Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer

Abstract Natural killer (NK) cells and T cells play crucial roles in liver metastatic cancer, particularly through their cytotoxic effects on tumor cells. Although existing evidence suggests a functional network of mutual regulation between NK and T cells, the nature of their interaction and its rol...

Full description

Saved in:
Bibliographic Details
Main Authors: Xiaoshuang Wang, Zhongen Wu, Yan Zhou, Dehua Yang, Qingtong Zhou, Di Zhu, Ming-Wei Wang, Lu Wang
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
Subjects:
Online Access:https://doi.org/10.1038/s41598-025-06241-0
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849399756322766848
author Xiaoshuang Wang
Zhongen Wu
Yan Zhou
Dehua Yang
Qingtong Zhou
Di Zhu
Ming-Wei Wang
Lu Wang
author_facet Xiaoshuang Wang
Zhongen Wu
Yan Zhou
Dehua Yang
Qingtong Zhou
Di Zhu
Ming-Wei Wang
Lu Wang
author_sort Xiaoshuang Wang
collection DOAJ
description Abstract Natural killer (NK) cells and T cells play crucial roles in liver metastatic cancer, particularly through their cytotoxic effects on tumor cells. Although existing evidence suggests a functional network of mutual regulation between NK and T cells, the nature of their interaction and its role in liver metastasis remain elusive. In this study, we analyzed single-cell transcriptomics of liver metastases and adjacent tissues from nasopharyngeal carcinoma (NPC), thyroid carcinoma (THCA), breast cancer (BC), colorectal cancer (CRC) and cervical cancer (CESC) to uncover the NK-T cell interaction network and its functional implications. In adjacent tissues, we observed increased infiltration of CD8+ NKT-like cells, γδT cells, and NK cells. The interaction between CD8+ NKT-like cells, CD8+ T cells, γδT cells, and NK cells were enhanced, with stronger signals associated with T and NK cell functions. Notably, CD8+ NKT-like cells, CD8+ T cells, and γδT cells exhibited an increased capacity to activate NK cells. These T cell subsets promoted NK cell anti-tumor activity via CD48-CD244 and TNF-TNFR signaling pathways, which in turn activated the ERK, JNK, and MAPK cascades. Our findings provide valuable insights into the NK-T cell interaction network in liver metastatic cancer, highlighting its potential as a therapeutic target.
format Article
id doaj-art-41855cde494841139f19ffbd63f14928
institution Kabale University
issn 2045-2322
language English
publishDate 2025-07-01
publisher Nature Portfolio
record_format Article
series Scientific Reports
spelling doaj-art-41855cde494841139f19ffbd63f149282025-08-20T03:38:15ZengNature PortfolioScientific Reports2045-23222025-07-0115111310.1038/s41598-025-06241-0Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancerXiaoshuang Wang0Zhongen Wu1Yan Zhou2Dehua Yang3Qingtong Zhou4Di Zhu5Ming-Wei Wang6Lu Wang7Shanghai Cancer Center, Shanghai Medical College, Fudan UniversitySchool of Pharmacy, Fudan UniversityThe National Center for Drug Screening, Shanghai Institute of Materia Medica, Chinese Academy of SciencesThe National Center for Drug Screening, Shanghai Institute of Materia Medica, Chinese Academy of SciencesSchool of Basic Medical Sciences, Fudan UniversitySchool of Pharmacy, Fudan UniversitySchool of Basic Medical Sciences, Fudan UniversityShanghai Cancer Center, Shanghai Medical College, Fudan UniversityAbstract Natural killer (NK) cells and T cells play crucial roles in liver metastatic cancer, particularly through their cytotoxic effects on tumor cells. Although existing evidence suggests a functional network of mutual regulation between NK and T cells, the nature of their interaction and its role in liver metastasis remain elusive. In this study, we analyzed single-cell transcriptomics of liver metastases and adjacent tissues from nasopharyngeal carcinoma (NPC), thyroid carcinoma (THCA), breast cancer (BC), colorectal cancer (CRC) and cervical cancer (CESC) to uncover the NK-T cell interaction network and its functional implications. In adjacent tissues, we observed increased infiltration of CD8+ NKT-like cells, γδT cells, and NK cells. The interaction between CD8+ NKT-like cells, CD8+ T cells, γδT cells, and NK cells were enhanced, with stronger signals associated with T and NK cell functions. Notably, CD8+ NKT-like cells, CD8+ T cells, and γδT cells exhibited an increased capacity to activate NK cells. These T cell subsets promoted NK cell anti-tumor activity via CD48-CD244 and TNF-TNFR signaling pathways, which in turn activated the ERK, JNK, and MAPK cascades. Our findings provide valuable insights into the NK-T cell interaction network in liver metastatic cancer, highlighting its potential as a therapeutic target.https://doi.org/10.1038/s41598-025-06241-0NK-T interactionLiver metastasisSingle-cell transcriptomics
spellingShingle Xiaoshuang Wang
Zhongen Wu
Yan Zhou
Dehua Yang
Qingtong Zhou
Di Zhu
Ming-Wei Wang
Lu Wang
Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
Scientific Reports
NK-T interaction
Liver metastasis
Single-cell transcriptomics
title Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
title_full Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
title_fullStr Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
title_full_unstemmed Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
title_short Single-cell transcriptomics analysis reveals a disrupted NK-T cell interaction network in liver metastatic cancer
title_sort single cell transcriptomics analysis reveals a disrupted nk t cell interaction network in liver metastatic cancer
topic NK-T interaction
Liver metastasis
Single-cell transcriptomics
url https://doi.org/10.1038/s41598-025-06241-0
work_keys_str_mv AT xiaoshuangwang singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT zhongenwu singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT yanzhou singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT dehuayang singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT qingtongzhou singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT dizhu singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT mingweiwang singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer
AT luwang singlecelltranscriptomicsanalysisrevealsadisruptednktcellinteractionnetworkinlivermetastaticcancer