Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma

Steroid-insensitive asthma is an infrequent but problematic airway disease that presents with persistent symptoms, airflow limitation, or recurrent exacerbations even when treated with steroid-based therapies. Because of unsatisfactory results obtained from currently available therapies for steroid-...

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Main Authors: Yuko Morishima, Satoshi Ano, Yukio Ishii, Shigeo Ohtsuka, Masashi Matsuyama, Mio Kawaguchi, Nobuyuki Hizawa
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Clinical and Developmental Immunology
Online Access:http://dx.doi.org/10.1155/2013/609395
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author Yuko Morishima
Satoshi Ano
Yukio Ishii
Shigeo Ohtsuka
Masashi Matsuyama
Mio Kawaguchi
Nobuyuki Hizawa
author_facet Yuko Morishima
Satoshi Ano
Yukio Ishii
Shigeo Ohtsuka
Masashi Matsuyama
Mio Kawaguchi
Nobuyuki Hizawa
author_sort Yuko Morishima
collection DOAJ
description Steroid-insensitive asthma is an infrequent but problematic airway disease that presents with persistent symptoms, airflow limitation, or recurrent exacerbations even when treated with steroid-based therapies. Because of unsatisfactory results obtained from currently available therapies for steroid-insensitive asthma, a better understanding of its pathogenesis and the development of new targeted molecular therapies are warranted. Recent studies indicated that levels of interleukin (IL)-17 are increased and both eosinophils and neutrophils infiltrate the airways of severe asthmatics. IL-17 is a proinflammatory cytokine mainly secreted from helper T (Th) 17 cells and is important for the induction of neutrophil recruitment and migration at sites of inflammation. This review focuses on the pathogenetic role of Th17 cells and their associated cytokines in steroid-insensitive asthma and discusses the prospects of novel therapeutic options targeting the Th17 signaling pathway.
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publishDate 2013-01-01
publisher Wiley
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series Clinical and Developmental Immunology
spelling doaj-art-4329eb2aa32a40db877d354ba32d478f2025-08-20T02:37:57ZengWileyClinical and Developmental Immunology1740-25221740-25302013-01-01201310.1155/2013/609395609395Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive AsthmaYuko Morishima0Satoshi Ano1Yukio Ishii2Shigeo Ohtsuka3Masashi Matsuyama4Mio Kawaguchi5Nobuyuki Hizawa6Division of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanDivision of Clinical Medicine, Department of Respiratory Medicine, Faculty of Medicine, University of Tsukuba, 1-1-1 Tennoudai, Tsukuba, Ibaraki 305-8575, JapanSteroid-insensitive asthma is an infrequent but problematic airway disease that presents with persistent symptoms, airflow limitation, or recurrent exacerbations even when treated with steroid-based therapies. Because of unsatisfactory results obtained from currently available therapies for steroid-insensitive asthma, a better understanding of its pathogenesis and the development of new targeted molecular therapies are warranted. Recent studies indicated that levels of interleukin (IL)-17 are increased and both eosinophils and neutrophils infiltrate the airways of severe asthmatics. IL-17 is a proinflammatory cytokine mainly secreted from helper T (Th) 17 cells and is important for the induction of neutrophil recruitment and migration at sites of inflammation. This review focuses on the pathogenetic role of Th17 cells and their associated cytokines in steroid-insensitive asthma and discusses the prospects of novel therapeutic options targeting the Th17 signaling pathway.http://dx.doi.org/10.1155/2013/609395
spellingShingle Yuko Morishima
Satoshi Ano
Yukio Ishii
Shigeo Ohtsuka
Masashi Matsuyama
Mio Kawaguchi
Nobuyuki Hizawa
Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
Clinical and Developmental Immunology
title Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
title_full Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
title_fullStr Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
title_full_unstemmed Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
title_short Th17-Associated Cytokines as a Therapeutic Target for Steroid-Insensitive Asthma
title_sort th17 associated cytokines as a therapeutic target for steroid insensitive asthma
url http://dx.doi.org/10.1155/2013/609395
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