Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells

We investigated the effect of ferulic acid (FA) and isoferulic acid (IFA), which are the main active components of the rhizoma of Cimicifuga heracleifolia (CH), an anti-inflammatory drug used frequently in Japanese traditional medicine, on the production of macrophage inflammatory protein-2 (MIR-2)...

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Main Authors: S. Sakai, H. Kawamata, T. Kogure, N. Mantani, K. Terasawa, M. Umatake, H. Ochiai
Format: Article
Language:English
Published: Wiley 1999-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1080/09629359990513
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author S. Sakai
H. Kawamata
T. Kogure
N. Mantani
K. Terasawa
M. Umatake
H. Ochiai
author_facet S. Sakai
H. Kawamata
T. Kogure
N. Mantani
K. Terasawa
M. Umatake
H. Ochiai
author_sort S. Sakai
collection DOAJ
description We investigated the effect of ferulic acid (FA) and isoferulic acid (IFA), which are the main active components of the rhizoma of Cimicifuga heracleifolia (CH), an anti-inflammatory drug used frequently in Japanese traditional medicine, on the production of macrophage inflammatory protein-2 (MIR-2) in a murine macrophage cell line, RAW264.7, in response to respiratory syncytial virus (RSV) infection. Following the exposure of cells to RSV for 20 h, the MIP-2 level in condition medium was increased to about 20 ng/ml, although this level in mock-infected cells was negligible. In the presence of either FA or IFA, RSV-infected cells reduced MIP-2 production in a dose-dependent manner. These data suggest that FA and IFA might be responsible, at least in part, for the anti-inflammatory drug effect of CH extract through the inhibition of MIP-2 production.
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institution OA Journals
issn 0962-9351
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language English
publishDate 1999-01-01
publisher Wiley
record_format Article
series Mediators of Inflammation
spelling doaj-art-3c89a2ce92eb4b92b754dc0df2dd76bf2025-08-20T02:39:08ZengWileyMediators of Inflammation0962-93511466-18611999-01-018317317510.1080/09629359990513Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 CellsS. Sakai0H. Kawamata1T. Kogure2N. Mantani3K. Terasawa4M. Umatake5H. Ochiai6Department of Japanese Oriental Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Japanese Oriental Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Japanese Oriental Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Japanese Oriental Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Japanese Oriental Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Human Sciences, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanDepartment of Human Sciences, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani 2630, Toyama 930– 0194, JapanWe investigated the effect of ferulic acid (FA) and isoferulic acid (IFA), which are the main active components of the rhizoma of Cimicifuga heracleifolia (CH), an anti-inflammatory drug used frequently in Japanese traditional medicine, on the production of macrophage inflammatory protein-2 (MIR-2) in a murine macrophage cell line, RAW264.7, in response to respiratory syncytial virus (RSV) infection. Following the exposure of cells to RSV for 20 h, the MIP-2 level in condition medium was increased to about 20 ng/ml, although this level in mock-infected cells was negligible. In the presence of either FA or IFA, RSV-infected cells reduced MIP-2 production in a dose-dependent manner. These data suggest that FA and IFA might be responsible, at least in part, for the anti-inflammatory drug effect of CH extract through the inhibition of MIP-2 production.http://dx.doi.org/10.1080/09629359990513
spellingShingle S. Sakai
H. Kawamata
T. Kogure
N. Mantani
K. Terasawa
M. Umatake
H. Ochiai
Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
Mediators of Inflammation
title Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
title_full Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
title_fullStr Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
title_full_unstemmed Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
title_short Inhibitory Effect of Ferulic Acid and Isoferulic Acid on the Production of Macrophage Inflammatory Protein-2 in Response to Respiratory Syncytial Virus Infection in RAW264.7 Cells
title_sort inhibitory effect of ferulic acid and isoferulic acid on the production of macrophage inflammatory protein 2 in response to respiratory syncytial virus infection in raw264 7 cells
url http://dx.doi.org/10.1080/09629359990513
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