jour/article/view/1214
work_keys_str_mv AT egkhaleva pathogeneticmechanismsofbiologicalagentsinmanagingofrelapsingpolychondritis
AT ganovik pathogeneticmechanismsofbiologicalagentsinmanagingofrelapsingpolychondritis
AT fvrokhlina pathogeneticmechanismsofbiologicalagentsinmanagingofrelapsingpolychondritis