I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.

Acute mesenteric ischemia (AMI) is a life-threatening condition that can result in multiple organ injury and death. A timely diagnosis and treatment would have a significant impact on the morbidity and mortality in high-risk patient population. The purpose of this study was to investigate if intesti...

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Main Authors: Rachel G Khadaroo, Spyridon Fortis, Saad Y Salim, Catherine Streutker, Thomas A Churchill, Haibo Zhang
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2014-01-01
Series:PLoS ONE
Online Access:https://doi.org/10.1371/journal.pone.0115242
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author Rachel G Khadaroo
Spyridon Fortis
Saad Y Salim
Catherine Streutker
Thomas A Churchill
Haibo Zhang
author_facet Rachel G Khadaroo
Spyridon Fortis
Saad Y Salim
Catherine Streutker
Thomas A Churchill
Haibo Zhang
author_sort Rachel G Khadaroo
collection DOAJ
description Acute mesenteric ischemia (AMI) is a life-threatening condition that can result in multiple organ injury and death. A timely diagnosis and treatment would have a significant impact on the morbidity and mortality in high-risk patient population. The purpose of this study was to investigate if intestinal fatty acid binding protein (I-FABP) and α-defensins can be used as biomarkers for early AMI and resultant lung injury. C57BL/6 mice were subjected to intestinal ischemia by occlusion of the superior mesenteric artery. A time course of intestinal ischemia from 0.5 to 3 h was performed and followed by reperfusion for 2 h. Additional mice were treated with N-acetyl-cysteine (NAC) at 300 mg/kg given intraperitoneally prior to reperfusion. AMI resulted in severe intestinal injury characterized by neutrophil infiltrate, myeloperoxidase (MPO) levels, cytokine/chemokine levels, and tissue histopathology. Pathologic signs of ischemia were evident at 1 h, and by 3 h of ischemia, the full thickness of the intestine mucosa had areas of coagulative necrosis. It was noted that the levels of α-defensins in intestinal tissue peaked at 1 h and I-FABP in plasma peaked at 3 h after AMI. Intestinal ischemia also resulted in lung injury in a time-dependent manner. Pretreatment with NAC decreased the levels of intestinal α-defensins and plasma I-FABP, as well as lung MPO and cytokines. In summary, the concentrations of intestinal α-defensins and plasma I-FABP predicted intestinal ischemia prior to pathological evidence of ischemia and I-FABP directly correlated with resultant lung injury. The antioxidant NAC reduced intestinal and lung injury induced by AMI, suggesting a role for oxidants in the mechanism for distant organ injury. I-FABP and α-defensins are promising biomarkers, and may guide the treatment with antioxidant in early intestinal and distal organ injury.
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spelling doaj-art-43cd5162a13c4e089118e0e6bd16c5b52025-08-20T03:10:09ZengPublic Library of Science (PLoS)PLoS ONE1932-62032014-01-01912e11524210.1371/journal.pone.0115242I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.Rachel G KhadarooSpyridon FortisSaad Y SalimCatherine StreutkerThomas A ChurchillHaibo ZhangAcute mesenteric ischemia (AMI) is a life-threatening condition that can result in multiple organ injury and death. A timely diagnosis and treatment would have a significant impact on the morbidity and mortality in high-risk patient population. The purpose of this study was to investigate if intestinal fatty acid binding protein (I-FABP) and α-defensins can be used as biomarkers for early AMI and resultant lung injury. C57BL/6 mice were subjected to intestinal ischemia by occlusion of the superior mesenteric artery. A time course of intestinal ischemia from 0.5 to 3 h was performed and followed by reperfusion for 2 h. Additional mice were treated with N-acetyl-cysteine (NAC) at 300 mg/kg given intraperitoneally prior to reperfusion. AMI resulted in severe intestinal injury characterized by neutrophil infiltrate, myeloperoxidase (MPO) levels, cytokine/chemokine levels, and tissue histopathology. Pathologic signs of ischemia were evident at 1 h, and by 3 h of ischemia, the full thickness of the intestine mucosa had areas of coagulative necrosis. It was noted that the levels of α-defensins in intestinal tissue peaked at 1 h and I-FABP in plasma peaked at 3 h after AMI. Intestinal ischemia also resulted in lung injury in a time-dependent manner. Pretreatment with NAC decreased the levels of intestinal α-defensins and plasma I-FABP, as well as lung MPO and cytokines. In summary, the concentrations of intestinal α-defensins and plasma I-FABP predicted intestinal ischemia prior to pathological evidence of ischemia and I-FABP directly correlated with resultant lung injury. The antioxidant NAC reduced intestinal and lung injury induced by AMI, suggesting a role for oxidants in the mechanism for distant organ injury. I-FABP and α-defensins are promising biomarkers, and may guide the treatment with antioxidant in early intestinal and distal organ injury.https://doi.org/10.1371/journal.pone.0115242
spellingShingle Rachel G Khadaroo
Spyridon Fortis
Saad Y Salim
Catherine Streutker
Thomas A Churchill
Haibo Zhang
I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
PLoS ONE
title I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
title_full I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
title_fullStr I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
title_full_unstemmed I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
title_short I-FABP as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury.
title_sort i fabp as biomarker for the early diagnosis of acute mesenteric ischemia and resultant lung injury
url https://doi.org/10.1371/journal.pone.0115242
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