Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome

Cranial sutures are unossified connective tissue structures between the cranial bones, which allow expansion of these bones during development. Premature ossification of these structures is called craniosynostosis. Apert syndrome is a well-defined genetic syndrome, which is characterized by cranios...

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Main Authors: Arda Çetinkaya, Ekim Taşkıran, Tutku Soyer, Pelin Özlem Şimşek-Kiper, Gülen Eda Utine, Gökhan Tunçbilek, Koray Boduroğlu, Mehmet Alikaşifoğlu
Format: Article
Language:English
Published: Hacettepe University Institute of Child Health 2017-12-01
Series:The Turkish Journal of Pediatrics
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Online Access:https://turkjpediatr.org/article/view/1043
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author Arda Çetinkaya
Ekim Taşkıran
Tutku Soyer
Pelin Özlem Şimşek-Kiper
Gülen Eda Utine
Gökhan Tunçbilek
Koray Boduroğlu
Mehmet Alikaşifoğlu
author_facet Arda Çetinkaya
Ekim Taşkıran
Tutku Soyer
Pelin Özlem Şimşek-Kiper
Gülen Eda Utine
Gökhan Tunçbilek
Koray Boduroğlu
Mehmet Alikaşifoğlu
author_sort Arda Çetinkaya
collection DOAJ
description Cranial sutures are unossified connective tissue structures between the cranial bones, which allow expansion of these bones during development. Premature ossification of these structures is called craniosynostosis. Apert syndrome is a well-defined genetic syndrome, which is characterized by craniosynostosis and arises as a result of two missense mutations in Fibroblast Growth Factor Receptor, type 2 gene (FGFR2). In this study, differentially expressed genes in dermal fibroblasts from individuals with Apert syndrome and controls were investigated to identify important pathways in the pathogenesis of Apert syndrome. For this purpose, primary skin fibroblast cultures obtained from 3 individuals with Apert syndrome and 3 controls without craniosynostosis were compared by transcriptome microarray, GeneChip Human Genome U133 Plus 2.0. As a result, 181 genes were shown to be differentially expressed between experimental groups. Among these, 10 genes, which significantly differ in Apert syndrome fibroblasts compared to controls, were shown to be involved in a common interaction network and have common Gene ontology (GO) biological processes terms. COL11A1, COMP, CPXM2, ITGA8, MGF and TNC are differentially expressed genes that have GO terms associated with extracellular matrix (ECM) organization, while FRZB, SFRP2 and WNT2 are involved in WNT signaling pathway. Reorganization of ECM and changes in WNT signaling pathway show that Apert syndrome primary fibroblast cultures may have an increased potential for bone differentiation. The results of this study support craniosynostosis in Apert syndrome may be the result of fast and early differentiation of connective tissue along the sutures.
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spelling doaj-art-41f64cd4292345cab03f143d8d1cdb3c2025-08-20T02:01:55ZengHacettepe University Institute of Child HealthThe Turkish Journal of Pediatrics0041-43012791-64212017-12-0159610.24953/turkjped.2017.06.001Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndromeArda ÇetinkayaEkim Taşkıran0Tutku Soyer1Pelin Özlem Şimşek-Kiper2Gülen Eda Utine3Gökhan Tunçbilek4Koray Boduroğlu5Mehmet Alikaşifoğlu6Department of Medical Genetics, Faculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Pediatric Surgery, aculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Pediatric Genetics, Faculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Pediatric Genetics, Faculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Plastic Reconstructive and Aesthetic Surgery, Faculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Pediatric Genetics, Faculty of Medicine, Hacettepe University, Ankara, Turkey.Department of Medical Genetics, Faculty of Medicine, Hacettepe University, Ankara, Turkey. Cranial sutures are unossified connective tissue structures between the cranial bones, which allow expansion of these bones during development. Premature ossification of these structures is called craniosynostosis. Apert syndrome is a well-defined genetic syndrome, which is characterized by craniosynostosis and arises as a result of two missense mutations in Fibroblast Growth Factor Receptor, type 2 gene (FGFR2). In this study, differentially expressed genes in dermal fibroblasts from individuals with Apert syndrome and controls were investigated to identify important pathways in the pathogenesis of Apert syndrome. For this purpose, primary skin fibroblast cultures obtained from 3 individuals with Apert syndrome and 3 controls without craniosynostosis were compared by transcriptome microarray, GeneChip Human Genome U133 Plus 2.0. As a result, 181 genes were shown to be differentially expressed between experimental groups. Among these, 10 genes, which significantly differ in Apert syndrome fibroblasts compared to controls, were shown to be involved in a common interaction network and have common Gene ontology (GO) biological processes terms. COL11A1, COMP, CPXM2, ITGA8, MGF and TNC are differentially expressed genes that have GO terms associated with extracellular matrix (ECM) organization, while FRZB, SFRP2 and WNT2 are involved in WNT signaling pathway. Reorganization of ECM and changes in WNT signaling pathway show that Apert syndrome primary fibroblast cultures may have an increased potential for bone differentiation. The results of this study support craniosynostosis in Apert syndrome may be the result of fast and early differentiation of connective tissue along the sutures. https://turkjpediatr.org/article/view/1043Apert syndromecraniosynostosisfibroblastfibroblast growth factor receptor type 2transcriptome
spellingShingle Arda Çetinkaya
Ekim Taşkıran
Tutku Soyer
Pelin Özlem Şimşek-Kiper
Gülen Eda Utine
Gökhan Tunçbilek
Koray Boduroğlu
Mehmet Alikaşifoğlu
Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
The Turkish Journal of Pediatrics
Apert syndrome
craniosynostosis
fibroblast
fibroblast growth factor receptor type 2
transcriptome
title Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
title_full Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
title_fullStr Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
title_full_unstemmed Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
title_short Dermal fibroblast transcriptome indicates contribution of WNT signaling pathways in the pathogenesis of Apert syndrome
title_sort dermal fibroblast transcriptome indicates contribution of wnt signaling pathways in the pathogenesis of apert syndrome
topic Apert syndrome
craniosynostosis
fibroblast
fibroblast growth factor receptor type 2
transcriptome
url https://turkjpediatr.org/article/view/1043
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