Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma

Purpose: The tumor microenvironment (TME) in lymphoma is influenced by M2 macrophages. This research proposes an novel predictive model that leverages M2 macrophage-associated genes to categorize risk, forecast outcomes, and evaluate the immune profile in patients with newly diagnosed diffuse large...

Full description

Saved in:
Bibliographic Details
Main Authors: Baoping Guo, Ying Duan, Hong Cen
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:Heliyon
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S2405844024170387
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850053983683477504
author Baoping Guo
Ying Duan
Hong Cen
author_facet Baoping Guo
Ying Duan
Hong Cen
author_sort Baoping Guo
collection DOAJ
description Purpose: The tumor microenvironment (TME) in lymphoma is influenced by M2 macrophages. This research proposes an novel predictive model that leverages M2 macrophage-associated genes to categorize risk, forecast outcomes, and evaluate the immune profile in patients with newly diagnosed diffuse large B-cell lymphoma (DLBCL) undergoing R-CHOP therapy. Methods: Gene expression data and clinical information from DLBCL patients were retrieved from The Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases. Co-expressed genes linked to M2 macrophage in DLBCL were analyzed using CIBERSORT. Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analyses were conducted to explore associated signaling pathways. The M2 macrophage-related gene prognostic model was developed and validated using Cox and LASSO regression. Prognostic signature genes were verified by single-cell RNA-seq analysis. Results: 92 M2 macrophage-related genes were identified based on bulk-seq data. MS4A4A, CCL13, LTB, CCL23, CCL18, XKR4, IL22RA2, and FOLR2 were used to construct the risk model. AUC values for 1-, 3-, and 5-year survival were 0.74, 0.72, and 0.72, respectively. High-risk patients demonstrated elevated immune scores and poorer overall survival. The high-risk subgroup also exhibited greater sensitivity to both chemotherapeutic agents and immune checkpoint inhibitors. Conclusion: This study presents an accurate and reliable M2 macrophage-related risk model, enhancing understanding of distinct prognostic subsets in DLBCL. It offers potential novel drug options for future treatments.
format Article
id doaj-art-2f63e857992643fca4b681afe7f2484a
institution DOAJ
issn 2405-8440
language English
publishDate 2024-12-01
publisher Elsevier
record_format Article
series Heliyon
spelling doaj-art-2f63e857992643fca4b681afe7f2484a2025-08-20T02:52:24ZengElsevierHeliyon2405-84402024-12-011024e4100710.1016/j.heliyon.2024.e41007Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphomaBaoping Guo0Ying Duan1Hong Cen2Corresponding author. Department of Hematology, Guangxi Medical University Cancer Hospital, 71 Hedi Avenue, Nanning, Guangxi, 530021, China.; Department of Hematology, Guangxi Medical University Cancer Hospital, Nanning, Guangxi, 530021, ChinaDepartment of Hematology, Guangxi Medical University Cancer Hospital, Nanning, Guangxi, 530021, ChinaCorresponding author. Department of Hematology, Guangxi Medical University Cancer Hospital, 71 Hedi Avenue, Nanning, Guangxi, 530021, China.; Department of Hematology, Guangxi Medical University Cancer Hospital, Nanning, Guangxi, 530021, ChinaPurpose: The tumor microenvironment (TME) in lymphoma is influenced by M2 macrophages. This research proposes an novel predictive model that leverages M2 macrophage-associated genes to categorize risk, forecast outcomes, and evaluate the immune profile in patients with newly diagnosed diffuse large B-cell lymphoma (DLBCL) undergoing R-CHOP therapy. Methods: Gene expression data and clinical information from DLBCL patients were retrieved from The Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases. Co-expressed genes linked to M2 macrophage in DLBCL were analyzed using CIBERSORT. Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analyses were conducted to explore associated signaling pathways. The M2 macrophage-related gene prognostic model was developed and validated using Cox and LASSO regression. Prognostic signature genes were verified by single-cell RNA-seq analysis. Results: 92 M2 macrophage-related genes were identified based on bulk-seq data. MS4A4A, CCL13, LTB, CCL23, CCL18, XKR4, IL22RA2, and FOLR2 were used to construct the risk model. AUC values for 1-, 3-, and 5-year survival were 0.74, 0.72, and 0.72, respectively. High-risk patients demonstrated elevated immune scores and poorer overall survival. The high-risk subgroup also exhibited greater sensitivity to both chemotherapeutic agents and immune checkpoint inhibitors. Conclusion: This study presents an accurate and reliable M2 macrophage-related risk model, enhancing understanding of distinct prognostic subsets in DLBCL. It offers potential novel drug options for future treatments.http://www.sciencedirect.com/science/article/pii/S2405844024170387DLBCLImmuneM2 macrophagePrognostic modelTumor microenvironment
spellingShingle Baoping Guo
Ying Duan
Hong Cen
Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
Heliyon
DLBCL
Immune
M2 macrophage
Prognostic model
Tumor microenvironment
title Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
title_full Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
title_fullStr Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
title_full_unstemmed Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
title_short Prognostic model based on M2 macrophage-related signatures for predicting outcomes, enhancing risk stratification, and providing therapeutic insights in diffuse large B-cell lymphoma
title_sort prognostic model based on m2 macrophage related signatures for predicting outcomes enhancing risk stratification and providing therapeutic insights in diffuse large b cell lymphoma
topic DLBCL
Immune
M2 macrophage
Prognostic model
Tumor microenvironment
url http://www.sciencedirect.com/science/article/pii/S2405844024170387
work_keys_str_mv AT baopingguo prognosticmodelbasedonm2macrophagerelatedsignaturesforpredictingoutcomesenhancingriskstratificationandprovidingtherapeuticinsightsindiffuselargebcelllymphoma
AT yingduan prognosticmodelbasedonm2macrophagerelatedsignaturesforpredictingoutcomesenhancingriskstratificationandprovidingtherapeuticinsightsindiffuselargebcelllymphoma
AT hongcen prognosticmodelbasedonm2macrophagerelatedsignaturesforpredictingoutcomesenhancingriskstratificationandprovidingtherapeuticinsightsindiffuselargebcelllymphoma