Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies

Myocardial infarction (MI), which is characterized by high morbidity and mortality, is a serious threat to human life and health, and timely reperfusion therapy to save ischemic myocardium is currently the most effective intervention. Although reperfusion therapy effectively restores coronary blood...

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Main Authors: Xiaoyu Xuan, Jilin Fan, Jingyi Zhang, Ming Ren, Limin Feng
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Immunology
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Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1558484/full
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author Xiaoyu Xuan
Jilin Fan
Jingyi Zhang
Ming Ren
Limin Feng
Limin Feng
author_facet Xiaoyu Xuan
Jilin Fan
Jingyi Zhang
Ming Ren
Limin Feng
Limin Feng
author_sort Xiaoyu Xuan
collection DOAJ
description Myocardial infarction (MI), which is characterized by high morbidity and mortality, is a serious threat to human life and health, and timely reperfusion therapy to save ischemic myocardium is currently the most effective intervention. Although reperfusion therapy effectively restores coronary blood flow and maximally limits the infarct size, it triggers additional cell death and tissue damage, which is known as myocardial ischemia/reperfusion injury (MIRI). Multiple immune cells are present in the reperfusion area, executing specific functions and engaging in crosstalk during diverse stages, constituting a complex immune microenvironment involved in tissue repair and regeneration after MIRI. Immunotherapy brings new hope for treating ischemic heart disease by modulating the immune microenvironment. In this paper, we explore the regulatory roles of various immune cells during MIRI and the close relationship between different cell deaths and the immune microenvironment. In addition, we present the current status of research on targeting the immune system to intervene in MIRI, with the expectation of providing a basis for achieving clinical translation.
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issn 1664-3224
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publishDate 2025-05-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Immunology
spelling doaj-art-3833a5ed64a846d28aab7d582fa39efd2025-08-20T02:27:13ZengFrontiers Media S.A.Frontiers in Immunology1664-32242025-05-011610.3389/fimmu.2025.15584841558484Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategiesXiaoyu Xuan0Jilin Fan1Jingyi Zhang2Ming Ren3Limin Feng4Limin Feng5College of Traditional Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaDepartment of Rehabilitation, The Affiliated Hospital of Binzhou Medical University, Binzhou, Shandong, ChinaDepartment of Traditional Chinese Medicine, The Second Affiliated Hospital of Shandong First Medical University, Shandong, Taian, ChinaBaokang Hospital Affiliated to Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaThe Second Affiliated Hospital of Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaTianjin Binhai New Area Traditional Chinese Medicine Hospital, Tianjin, ChinaMyocardial infarction (MI), which is characterized by high morbidity and mortality, is a serious threat to human life and health, and timely reperfusion therapy to save ischemic myocardium is currently the most effective intervention. Although reperfusion therapy effectively restores coronary blood flow and maximally limits the infarct size, it triggers additional cell death and tissue damage, which is known as myocardial ischemia/reperfusion injury (MIRI). Multiple immune cells are present in the reperfusion area, executing specific functions and engaging in crosstalk during diverse stages, constituting a complex immune microenvironment involved in tissue repair and regeneration after MIRI. Immunotherapy brings new hope for treating ischemic heart disease by modulating the immune microenvironment. In this paper, we explore the regulatory roles of various immune cells during MIRI and the close relationship between different cell deaths and the immune microenvironment. In addition, we present the current status of research on targeting the immune system to intervene in MIRI, with the expectation of providing a basis for achieving clinical translation.https://www.frontiersin.org/articles/10.3389/fimmu.2025.1558484/fullmyocardial ischemia/reperfusion injurymyocardial infarctionimmune cellcell deathimmunotherapy
spellingShingle Xiaoyu Xuan
Jilin Fan
Jingyi Zhang
Ming Ren
Limin Feng
Limin Feng
Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
Frontiers in Immunology
myocardial ischemia/reperfusion injury
myocardial infarction
immune cell
cell death
immunotherapy
title Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
title_full Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
title_fullStr Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
title_full_unstemmed Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
title_short Immune in myocardial ischemia/reperfusion injury: potential mechanisms and therapeutic strategies
title_sort immune in myocardial ischemia reperfusion injury potential mechanisms and therapeutic strategies
topic myocardial ischemia/reperfusion injury
myocardial infarction
immune cell
cell death
immunotherapy
url https://www.frontiersin.org/articles/10.3389/fimmu.2025.1558484/full
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AT jingyizhang immuneinmyocardialischemiareperfusioninjurypotentialmechanismsandtherapeuticstrategies
AT mingren immuneinmyocardialischemiareperfusioninjurypotentialmechanismsandtherapeuticstrategies
AT liminfeng immuneinmyocardialischemiareperfusioninjurypotentialmechanismsandtherapeuticstrategies
AT liminfeng immuneinmyocardialischemiareperfusioninjurypotentialmechanismsandtherapeuticstrategies