Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.

The naked mole rat (NMR), Heterocephalus glaber, is known as the longest-lived rodent and is extraordinarily resistant to hypoxia and cancer. Here, both NMR embryonic fibroblasts (NEFs) and their mouse counterparts (MEFs) were subjected to anoxic conditions (0% O2, 5% CO2). A combination of comparat...

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Main Authors: Juan Du, Weiqiang Liu, Meng Li, Zihao Li, Xuanjing Li, Yichen Dai, Gaoming Liu, Xiao Wang, Pingfen Zhu, Vadim N Gladyshev, Xuming Zhou
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2024-08-01
Series:PLoS Biology
Online Access:https://doi.org/10.1371/journal.pbio.3002778
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author Juan Du
Weiqiang Liu
Meng Li
Zihao Li
Xuanjing Li
Yichen Dai
Gaoming Liu
Xiao Wang
Pingfen Zhu
Vadim N Gladyshev
Xuming Zhou
author_facet Juan Du
Weiqiang Liu
Meng Li
Zihao Li
Xuanjing Li
Yichen Dai
Gaoming Liu
Xiao Wang
Pingfen Zhu
Vadim N Gladyshev
Xuming Zhou
author_sort Juan Du
collection DOAJ
description The naked mole rat (NMR), Heterocephalus glaber, is known as the longest-lived rodent and is extraordinarily resistant to hypoxia and cancer. Here, both NMR embryonic fibroblasts (NEFs) and their mouse counterparts (MEFs) were subjected to anoxic conditions (0% O2, 5% CO2). A combination of comparative transcriptomics and proteomics was then employed to identify differentially expressed genes (DEGs). Notably, we observed distinct levels of histone H1.2 (encoded by HIST1H1C) accumulation between NEFs and MEFs. Subsequent mechanistic analyses showed that higher H1.2 expression in NEFs was associated with the lower expression of its inhibitor, PARP1. Additionally, we discovered that H1.2 can directly interact with HIF-1α PAS domains, thereby promoting the expression of HIF-1α through facilitating the dimerization with HIF-1β. The overexpression of H1.2 was also found to trigger autophagy and to suppress the migration of cancer cells, as well as the formation of xenograft tumors, via the NRF2/P62 signaling pathway. Moreover, an engineered H1.2 knock-in mouse model exhibited significantly extended survival in hypoxic conditions (4% O2) and showed a reduced rate of tumor formation. Collectively, our results indicate a potential mechanistic link between H1.2 and the dual phenomena of anoxic adaptation and cancer resistance.
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institution Kabale University
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spelling doaj-art-4244d1b9a66e4a2b9dcb975b7e59a4722025-08-20T03:29:53ZengPublic Library of Science (PLoS)PLoS Biology1544-91731545-78852024-08-01228e300277810.1371/journal.pbio.3002778Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.Juan DuWeiqiang LiuMeng LiZihao LiXuanjing LiYichen DaiGaoming LiuXiao WangPingfen ZhuVadim N GladyshevXuming ZhouThe naked mole rat (NMR), Heterocephalus glaber, is known as the longest-lived rodent and is extraordinarily resistant to hypoxia and cancer. Here, both NMR embryonic fibroblasts (NEFs) and their mouse counterparts (MEFs) were subjected to anoxic conditions (0% O2, 5% CO2). A combination of comparative transcriptomics and proteomics was then employed to identify differentially expressed genes (DEGs). Notably, we observed distinct levels of histone H1.2 (encoded by HIST1H1C) accumulation between NEFs and MEFs. Subsequent mechanistic analyses showed that higher H1.2 expression in NEFs was associated with the lower expression of its inhibitor, PARP1. Additionally, we discovered that H1.2 can directly interact with HIF-1α PAS domains, thereby promoting the expression of HIF-1α through facilitating the dimerization with HIF-1β. The overexpression of H1.2 was also found to trigger autophagy and to suppress the migration of cancer cells, as well as the formation of xenograft tumors, via the NRF2/P62 signaling pathway. Moreover, an engineered H1.2 knock-in mouse model exhibited significantly extended survival in hypoxic conditions (4% O2) and showed a reduced rate of tumor formation. Collectively, our results indicate a potential mechanistic link between H1.2 and the dual phenomena of anoxic adaptation and cancer resistance.https://doi.org/10.1371/journal.pbio.3002778
spellingShingle Juan Du
Weiqiang Liu
Meng Li
Zihao Li
Xuanjing Li
Yichen Dai
Gaoming Liu
Xiao Wang
Pingfen Zhu
Vadim N Gladyshev
Xuming Zhou
Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
PLoS Biology
title Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
title_full Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
title_fullStr Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
title_full_unstemmed Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
title_short Comparative time-series multi-omics analyses suggest H1.2 involvement in anoxic adaptation and cancer resistance.
title_sort comparative time series multi omics analyses suggest h1 2 involvement in anoxic adaptation and cancer resistance
url https://doi.org/10.1371/journal.pbio.3002778
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