Accelerated Hypertension after Venlafaxine Usage

Venlafaxine is the first antidepressant that acts via inhibiting serotonin and noradrenaline reuptake. Hypertension is observed in doses exceeding 300 mg/day and is the most feared complication. We report a patient with accelerated hypertension after venlafaxine use observed at a dose of 150 mg/day....

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Main Authors: Yüksel Kıvrak, Tolga Sinan Güvenç, Nurcihan Akbulut, İbrahim Yağcı, Gülşen Çığşar, Süleyman Gündüz, Bahattin Balcı
Format: Article
Language:English
Published: Wiley 2014-01-01
Series:Case Reports in Psychiatry
Online Access:http://dx.doi.org/10.1155/2014/659715
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author Yüksel Kıvrak
Tolga Sinan Güvenç
Nurcihan Akbulut
İbrahim Yağcı
Gülşen Çığşar
Süleyman Gündüz
Bahattin Balcı
author_facet Yüksel Kıvrak
Tolga Sinan Güvenç
Nurcihan Akbulut
İbrahim Yağcı
Gülşen Çığşar
Süleyman Gündüz
Bahattin Balcı
author_sort Yüksel Kıvrak
collection DOAJ
description Venlafaxine is the first antidepressant that acts via inhibiting serotonin and noradrenaline reuptake. Hypertension is observed in doses exceeding 300 mg/day and is the most feared complication. We report a patient with accelerated hypertension after venlafaxine use observed at a dose of 150 mg/day. A 23-year-old patient with symptoms of insomnia, depression, anhedonia, fatigue admitted our clinic. Venlafaxine at a dose of 75 mg/day was initiated after he was diagnosed with major depressive disorder. After 5 months, venlafaxine dose was uptitrated to 150 mg/day due to inadequate response to drug. After using venlafaxine for ten months at the dose of 150 mg/day, he admitted our clinic with headache and epistaxis. He was hospitalized after his blood pressure was measured as 210/170 mmHg. No secondary causes for hypertension were found, and venlafaxine treatment was considered possible etiologic factor. After stopping venlafaxine treatment, his blood pressure was reverted back to normal limits. While mild elevation of blood pressure could be observed after venlafaxine treatment, this case shows that accelerated hypertension with a diastolic blood pressure rise above 120 mmHg could be observed at relatively low doses of venlafaxine. Close monitoring of blood pressure is necessary after initiation of treatment, as accelerated hypertension could cause endorgan damage with potentially catastrophic results.
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publishDate 2014-01-01
publisher Wiley
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series Case Reports in Psychiatry
spelling doaj-art-3173daee5a55402991c10f1570898ee32025-08-20T02:38:54ZengWileyCase Reports in Psychiatry2090-682X2090-68382014-01-01201410.1155/2014/659715659715Accelerated Hypertension after Venlafaxine UsageYüksel Kıvrak0Tolga Sinan Güvenç1Nurcihan Akbulut2İbrahim Yağcı3Gülşen Çığşar4Süleyman Gündüz5Bahattin Balcı6Department of Psychiatry, Kafkas University School of Medicine, 36000 Kars, TurkeyDepartment of Cardiology, Kafkas University School of Medicine, Kars, TurkeyDepartment of Psychiatry, Kafkas University School of Medicine, 36000 Kars, TurkeyDepartment of Psychiatry, Kafkas University School of Medicine, 36000 Kars, TurkeyDepartment of Emergency, Kafkas University School of Medicine, Kars, TurkeyDepartment of Psychiatry, Kars State Hospital, Kars, TurkeyDepartment of Cardiology, Kafkas University School of Medicine, Kars, TurkeyVenlafaxine is the first antidepressant that acts via inhibiting serotonin and noradrenaline reuptake. Hypertension is observed in doses exceeding 300 mg/day and is the most feared complication. We report a patient with accelerated hypertension after venlafaxine use observed at a dose of 150 mg/day. A 23-year-old patient with symptoms of insomnia, depression, anhedonia, fatigue admitted our clinic. Venlafaxine at a dose of 75 mg/day was initiated after he was diagnosed with major depressive disorder. After 5 months, venlafaxine dose was uptitrated to 150 mg/day due to inadequate response to drug. After using venlafaxine for ten months at the dose of 150 mg/day, he admitted our clinic with headache and epistaxis. He was hospitalized after his blood pressure was measured as 210/170 mmHg. No secondary causes for hypertension were found, and venlafaxine treatment was considered possible etiologic factor. After stopping venlafaxine treatment, his blood pressure was reverted back to normal limits. While mild elevation of blood pressure could be observed after venlafaxine treatment, this case shows that accelerated hypertension with a diastolic blood pressure rise above 120 mmHg could be observed at relatively low doses of venlafaxine. Close monitoring of blood pressure is necessary after initiation of treatment, as accelerated hypertension could cause endorgan damage with potentially catastrophic results.http://dx.doi.org/10.1155/2014/659715
spellingShingle Yüksel Kıvrak
Tolga Sinan Güvenç
Nurcihan Akbulut
İbrahim Yağcı
Gülşen Çığşar
Süleyman Gündüz
Bahattin Balcı
Accelerated Hypertension after Venlafaxine Usage
Case Reports in Psychiatry
title Accelerated Hypertension after Venlafaxine Usage
title_full Accelerated Hypertension after Venlafaxine Usage
title_fullStr Accelerated Hypertension after Venlafaxine Usage
title_full_unstemmed Accelerated Hypertension after Venlafaxine Usage
title_short Accelerated Hypertension after Venlafaxine Usage
title_sort accelerated hypertension after venlafaxine usage
url http://dx.doi.org/10.1155/2014/659715
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AT nurcihanakbulut acceleratedhypertensionaftervenlafaxineusage
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AT gulsencıgsar acceleratedhypertensionaftervenlafaxineusage
AT suleymangunduz acceleratedhypertensionaftervenlafaxineusage
AT bahattinbalcı acceleratedhypertensionaftervenlafaxineusage