Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis

Background Dysregulated circular RNAs (circRNAs) are involved in osteoarthritis (OA) progression.Objective We aimed to explore the effect of hsa_circ_0044719 (circTRIM25) on the ferroptosis of chondrocytes.Methods Chondrocytes were treated with interleukin (IL)-1β to generate cell model. Cellular be...

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Main Authors: Chunlei He, Zhaogan Zeng, Yadong Yang, Shanshan Ye, Qiang Wu, Xunzhi Liu, Chenghong Liu, Wanhui Zeng, Sheng Liu
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Autoimmunity
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Online Access:https://www.tandfonline.com/doi/10.1080/08916934.2024.2361749
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author Chunlei He
Zhaogan Zeng
Yadong Yang
Shanshan Ye
Qiang Wu
Xunzhi Liu
Chenghong Liu
Wanhui Zeng
Sheng Liu
author_facet Chunlei He
Zhaogan Zeng
Yadong Yang
Shanshan Ye
Qiang Wu
Xunzhi Liu
Chenghong Liu
Wanhui Zeng
Sheng Liu
author_sort Chunlei He
collection DOAJ
description Background Dysregulated circular RNAs (circRNAs) are involved in osteoarthritis (OA) progression.Objective We aimed to explore the effect of hsa_circ_0044719 (circTRIM25) on the ferroptosis of chondrocytes.Methods Chondrocytes were treated with interleukin (IL)-1β to generate cell model. Cellular behaviours were measured using cell counting kit-8, enzyme-linked immunosorbent assay, relevant kits, propidium iodide staining, and immunofluorescence assay. Quantitative real-time polymerase chain reaction was performed to examine the expression of circTRIM25, miR-138-5p, and cAMP responsive element binding protein 1 (CREB1), and their interactions were assessed using luciferase reporter analysis and RNA pull-down assay.Results CircTRIM25 was upregulated in OA tissues and IL-1β-stimulated chondrocytes. Knockdown of circTRIM25 facilitated the viability and suppressed ferroptosis and inflammation of IL-1β-induced cells. CircTRIM25 served as a sponge of miR-138-5p, which directly targets CREB1. Downregulation of miR-138-5p abrogated the effect induced by knockdown of circTRIM25. Furthermore, enforced CREB1 reversed the miR-138-5p induced effect. Moreover, knockdown of circTRIM25 attenuated cartilage injury in vivo.Conclusion Silencing of circTRIM25 inhibited ferroptosis of chondrocytes via the miR-138-5p/CREB axis and thus attenuated OA progression.
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spelling doaj-art-2f1dd11b5b77478bbd7a4c746dccaf5b2025-08-20T02:38:31ZengTaylor & Francis GroupAutoimmunity0891-69341607-842X2024-12-0157110.1080/08916934.2024.2361749Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritisChunlei He0Zhaogan Zeng1Yadong Yang2Shanshan Ye3Qiang Wu4Xunzhi Liu5Chenghong Liu6Wanhui Zeng7Sheng Liu8Department of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaGannan Medical University, Ganzhou, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaGannan Medical University, Ganzhou, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaBackground Dysregulated circular RNAs (circRNAs) are involved in osteoarthritis (OA) progression.Objective We aimed to explore the effect of hsa_circ_0044719 (circTRIM25) on the ferroptosis of chondrocytes.Methods Chondrocytes were treated with interleukin (IL)-1β to generate cell model. Cellular behaviours were measured using cell counting kit-8, enzyme-linked immunosorbent assay, relevant kits, propidium iodide staining, and immunofluorescence assay. Quantitative real-time polymerase chain reaction was performed to examine the expression of circTRIM25, miR-138-5p, and cAMP responsive element binding protein 1 (CREB1), and their interactions were assessed using luciferase reporter analysis and RNA pull-down assay.Results CircTRIM25 was upregulated in OA tissues and IL-1β-stimulated chondrocytes. Knockdown of circTRIM25 facilitated the viability and suppressed ferroptosis and inflammation of IL-1β-induced cells. CircTRIM25 served as a sponge of miR-138-5p, which directly targets CREB1. Downregulation of miR-138-5p abrogated the effect induced by knockdown of circTRIM25. Furthermore, enforced CREB1 reversed the miR-138-5p induced effect. Moreover, knockdown of circTRIM25 attenuated cartilage injury in vivo.Conclusion Silencing of circTRIM25 inhibited ferroptosis of chondrocytes via the miR-138-5p/CREB axis and thus attenuated OA progression.https://www.tandfonline.com/doi/10.1080/08916934.2024.2361749OsteoarthritiscircTRIM25ferroptosismiR-138a-5pcartilage regeneration
spellingShingle Chunlei He
Zhaogan Zeng
Yadong Yang
Shanshan Ye
Qiang Wu
Xunzhi Liu
Chenghong Liu
Wanhui Zeng
Sheng Liu
Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
Autoimmunity
Osteoarthritis
circTRIM25
ferroptosis
miR-138a-5p
cartilage regeneration
title Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
title_full Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
title_fullStr Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
title_full_unstemmed Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
title_short Silencing of CircTRIM25/miR-138-5p/CREB1 axis promotes chondrogenesis in osteoarthritis
title_sort silencing of circtrim25 mir 138 5p creb1 axis promotes chondrogenesis in osteoarthritis
topic Osteoarthritis
circTRIM25
ferroptosis
miR-138a-5p
cartilage regeneration
url https://www.tandfonline.com/doi/10.1080/08916934.2024.2361749
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