High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea

Anthracnose crown rot caused by Colletotrichum spp. is one of the most important diseases of strawberry (Fragaria × ananassa) in Korea. Single spore isolates of 72 Colletotrichum species collected in 2018, 2020, and 2023 from different locations in Chungbuk, Chungnam, Gyeongbuk, Gyeongnam, and Jeoll...

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Main Authors: Abdullahi Abdulkareem, Myeong Hyeon Nam, Subin Park, Young Ju Nam, Heung Tae Kim
Format: Article
Language:English
Published: Hanrimwon Publishing Company 2024-12-01
Series:Research in Plant Disease
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Online Access:http://www.online-rpd.org/upload/pdf/RPD-2024-30-4-372.pdf
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author Abdullahi Abdulkareem
Myeong Hyeon Nam
Subin Park
Young Ju Nam
Heung Tae Kim
author_facet Abdullahi Abdulkareem
Myeong Hyeon Nam
Subin Park
Young Ju Nam
Heung Tae Kim
author_sort Abdullahi Abdulkareem
collection DOAJ
description Anthracnose crown rot caused by Colletotrichum spp. is one of the most important diseases of strawberry (Fragaria × ananassa) in Korea. Single spore isolates of 72 Colletotrichum species collected in 2018, 2020, and 2023 from different locations in Chungbuk, Chungnam, Gyeongbuk, Gyeongnam, and Jeolla regions were characterized using a multi-locus sequence typing (MLST) of four genetic loci (GAPDH, ACT, TUB2, and ITS). Isolates were identified as C. fructicola (n=64) and C. siamense (n=8). Quinone outside inhibitors (QoIs) active ingredient pyraclostrobin is utilized to manage anthracnose crown rot in the field. Sensitivity to pyraclostrobin were assessed on potato dextrose agar using a mycelial growth inhibition assay at 20, 4, 0.8, 0.16, and 0.032 µg/ml fungicide concentrations. Our results demonstrated that all the 72 Colletotrichum spp. isolates were not inhibited by pyraclostrobin at all the tested fungicide concentrations, with EC50 ranging between 13 and >100 µg/ml. Sequences of the partial cytochrome b (cyt b) gene flanking the mutation site revealed the presence of the G143A mutation in all the isolates. No other site modifications were detected in the cyt b gene. Cross resistance assays with 10 selected isolates involving EC50 values of pyraclostrobin, mandestrobin, trifloxystrobin, and kresoxim-methyl showed positive correlations among these fungicides. These findings suggest a high risk of resistance development associated with using QoI fungicides to control the anthracnose crown rot of strawberries in Korea.
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spelling doaj-art-3d163eff0c4f41e386645d06dfd48bf52025-08-20T03:04:38ZengHanrimwon Publishing CompanyResearch in Plant Disease1598-22622233-91912024-12-0130437238010.5423/RPD.2024.30.4.3721870High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in KoreaAbdullahi Abdulkareem0Myeong Hyeon Nam1Subin Park2Young Ju Nam3Heung Tae Kim4Department of Plant Medicine, College of Agriculture, Life & Environment Sciences, Chungbuk National University, Cheongju 28644, KoreaStrawberry Research Institute, Chungnam ARES, Nonsan 32914, KoreaGlobal Agro-Consulting Corporation, Suwon 16614, KoreaGlobal Agro-Consulting Corporation, Suwon 16614, KoreaDepartment of Plant Medicine, College of Agriculture, Life & Environment Sciences, Chungbuk National University, Cheongju 28644, KoreaAnthracnose crown rot caused by Colletotrichum spp. is one of the most important diseases of strawberry (Fragaria × ananassa) in Korea. Single spore isolates of 72 Colletotrichum species collected in 2018, 2020, and 2023 from different locations in Chungbuk, Chungnam, Gyeongbuk, Gyeongnam, and Jeolla regions were characterized using a multi-locus sequence typing (MLST) of four genetic loci (GAPDH, ACT, TUB2, and ITS). Isolates were identified as C. fructicola (n=64) and C. siamense (n=8). Quinone outside inhibitors (QoIs) active ingredient pyraclostrobin is utilized to manage anthracnose crown rot in the field. Sensitivity to pyraclostrobin were assessed on potato dextrose agar using a mycelial growth inhibition assay at 20, 4, 0.8, 0.16, and 0.032 µg/ml fungicide concentrations. Our results demonstrated that all the 72 Colletotrichum spp. isolates were not inhibited by pyraclostrobin at all the tested fungicide concentrations, with EC50 ranging between 13 and >100 µg/ml. Sequences of the partial cytochrome b (cyt b) gene flanking the mutation site revealed the presence of the G143A mutation in all the isolates. No other site modifications were detected in the cyt b gene. Cross resistance assays with 10 selected isolates involving EC50 values of pyraclostrobin, mandestrobin, trifloxystrobin, and kresoxim-methyl showed positive correlations among these fungicides. These findings suggest a high risk of resistance development associated with using QoI fungicides to control the anthracnose crown rot of strawberries in Korea.http://www.online-rpd.org/upload/pdf/RPD-2024-30-4-372.pdfcolletotrichum spp.cyt b geneqoi fungicidesstrawberry anthracnose
spellingShingle Abdullahi Abdulkareem
Myeong Hyeon Nam
Subin Park
Young Ju Nam
Heung Tae Kim
High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
Research in Plant Disease
colletotrichum spp.
cyt b gene
qoi fungicides
strawberry anthracnose
title High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
title_full High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
title_fullStr High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
title_full_unstemmed High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
title_short High Quinone Outside Inhibitor (QoI) Resistance Risk in Colletotrichum Species Causing Strawberry Crown Rot in Korea
title_sort high quinone outside inhibitor qoi resistance risk in colletotrichum species causing strawberry crown rot in korea
topic colletotrichum spp.
cyt b gene
qoi fungicides
strawberry anthracnose
url http://www.online-rpd.org/upload/pdf/RPD-2024-30-4-372.pdf
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