Lipopolysaccharide Animal Models for Parkinson's Disease

Lipopolysaccharide (LPS), an endotoxin from Gram-negative bacteria, acts as a potent stimulator of microglia and has been used to study the inflammatory process in the pathogenesis of Parkinson's disease (PD) and anti-inflammatory therapy for PD treatment. Here, we review the growing body of li...

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Main Authors: Mei Liu, Guoying Bing
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:Parkinson's Disease
Online Access:http://dx.doi.org/10.4061/2011/327089
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author Mei Liu
Guoying Bing
author_facet Mei Liu
Guoying Bing
author_sort Mei Liu
collection DOAJ
description Lipopolysaccharide (LPS), an endotoxin from Gram-negative bacteria, acts as a potent stimulator of microglia and has been used to study the inflammatory process in the pathogenesis of Parkinson's disease (PD) and anti-inflammatory therapy for PD treatment. Here, we review the growing body of literature on both in vitro and in vivo LPS PD models. Primary cell cultures from mesencephalic tissue were exposed to LPS in vitro; LPS was stereotaxically injected into the substantia nigra, striatum, or globus pallidus of brain or injected into the peritoneal cavity of the animal in vivo. In conclusion, the LPS PD models are summarized as (1) local and direct LPS treatment and (2) systemic LPS treatment. Mechanisms underlying the PD models are investigated and indicated that LPS induces microglial activation to release a variety of neurotoxic factors, and damaged neurons may trigger reactive microgliosis, which lead to progressive dopaminergic neurodegeneration.
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spelling doaj-art-3e8358d8b6c5438f9d969a8fd605f4582025-08-20T02:08:20ZengWileyParkinson's Disease2042-00802011-01-01201110.4061/2011/327089327089Lipopolysaccharide Animal Models for Parkinson's DiseaseMei Liu0Guoying Bing1Department of Anatomy and Neurobiology, College of Medicine, University of Kentucky, Lexington, KY 40536, USADepartment of Anatomy and Neurobiology, College of Medicine, University of Kentucky, Lexington, KY 40536, USALipopolysaccharide (LPS), an endotoxin from Gram-negative bacteria, acts as a potent stimulator of microglia and has been used to study the inflammatory process in the pathogenesis of Parkinson's disease (PD) and anti-inflammatory therapy for PD treatment. Here, we review the growing body of literature on both in vitro and in vivo LPS PD models. Primary cell cultures from mesencephalic tissue were exposed to LPS in vitro; LPS was stereotaxically injected into the substantia nigra, striatum, or globus pallidus of brain or injected into the peritoneal cavity of the animal in vivo. In conclusion, the LPS PD models are summarized as (1) local and direct LPS treatment and (2) systemic LPS treatment. Mechanisms underlying the PD models are investigated and indicated that LPS induces microglial activation to release a variety of neurotoxic factors, and damaged neurons may trigger reactive microgliosis, which lead to progressive dopaminergic neurodegeneration.http://dx.doi.org/10.4061/2011/327089
spellingShingle Mei Liu
Guoying Bing
Lipopolysaccharide Animal Models for Parkinson's Disease
Parkinson's Disease
title Lipopolysaccharide Animal Models for Parkinson's Disease
title_full Lipopolysaccharide Animal Models for Parkinson's Disease
title_fullStr Lipopolysaccharide Animal Models for Parkinson's Disease
title_full_unstemmed Lipopolysaccharide Animal Models for Parkinson's Disease
title_short Lipopolysaccharide Animal Models for Parkinson's Disease
title_sort lipopolysaccharide animal models for parkinson s disease
url http://dx.doi.org/10.4061/2011/327089
work_keys_str_mv AT meiliu lipopolysaccharideanimalmodelsforparkinsonsdisease
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