The Impact of Immunosenescence on Pulmonary Disease

The global population is aging with significant gains in life expectancy particularly in the developed world. Consequently, greater focus on understanding the processes that underlie physiological aging has occurred. Key facets of advancing age include genomic instability, telomere shortening, epige...

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Main Authors: Michelle A. Murray, Sanjay H. Chotirmall
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2015/692546
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author Michelle A. Murray
Sanjay H. Chotirmall
author_facet Michelle A. Murray
Sanjay H. Chotirmall
author_sort Michelle A. Murray
collection DOAJ
description The global population is aging with significant gains in life expectancy particularly in the developed world. Consequently, greater focus on understanding the processes that underlie physiological aging has occurred. Key facets of advancing age include genomic instability, telomere shortening, epigenetic changes, and declines in immune function termed immunosenescence. Immunosenescence and its associated chronic low grade systemic “inflamm-aging” contribute to the development and progression of pulmonary disease in older individuals. These physiological processes predispose to pulmonary infection and confer specific and unique clinical phenotypes observed in chronic respiratory disease including late-onset asthma, chronic obstructive pulmonary disease, and pulmonary fibrosis. Emerging concepts of the gut and airway microbiome further complicate the interrelationship between host and microorganism particularly from an immunological perspective and especially so in the setting of immunosenescence. This review focuses on our current understanding of the aging process, immunosenescence, and how it can potentially impact on various pulmonary diseases and the human microbiome.
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spelling doaj-art-3eb27e7ee2b34bd087b1d8230652430c2025-08-20T02:08:24ZengWileyMediators of Inflammation0962-93511466-18612015-01-01201510.1155/2015/692546692546The Impact of Immunosenescence on Pulmonary DiseaseMichelle A. Murray0Sanjay H. Chotirmall1Department of Respiratory Medicine, Mater Misericordiae Hospital, Eccles Street, Dublin 7, IrelandLee Kong Chian School of Medicine, Nanyang Technological University, 308232, SingaporeThe global population is aging with significant gains in life expectancy particularly in the developed world. Consequently, greater focus on understanding the processes that underlie physiological aging has occurred. Key facets of advancing age include genomic instability, telomere shortening, epigenetic changes, and declines in immune function termed immunosenescence. Immunosenescence and its associated chronic low grade systemic “inflamm-aging” contribute to the development and progression of pulmonary disease in older individuals. These physiological processes predispose to pulmonary infection and confer specific and unique clinical phenotypes observed in chronic respiratory disease including late-onset asthma, chronic obstructive pulmonary disease, and pulmonary fibrosis. Emerging concepts of the gut and airway microbiome further complicate the interrelationship between host and microorganism particularly from an immunological perspective and especially so in the setting of immunosenescence. This review focuses on our current understanding of the aging process, immunosenescence, and how it can potentially impact on various pulmonary diseases and the human microbiome.http://dx.doi.org/10.1155/2015/692546
spellingShingle Michelle A. Murray
Sanjay H. Chotirmall
The Impact of Immunosenescence on Pulmonary Disease
Mediators of Inflammation
title The Impact of Immunosenescence on Pulmonary Disease
title_full The Impact of Immunosenescence on Pulmonary Disease
title_fullStr The Impact of Immunosenescence on Pulmonary Disease
title_full_unstemmed The Impact of Immunosenescence on Pulmonary Disease
title_short The Impact of Immunosenescence on Pulmonary Disease
title_sort impact of immunosenescence on pulmonary disease
url http://dx.doi.org/10.1155/2015/692546
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