The genetic regulation of protein expression in cerebrospinal fluid

Abstract Studies of the genetic regulation of cerebrospinal fluid (CSF) proteins may reveal pathways for treatment of neurological diseases. 398 proteins in CSF were measured in 1,591 participants from the BioFINDER study. Protein quantitative trait loci (pQTL) were identified as associations betwee...

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Main Authors: Oskar Hansson, Atul Kumar, Shorena Janelidze, Erik Stomrud, Philip S Insel, Kaj Blennow, Henrik Zetterberg, Eric Fauman, Åsa K Hedman, Michael W Nagle, Christopher D Whelan, Denis Baird, Anders Mälarstig, Niklas Mattsson‐Carlgren
Format: Article
Language:English
Published: Springer Nature 2022-12-01
Series:EMBO Molecular Medicine
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Online Access:https://doi.org/10.15252/emmm.202216359
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author Oskar Hansson
Atul Kumar
Shorena Janelidze
Erik Stomrud
Philip S Insel
Kaj Blennow
Henrik Zetterberg
Eric Fauman
Åsa K Hedman
Michael W Nagle
Christopher D Whelan
Denis Baird
Anders Mälarstig
Niklas Mattsson‐Carlgren
author_facet Oskar Hansson
Atul Kumar
Shorena Janelidze
Erik Stomrud
Philip S Insel
Kaj Blennow
Henrik Zetterberg
Eric Fauman
Åsa K Hedman
Michael W Nagle
Christopher D Whelan
Denis Baird
Anders Mälarstig
Niklas Mattsson‐Carlgren
author_sort Oskar Hansson
collection DOAJ
description Abstract Studies of the genetic regulation of cerebrospinal fluid (CSF) proteins may reveal pathways for treatment of neurological diseases. 398 proteins in CSF were measured in 1,591 participants from the BioFINDER study. Protein quantitative trait loci (pQTL) were identified as associations between genetic variants and proteins, with 176 pQTLs for 145 CSF proteins (P < 1.25 × 10−10, 117 cis‐pQTLs and 59 trans‐pQTLs). Ventricular volume (measured with brain magnetic resonance imaging) was a confounder for several pQTLs. pQTLs for CSF and plasma proteins were overall correlated, but CSF‐specific pQTLs were also observed. Mendelian randomization analyses suggested causal roles for several proteins, for example, ApoE, CD33, and GRN in Alzheimer's disease, MMP‐10 in preclinical Alzheimer's disease, SIGLEC9 in amyotrophic lateral sclerosis, and CD38, GPNMB, and ADAM15 in Parkinson's disease. CSF levels of GRN, MMP‐10, and GPNMB were altered in Alzheimer's disease, preclinical Alzheimer's disease, and Parkinson's disease, respectively. These findings point to pathways to be explored for novel therapies. The novel finding that ventricular volume confounded pQTLs has implications for design of future studies of the genetic regulation of the CSF proteome.
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spelling doaj-art-4174b4e6116f45eb86dccf005c92af0b2025-08-24T11:43:32ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842022-12-0115111610.15252/emmm.202216359The genetic regulation of protein expression in cerebrospinal fluidOskar Hansson0Atul Kumar1Shorena Janelidze2Erik Stomrud3Philip S Insel4Kaj Blennow5Henrik Zetterberg6Eric Fauman7Åsa K Hedman8Michael W Nagle9Christopher D Whelan10Denis Baird11Anders Mälarstig12Niklas Mattsson‐Carlgren13Clinical Memory Research Unit, Faculty of Medicine, Lund UniversityClinical Memory Research Unit, Faculty of Medicine, Lund UniversityClinical Memory Research Unit, Faculty of Medicine, Lund UniversityClinical Memory Research Unit, Faculty of Medicine, Lund UniversityClinical Memory Research Unit, Faculty of Medicine, Lund UniversityClinical Neurochemistry Laboratory, Sahlgrenska University HospitalClinical Neurochemistry Laboratory, Sahlgrenska University HospitalInternal Medicine Research Unit, Pfizer Worldwide Research, Development and MedicalPfizer Worldwide Research, Development and MedicalNeurogenomics, Genetics‐Guided Dementia Discovery, Eisai, IncTranslational Biology, Biogen Research & Development, Biogen IncDepartment of Neurology, Skåne University Hospital, Lund UniversityPfizer Worldwide Research, Development and MedicalClinical Memory Research Unit, Faculty of Medicine, Lund UniversityAbstract Studies of the genetic regulation of cerebrospinal fluid (CSF) proteins may reveal pathways for treatment of neurological diseases. 398 proteins in CSF were measured in 1,591 participants from the BioFINDER study. Protein quantitative trait loci (pQTL) were identified as associations between genetic variants and proteins, with 176 pQTLs for 145 CSF proteins (P < 1.25 × 10−10, 117 cis‐pQTLs and 59 trans‐pQTLs). Ventricular volume (measured with brain magnetic resonance imaging) was a confounder for several pQTLs. pQTLs for CSF and plasma proteins were overall correlated, but CSF‐specific pQTLs were also observed. Mendelian randomization analyses suggested causal roles for several proteins, for example, ApoE, CD33, and GRN in Alzheimer's disease, MMP‐10 in preclinical Alzheimer's disease, SIGLEC9 in amyotrophic lateral sclerosis, and CD38, GPNMB, and ADAM15 in Parkinson's disease. CSF levels of GRN, MMP‐10, and GPNMB were altered in Alzheimer's disease, preclinical Alzheimer's disease, and Parkinson's disease, respectively. These findings point to pathways to be explored for novel therapies. The novel finding that ventricular volume confounded pQTLs has implications for design of future studies of the genetic regulation of the CSF proteome.https://doi.org/10.15252/emmm.202216359biomarkerscerebrospinal fluidgenetic regulationMendelian randomizationpQTL
spellingShingle Oskar Hansson
Atul Kumar
Shorena Janelidze
Erik Stomrud
Philip S Insel
Kaj Blennow
Henrik Zetterberg
Eric Fauman
Åsa K Hedman
Michael W Nagle
Christopher D Whelan
Denis Baird
Anders Mälarstig
Niklas Mattsson‐Carlgren
The genetic regulation of protein expression in cerebrospinal fluid
EMBO Molecular Medicine
biomarkers
cerebrospinal fluid
genetic regulation
Mendelian randomization
pQTL
title The genetic regulation of protein expression in cerebrospinal fluid
title_full The genetic regulation of protein expression in cerebrospinal fluid
title_fullStr The genetic regulation of protein expression in cerebrospinal fluid
title_full_unstemmed The genetic regulation of protein expression in cerebrospinal fluid
title_short The genetic regulation of protein expression in cerebrospinal fluid
title_sort genetic regulation of protein expression in cerebrospinal fluid
topic biomarkers
cerebrospinal fluid
genetic regulation
Mendelian randomization
pQTL
url https://doi.org/10.15252/emmm.202216359
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