Association between circulatory complement activation and hypertensive renal damage: a case–control study

Objective The aim of this study was to investigate the potential importance of complement system activation, with particular emphasis on the complement alternative pathway (AP), in the pathogenesis of hypertensive renal damage.Methods Serum complement C3, complement Factor H (CFH) and AP activation...

Full description

Saved in:
Bibliographic Details
Main Authors: Zhongli Wang, Tingting Zhang, Xinyu Wang, Jianlong Zhai, Lili He, Sai Ma, Qingjuan Zuo, Guorui Zhang, Yifang Guo
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Renal Failure
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/0886022X.2024.2365396
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849763039404883968
author Zhongli Wang
Tingting Zhang
Xinyu Wang
Jianlong Zhai
Lili He
Sai Ma
Qingjuan Zuo
Guorui Zhang
Yifang Guo
author_facet Zhongli Wang
Tingting Zhang
Xinyu Wang
Jianlong Zhai
Lili He
Sai Ma
Qingjuan Zuo
Guorui Zhang
Yifang Guo
author_sort Zhongli Wang
collection DOAJ
description Objective The aim of this study was to investigate the potential importance of complement system activation, with particular emphasis on the complement alternative pathway (AP), in the pathogenesis of hypertensive renal damage.Methods Serum complement C3, complement Factor H (CFH) and AP activation were assessed in 66 participants with established essential hypertension with renal damage (RD). Fifty-nine patients with age- and sex-matched essential hypertension without renal damage (NRD) and 58 healthy participants (normal) were selected.Results Our study revealed that C3 and AP50 continuously increased from normal to NRD to RD (p < 0.05, respectively), while CFH was significantly lower than that in NRD and healthy participants (p < 0.05, respectively). After multifactorial logistic regression analysis corrected for confounders, elevated serum C3 (p = 0.001) and decreased CFH (p < 0.001) were found to be independent risk factors for hypertension in healthy participants; elevated serum C3 (p = 0.034), elevated AP50 (p < 0.001), decreased CFH (p < 0.001), increased age (p = 0.011) and increased BMI (p = 0.013) were found to be independent risk factors for the progression of hypertension to hypertensive renal damage; elevated serum C3 (p = 0.017), elevated AP50 (p = 0.023), decreased CFH (p = 0.005) and increased age (p = 0.041) were found to be independent risk factors for the development of hypertensive renal damage in healthy participants.Conclusion Abnormal activation of complement, particularly complement AP, may be a risk factor for the development and progression of hypertensive renal damage.
format Article
id doaj-art-3ae8cd875aaa4a5793f5b4afb573a1ab
institution DOAJ
issn 0886-022X
1525-6049
language English
publishDate 2024-12-01
publisher Taylor & Francis Group
record_format Article
series Renal Failure
spelling doaj-art-3ae8cd875aaa4a5793f5b4afb573a1ab2025-08-20T03:05:33ZengTaylor & Francis GroupRenal Failure0886-022X1525-60492024-12-0146210.1080/0886022X.2024.2365396Association between circulatory complement activation and hypertensive renal damage: a case–control studyZhongli Wang0Tingting Zhang1Xinyu Wang2Jianlong Zhai3Lili He4Sai Ma5Qingjuan Zuo6Guorui Zhang7Yifang Guo8Department of Physical Examination Center, Hebei General Hospital, Shijiazhuang, Hebei, ChinaDepartment of Geriatric Cardiology, Hebei General Hospital, Shijiazhuang, Hebei, ChinaCollege of Postgraduate, Hebei North University, Zhangjiakou, Hebei, ChinaDepartment of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei, ChinaDepartment of Geriatric Cardiology, Hebei General Hospital, Shijiazhuang, Hebei, ChinaDepartment of Internal Medicine, Hebei General Hospital, Shijiazhuang, Hebei, ChinaDepartment of Geriatric Cardiology, Hebei General Hospital, Shijiazhuang, Hebei, ChinaDepartment of Cardiology, The Third Hospital of Shijiazhuang City Affiliated to Hebei Medical University, Shijiazhuang, Hebei, ChinaDepartment of Geriatric Cardiology, Hebei General Hospital, Shijiazhuang, Hebei, ChinaObjective The aim of this study was to investigate the potential importance of complement system activation, with particular emphasis on the complement alternative pathway (AP), in the pathogenesis of hypertensive renal damage.Methods Serum complement C3, complement Factor H (CFH) and AP activation were assessed in 66 participants with established essential hypertension with renal damage (RD). Fifty-nine patients with age- and sex-matched essential hypertension without renal damage (NRD) and 58 healthy participants (normal) were selected.Results Our study revealed that C3 and AP50 continuously increased from normal to NRD to RD (p < 0.05, respectively), while CFH was significantly lower than that in NRD and healthy participants (p < 0.05, respectively). After multifactorial logistic regression analysis corrected for confounders, elevated serum C3 (p = 0.001) and decreased CFH (p < 0.001) were found to be independent risk factors for hypertension in healthy participants; elevated serum C3 (p = 0.034), elevated AP50 (p < 0.001), decreased CFH (p < 0.001), increased age (p = 0.011) and increased BMI (p = 0.013) were found to be independent risk factors for the progression of hypertension to hypertensive renal damage; elevated serum C3 (p = 0.017), elevated AP50 (p = 0.023), decreased CFH (p = 0.005) and increased age (p = 0.041) were found to be independent risk factors for the development of hypertensive renal damage in healthy participants.Conclusion Abnormal activation of complement, particularly complement AP, may be a risk factor for the development and progression of hypertensive renal damage.https://www.tandfonline.com/doi/10.1080/0886022X.2024.2365396Complement systemhypertensive renal damagecomplement C3alternative complement pathway (AP)CFH
spellingShingle Zhongli Wang
Tingting Zhang
Xinyu Wang
Jianlong Zhai
Lili He
Sai Ma
Qingjuan Zuo
Guorui Zhang
Yifang Guo
Association between circulatory complement activation and hypertensive renal damage: a case–control study
Renal Failure
Complement system
hypertensive renal damage
complement C3
alternative complement pathway (AP)
CFH
title Association between circulatory complement activation and hypertensive renal damage: a case–control study
title_full Association between circulatory complement activation and hypertensive renal damage: a case–control study
title_fullStr Association between circulatory complement activation and hypertensive renal damage: a case–control study
title_full_unstemmed Association between circulatory complement activation and hypertensive renal damage: a case–control study
title_short Association between circulatory complement activation and hypertensive renal damage: a case–control study
title_sort association between circulatory complement activation and hypertensive renal damage a case control study
topic Complement system
hypertensive renal damage
complement C3
alternative complement pathway (AP)
CFH
url https://www.tandfonline.com/doi/10.1080/0886022X.2024.2365396
work_keys_str_mv AT zhongliwang associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT tingtingzhang associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT xinyuwang associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT jianlongzhai associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT lilihe associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT saima associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT qingjuanzuo associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT guoruizhang associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy
AT yifangguo associationbetweencirculatorycomplementactivationandhypertensiverenaldamageacasecontrolstudy