Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy

Myocardial ischemia/reperfusion (I/R) injury seriously threats the health and life of patients with ischemia heart disease. Herein, we probed the potential influence of myosin 1b (myo1b) on hypoxia/reoxygenation- (H/R-) stimulated cardiomyocyte H9c2 cell apoptosis and autophagy. After H/R stimulatio...

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Main Authors: Jing Xu, Jin Huang, Xiaojie He, Mingshuang Hu, Shan Su, Ping Liu
Format: Article
Language:English
Published: Wiley 2022-01-01
Series:Analytical Cellular Pathology
Online Access:http://dx.doi.org/10.1155/2022/5187304
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author Jing Xu
Jin Huang
Xiaojie He
Mingshuang Hu
Shan Su
Ping Liu
author_facet Jing Xu
Jin Huang
Xiaojie He
Mingshuang Hu
Shan Su
Ping Liu
author_sort Jing Xu
collection DOAJ
description Myocardial ischemia/reperfusion (I/R) injury seriously threats the health and life of patients with ischemia heart disease. Herein, we probed the potential influence of myosin 1b (myo1b) on hypoxia/reoxygenation- (H/R-) stimulated cardiomyocyte H9c2 cell apoptosis and autophagy. After H/R stimulation, the myo1b mRNA level in H9c2 cells was tested via qRT-PCR. Myo1b overexpression plasmid (OE-myo1b) and small interfering RNA (siRNA) targeting myo1b (si-myo1b) were transfected into H9c2 cells to alter myo1b expression in H9c2 cells. Following H/R stimulation and/or OE-myo1b (or si-myo1b) transfection, H9c2 cell apoptosis, proliferation, and autophagy were detected, respectively. We found that H/R stimulation reduced the mRNA level of myo1b in H9c2 cells and resulted in H9c2 cell apoptosis, proliferation inhibition, and autophagy. Overexpression of myo1b reversed the H/R-resulted H9c2 cell apoptosis, proliferation inhibition, and autophagy. Silence of myo1b had opposite effects, which promoted H9c2 cell apoptosis, reduced cell proliferation, and accelerated cell autophagy. Taken together, Myo1b took part in the modulation of H/R-stimulated cardiomyocyte apoptosis and autophagy, which might be serve as a potential endogenous target for prevention and therapy of I/R injury.
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spelling doaj-art-2f54e45d75974292b084fb4c56fc3d622025-08-20T03:18:42ZengWileyAnalytical Cellular Pathology2210-71852022-01-01202210.1155/2022/5187304Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and AutophagyJing Xu0Jin Huang1Xiaojie He2Mingshuang Hu3Shan Su4Ping Liu5Clinical Nursing Teaching and Research SectionClinical Nursing Teaching and Research SectionNephrology LaboratoryClinical Nursing Teaching and Research SectionClinical Nursing Teaching and Research SectionClinical Nursing Teaching and Research SectionMyocardial ischemia/reperfusion (I/R) injury seriously threats the health and life of patients with ischemia heart disease. Herein, we probed the potential influence of myosin 1b (myo1b) on hypoxia/reoxygenation- (H/R-) stimulated cardiomyocyte H9c2 cell apoptosis and autophagy. After H/R stimulation, the myo1b mRNA level in H9c2 cells was tested via qRT-PCR. Myo1b overexpression plasmid (OE-myo1b) and small interfering RNA (siRNA) targeting myo1b (si-myo1b) were transfected into H9c2 cells to alter myo1b expression in H9c2 cells. Following H/R stimulation and/or OE-myo1b (or si-myo1b) transfection, H9c2 cell apoptosis, proliferation, and autophagy were detected, respectively. We found that H/R stimulation reduced the mRNA level of myo1b in H9c2 cells and resulted in H9c2 cell apoptosis, proliferation inhibition, and autophagy. Overexpression of myo1b reversed the H/R-resulted H9c2 cell apoptosis, proliferation inhibition, and autophagy. Silence of myo1b had opposite effects, which promoted H9c2 cell apoptosis, reduced cell proliferation, and accelerated cell autophagy. Taken together, Myo1b took part in the modulation of H/R-stimulated cardiomyocyte apoptosis and autophagy, which might be serve as a potential endogenous target for prevention and therapy of I/R injury.http://dx.doi.org/10.1155/2022/5187304
spellingShingle Jing Xu
Jin Huang
Xiaojie He
Mingshuang Hu
Shan Su
Ping Liu
Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
Analytical Cellular Pathology
title Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
title_full Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
title_fullStr Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
title_full_unstemmed Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
title_short Myosin 1b Participated in the Modulation of Hypoxia/Reoxygenation-Caused H9c2 Cell Apoptosis and Autophagy
title_sort myosin 1b participated in the modulation of hypoxia reoxygenation caused h9c2 cell apoptosis and autophagy
url http://dx.doi.org/10.1155/2022/5187304
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AT shansu myosin1bparticipatedinthemodulationofhypoxiareoxygenationcausedh9c2cellapoptosisandautophagy
AT pingliu myosin1bparticipatedinthemodulationofhypoxiareoxygenationcausedh9c2cellapoptosisandautophagy