<i>FoxO1</i> Mediated by H3K27me3 Inhibits Porcine Follicular Development by Regulating the Transcription of <i>CYP1A1</i>

It is well known that the function of granulosa cells (GCs) is closely related to follicular development, and <i>FoxO1</i> and histone methylation have been implicated in follicular development. However, the specific mechanisms by which <i>FoxO1</i> and histone methylation re...

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Main Authors: Zhi Zhou, Yuanyuan Lv, Liying Li, Xiaolong Yuan, Xiaofeng Zhou, Jiaqi Li
Format: Article
Language:English
Published: MDPI AG 2024-12-01
Series:Animals
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Online Access:https://www.mdpi.com/2076-2615/14/23/3514
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Summary:It is well known that the function of granulosa cells (GCs) is closely related to follicular development, and <i>FoxO1</i> and histone methylation have been implicated in follicular development. However, the specific mechanisms by which <i>FoxO1</i> and histone methylation regulate follicular development are still largely unknown. To explore the specific mechanism of <i>FoxO1</i> in regulating follicular development, in this study, we showed that the expression of <i>FoxO1</i> in immature ovaries and small follicles was significantly higher than in mature ovaries and large follicles of sows, respectively. <i>FoxO1</i> was found to inhibit the secretion of testosterone and proliferation of porcine GCs and promote the secretion of progesterone and apoptosis of porcine GCs. Furthermore, H3K27me3, as a transcriptional inhibitor, can inhibit the transcription of <i>FoxO1</i>. <i>FoxO1</i> could promote the transcription of <i>CYP1A1</i>, and <i>CYP1A1</i> was found to inhibit the proliferation and facilitate the ferroptosis of porcine GCs. Collectively, our results revealed that the H3K27me3-<i>FoxO1</i>-<i>CYP1A1</i> pathway might participate in follicular development, and these findings could provide potential targets for improving follicular development in sows.
ISSN:2076-2615