NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS

Role of nitric oxide in the pathogenesis of pulmonary tuberculosis has been studied in 77 patients with new infiltrate pulmonary tuberculosis and 34 patients with fibrous cavernous pulmonary tuberculosis. The level of cumulative and endogenous nitrite depended on the clinical form of tuberculosis: i...

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Main Authors: M. E. Dyakova, N. P. Alekseeva, D. S. Esmedlyaeva, T. L. Perova, N. N. Petrischev, P. K. Yablonskiy
Format: Article
Language:Russian
Published: New Terra Publishing House 2017-02-01
Series:Туберкулез и болезни лёгких
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Online Access:https://www.tibl-journal.com/jour/article/view/975
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author M. E. Dyakova
N. P. Alekseeva
D. S. Esmedlyaeva
T. L. Perova
N. N. Petrischev
P. K. Yablonskiy
author_facet M. E. Dyakova
N. P. Alekseeva
D. S. Esmedlyaeva
T. L. Perova
N. N. Petrischev
P. K. Yablonskiy
author_sort M. E. Dyakova
collection DOAJ
description Role of nitric oxide in the pathogenesis of pulmonary tuberculosis has been studied in 77 patients with new infiltrate pulmonary tuberculosis and 34 patients with fibrous cavernous pulmonary tuberculosis. The level of cumulative and endogenous nitrite depended on the clinical form of tuberculosis: in infiltrate pulmonary tuberculosis patients it was within the limits of reference ranges, and in fibrous cavernous pulmonary tuberculosis patients it was significantly lower. Results of statistical analysis point out at the significant homogeneity (monofunctionality) of the set of rates, defining the level of NO metabolites in blood serum in infiltrate pulmonary tuberculosis, namely: impact of adenosine deaminase, levels of α1-protease inhibitor, ceruleoplasmin and age. On the contrary in case of fibrous cavernous pulmonary tuberculosis the diverse (multi-functional) complexes were detected including clinical blood rates providing influence on the reduction of NO level in blood. Nitric oxide in the patients with both clinical forms of tuberculosis correlated with classical markers of system inflammatory response, thus nitric oxide can be regarded as an integral component of inflammatory response with potential evaluation of prognosis of specific lesions during follow-up of changes.
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institution OA Journals
issn 2075-1230
2542-1506
language Russian
publishDate 2017-02-01
publisher New Terra Publishing House
record_format Article
series Туберкулез и болезни лёгких
spelling doaj-art-439d424e7ca545ee8131b6092bfa4a7c2025-08-20T01:47:52ZrusNew Terra Publishing HouseТуберкулез и болезни лёгких2075-12302542-15062017-02-01952455010.21292/2075-1230-2017-95-2-45-50975NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESISM. E. Dyakova0N. P. Alekseeva1D. S. Esmedlyaeva2T. L. Perova3N. N. Petrischev4P. K. Yablonskiy5St. Petersburg Research Institute of PhthisiopulmonologySt. Petersburg Research Institute of PhthisiopulmonologySt. Petersburg Research Institute of PhthisiopulmonologySt. Petersburg Research Institute of PhthisiopulmonologyPavlov First Saint Petersburg State Medical UniversitySt. Petersburg Research Institute of PhthisiopulmonologyRole of nitric oxide in the pathogenesis of pulmonary tuberculosis has been studied in 77 patients with new infiltrate pulmonary tuberculosis and 34 patients with fibrous cavernous pulmonary tuberculosis. The level of cumulative and endogenous nitrite depended on the clinical form of tuberculosis: in infiltrate pulmonary tuberculosis patients it was within the limits of reference ranges, and in fibrous cavernous pulmonary tuberculosis patients it was significantly lower. Results of statistical analysis point out at the significant homogeneity (monofunctionality) of the set of rates, defining the level of NO metabolites in blood serum in infiltrate pulmonary tuberculosis, namely: impact of adenosine deaminase, levels of α1-protease inhibitor, ceruleoplasmin and age. On the contrary in case of fibrous cavernous pulmonary tuberculosis the diverse (multi-functional) complexes were detected including clinical blood rates providing influence on the reduction of NO level in blood. Nitric oxide in the patients with both clinical forms of tuberculosis correlated with classical markers of system inflammatory response, thus nitric oxide can be regarded as an integral component of inflammatory response with potential evaluation of prognosis of specific lesions during follow-up of changes.https://www.tibl-journal.com/jour/article/view/975tuberculosissystem inflammatory responsenitric oxidegeneral and endogenous nitritesacute phase reactants
spellingShingle M. E. Dyakova
N. P. Alekseeva
D. S. Esmedlyaeva
T. L. Perova
N. N. Petrischev
P. K. Yablonskiy
NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
Туберкулез и болезни лёгких
tuberculosis
system inflammatory response
nitric oxide
general and endogenous nitrites
acute phase reactants
title NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
title_full NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
title_fullStr NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
title_full_unstemmed NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
title_short NITRIC OXIDE – BIOCHEMICAL MARKER OF TUBERCULOSIS PATHOGENESIS
title_sort nitric oxide biochemical marker of tuberculosis pathogenesis
topic tuberculosis
system inflammatory response
nitric oxide
general and endogenous nitrites
acute phase reactants
url https://www.tibl-journal.com/jour/article/view/975
work_keys_str_mv AT medyakova nitricoxidebiochemicalmarkeroftuberculosispathogenesis
AT npalekseeva nitricoxidebiochemicalmarkeroftuberculosispathogenesis
AT dsesmedlyaeva nitricoxidebiochemicalmarkeroftuberculosispathogenesis
AT tlperova nitricoxidebiochemicalmarkeroftuberculosispathogenesis
AT nnpetrischev nitricoxidebiochemicalmarkeroftuberculosispathogenesis
AT pkyablonskiy nitricoxidebiochemicalmarkeroftuberculosispathogenesis