Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons

Kalirin-7 (Kal7), a major isoform of Kalirin in the adult rodent hippocampus, is exclusively localized to the postsynaptic side of mature excitatory synapses in hippocampal neurons. Kal7 interacts with multiple PDZ domain—containing proteins through its unique PDZ binding motif. Overexpression of Ka...

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Main Author: Xin-Ming Ma
Format: Article
Language:English
Published: Wiley 2010-01-01
Series:The Scientific World Journal
Online Access:http://dx.doi.org/10.1100/tsw.2010.148
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author Xin-Ming Ma
author_facet Xin-Ming Ma
author_sort Xin-Ming Ma
collection DOAJ
description Kalirin-7 (Kal7), a major isoform of Kalirin in the adult rodent hippocampus, is exclusively localized to the postsynaptic side of mature excitatory synapses in hippocampal neurons. Kal7 interacts with multiple PDZ domain—containing proteins through its unique PDZ binding motif. Overexpression of Kal7 increases spine density and spine size, whereas reduction of endogenous Kal7 expression by small hairpin RNA (shRNA) causes a decrease in synapse number and spine density in cultured hippocampal neurons. Hippocampal CA1 pyramidal neurons of Kal7 knockout (Kal7KO) mice show decreased spine density, spine length, synapse number, and postsynaptic density (PSD) size in their apical dendrites; are deficient in long-term potentiation (LTP); and exhibit decreased frequency of spontaneous excitatory postsynaptic current (sEPSC). Kal7 plays a key role in estrogen-mediated spine/synapse formation in hippocampal neurons. Kal7 is also an essential determinant of dendritic spine formation following chronic cocaine treatment. Kal7 plays a key role in excitatory synapse formation and function.
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spelling doaj-art-2e6a3826ed6f4a0bb597cdea5dd96ca42025-08-20T03:18:28ZengWileyThe Scientific World Journal1537-744X2010-01-01101655166610.1100/tsw.2010.148Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal NeuronsXin-Ming Ma0Department of Neuroscience, University of Connecticut Health Center, Farmington, CT, USAKalirin-7 (Kal7), a major isoform of Kalirin in the adult rodent hippocampus, is exclusively localized to the postsynaptic side of mature excitatory synapses in hippocampal neurons. Kal7 interacts with multiple PDZ domain—containing proteins through its unique PDZ binding motif. Overexpression of Kal7 increases spine density and spine size, whereas reduction of endogenous Kal7 expression by small hairpin RNA (shRNA) causes a decrease in synapse number and spine density in cultured hippocampal neurons. Hippocampal CA1 pyramidal neurons of Kal7 knockout (Kal7KO) mice show decreased spine density, spine length, synapse number, and postsynaptic density (PSD) size in their apical dendrites; are deficient in long-term potentiation (LTP); and exhibit decreased frequency of spontaneous excitatory postsynaptic current (sEPSC). Kal7 plays a key role in estrogen-mediated spine/synapse formation in hippocampal neurons. Kal7 is also an essential determinant of dendritic spine formation following chronic cocaine treatment. Kal7 plays a key role in excitatory synapse formation and function.http://dx.doi.org/10.1100/tsw.2010.148
spellingShingle Xin-Ming Ma
Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
The Scientific World Journal
title Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
title_full Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
title_fullStr Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
title_full_unstemmed Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
title_short Kalirin-7 is a Key Player in the Formation of Excitatory Synapses in Hippocampal Neurons
title_sort kalirin 7 is a key player in the formation of excitatory synapses in hippocampal neurons
url http://dx.doi.org/10.1100/tsw.2010.148
work_keys_str_mv AT xinmingma kalirin7isakeyplayerintheformationofexcitatorysynapsesinhippocampalneurons