Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion

Intestinal ischemia/reperfusion (II/R) is a common and grave clinical event, with high morbidity and mortality which can cause cerebral dysfunctions. There are no ideal prevention and treatment measures yet. The present study aimed to determine whether muscle-derived factors can alleviate gut-associ...

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Main Authors: Yafang Tan, Guo Mu, Feixiang Wang, Xin Fan, Chengjie Yang, Zuan Shi, Yiping Bai, Bingqing Xie, Xuan Yu, Jianguo Feng, Jing Jia, Xiaobin Wang, Ye Chen, Jun Zhou
Format: Article
Language:English
Published: Elsevier 2025-07-01
Series:Redox Biology
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Online Access:http://www.sciencedirect.com/science/article/pii/S2213231725001958
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author Yafang Tan
Guo Mu
Feixiang Wang
Xin Fan
Chengjie Yang
Zuan Shi
Yiping Bai
Bingqing Xie
Xuan Yu
Jianguo Feng
Jing Jia
Xiaobin Wang
Ye Chen
Jun Zhou
author_facet Yafang Tan
Guo Mu
Feixiang Wang
Xin Fan
Chengjie Yang
Zuan Shi
Yiping Bai
Bingqing Xie
Xuan Yu
Jianguo Feng
Jing Jia
Xiaobin Wang
Ye Chen
Jun Zhou
author_sort Yafang Tan
collection DOAJ
description Intestinal ischemia/reperfusion (II/R) is a common and grave clinical event, with high morbidity and mortality which can cause cerebral dysfunctions. There are no ideal prevention and treatment measures yet. The present study aimed to determine whether muscle-derived factors can alleviate gut-associated cerebral dysfunctions (GACD) following II/R. We measured the tibialis anterior muscle thickness and irisin levels in patients with and without cognitive dysfunction following cardiopulmonary bypass surgery, calculating the correlation between irisin and cognitive impairment. We found that this protective effect is related to muscle-derived irisin. To elucidate the role of irisin in improving GACD, we knocked out FNDC5 to deplete endogenous irisin and supplemented exogenous irisin. Mechanistic insights into irisin's effects on GACD were investigated using in vivo and in vitro models, incorporating techniques such as transmission electron microscopy, protein docking analysis, gene overexpression, and western blotting. FNDC5/irisin deficiency aggravated cognitive impairments, the pro-inflammation microglia activation, oxidative injury, inflammatory response, neuronal apoptosis and ferroptosis, while recombinant FNDC5/irisin reversed the above changes leading to neurostructural and cognition recovery. Mechanistically, thioredoxin-interacting protein (TXNIP) was activated in the II/R-related neuropathology and was deteriorated in FNDC5/irisin knockout mice. Our results highlight the potential of FNDC5/irisin to slow GACD, providing new insights and potential therapeutic strategies for the prevention and treatment of GACD.
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spelling doaj-art-2fccc9fa4c7f4eeba03d101e7644b0332025-08-20T02:32:23ZengElsevierRedox Biology2213-23172025-07-018410368210.1016/j.redox.2025.103682Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusionYafang Tan0Guo Mu1Feixiang Wang2Xin Fan3Chengjie Yang4Zuan Shi5Yiping Bai6Bingqing Xie7Xuan Yu8Jianguo Feng9Jing Jia10Xiaobin Wang11Ye Chen12Jun Zhou13Department of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, China; Department of Anesthesiology, Biejing Anzhen Nanchong Hospital of Capital Medical University & Nanchong Central Hospital, Nanchong, 634700, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Department of Anesthesiology, Zigong Fourth People's Hospital, Zigong, 643000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Department of Anesthesiology, Biejing Anzhen Nanchong Hospital of Capital Medical University & Nanchong Central Hospital, Nanchong, 634700, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaLaboratory of Neurological Diseases and Brain Function, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Institute of Epigenetics and Brain Science, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, Zigong Fourth People's Hospital, Zigong, 643000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaDepartment of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, ChinaAnesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, China; Department of Traditional Chinese Medicine, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Corresponding author. Department of Traditional Chinese Medicine, The Affiliated Hospital, Southwest Medical University, Luzhou, China.Department of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou, 646000, China; Anesthesiology and Critical Care Medicine Key Laboratory of Luzhou, Southwest Medical University, Luzhou, 646000, China; Corresponding author. Department of Anesthesiology, The Affiliated Hospital, Southwest Medical University, Luzhou,646000, China.Intestinal ischemia/reperfusion (II/R) is a common and grave clinical event, with high morbidity and mortality which can cause cerebral dysfunctions. There are no ideal prevention and treatment measures yet. The present study aimed to determine whether muscle-derived factors can alleviate gut-associated cerebral dysfunctions (GACD) following II/R. We measured the tibialis anterior muscle thickness and irisin levels in patients with and without cognitive dysfunction following cardiopulmonary bypass surgery, calculating the correlation between irisin and cognitive impairment. We found that this protective effect is related to muscle-derived irisin. To elucidate the role of irisin in improving GACD, we knocked out FNDC5 to deplete endogenous irisin and supplemented exogenous irisin. Mechanistic insights into irisin's effects on GACD were investigated using in vivo and in vitro models, incorporating techniques such as transmission electron microscopy, protein docking analysis, gene overexpression, and western blotting. FNDC5/irisin deficiency aggravated cognitive impairments, the pro-inflammation microglia activation, oxidative injury, inflammatory response, neuronal apoptosis and ferroptosis, while recombinant FNDC5/irisin reversed the above changes leading to neurostructural and cognition recovery. Mechanistically, thioredoxin-interacting protein (TXNIP) was activated in the II/R-related neuropathology and was deteriorated in FNDC5/irisin knockout mice. Our results highlight the potential of FNDC5/irisin to slow GACD, providing new insights and potential therapeutic strategies for the prevention and treatment of GACD.http://www.sciencedirect.com/science/article/pii/S2213231725001958MuscleIrisinIntestinal ischemia reperfusionCerebral dysfunctionsMicroglia
spellingShingle Yafang Tan
Guo Mu
Feixiang Wang
Xin Fan
Chengjie Yang
Zuan Shi
Yiping Bai
Bingqing Xie
Xuan Yu
Jianguo Feng
Jing Jia
Xiaobin Wang
Ye Chen
Jun Zhou
Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
Redox Biology
Muscle
Irisin
Intestinal ischemia reperfusion
Cerebral dysfunctions
Microglia
title Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
title_full Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
title_fullStr Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
title_full_unstemmed Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
title_short Muscle-derived factor alleviated cognitive impairment caused by intestinal ischemia-reperfusion
title_sort muscle derived factor alleviated cognitive impairment caused by intestinal ischemia reperfusion
topic Muscle
Irisin
Intestinal ischemia reperfusion
Cerebral dysfunctions
Microglia
url http://www.sciencedirect.com/science/article/pii/S2213231725001958
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