PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc

Objective: The roles of transcriptional factor PR/SET domain 1 (PRDM1) have been revealed in numerous tumor types. However, its effects on gastric cancer progression are still confusing. This study aimed to explore the roles and underlying mechanisms of PRDM1 in gastric cancer progression. Methods:...

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Main Authors: Hai Qin, Manqin Yuan, Yaqin Yuan, Xue Liu, Siye Yi
Format: Article
Language:English
Published: Elsevier 2025-09-01
Series:Translational Oncology
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S1936523325001743
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author Hai Qin
Manqin Yuan
Yaqin Yuan
Xue Liu
Siye Yi
author_facet Hai Qin
Manqin Yuan
Yaqin Yuan
Xue Liu
Siye Yi
author_sort Hai Qin
collection DOAJ
description Objective: The roles of transcriptional factor PR/SET domain 1 (PRDM1) have been revealed in numerous tumor types. However, its effects on gastric cancer progression are still confusing. This study aimed to explore the roles and underlying mechanisms of PRDM1 in gastric cancer progression. Methods: In silico analysis was used to analyze gene expression levels, expression correlation, and the relationship with patients’ survivals. Functional experiments, including Western blot, RT-qPCR, sphere-formation ability, cytometry analysis, and tumorigenic analysis, were conducted to evaluate PRDM1’s effects on gastric cancer stemness. ChIP, luciferase reporter, and bioinformatic analysis were performed to reveal the underlying mechanisms. Results: we demonstrated that PRDM1 was highly expressed in gastric cancer tissues and cells. Knockdown of PRDM1 significantly attenuated gastric cancer stemness. Mechanistical studies indicated that PRDM1 directly bound to the promoter region of Myc, a critical contributor of cancer stem cells (CSCs), and thus enhanced Myc transcription activity. Conclusions: Taken together, this study indicates that PRDM1 could enhance gastric cancer stemness by increasing the transcriptional activity.
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series Translational Oncology
spelling doaj-art-2c015cede2e94775aa826b00c4be5d682025-08-20T02:10:01ZengElsevierTranslational Oncology1936-52332025-09-015910244310.1016/j.tranon.2025.102443PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of MycHai Qin0Manqin Yuan1Yaqin Yuan2Xue Liu3Siye Yi4Department of Clinical Laboratory, Beijing Jishuitan Hospital Guizhou Hospital, No. 206, Sixian Street, Baiyun District, Guiyang City, Guizhou Province 550014, ChinaDepartment of Clinical Laboratory Medicine, Guizhou Medical University, Guiyang. Guizhou, ChinaMicrobiological Laboratory, Guizhou Center for Medical Device Testing, Guiyang, Guizhou, ChinaComprehensive Treatment Ward, Mudan People's Hospital of Heze, 2111 Kangzhuang Road, Mudan District, Heze, Shandong 274000, ChinaDepartment of Clinical Laboratory, Beijing Jishuitan Hospital Guizhou Hospital, No. 206, Sixian Street, Baiyun District, Guiyang City, Guizhou Province 550014, China; Corresponding author.Objective: The roles of transcriptional factor PR/SET domain 1 (PRDM1) have been revealed in numerous tumor types. However, its effects on gastric cancer progression are still confusing. This study aimed to explore the roles and underlying mechanisms of PRDM1 in gastric cancer progression. Methods: In silico analysis was used to analyze gene expression levels, expression correlation, and the relationship with patients’ survivals. Functional experiments, including Western blot, RT-qPCR, sphere-formation ability, cytometry analysis, and tumorigenic analysis, were conducted to evaluate PRDM1’s effects on gastric cancer stemness. ChIP, luciferase reporter, and bioinformatic analysis were performed to reveal the underlying mechanisms. Results: we demonstrated that PRDM1 was highly expressed in gastric cancer tissues and cells. Knockdown of PRDM1 significantly attenuated gastric cancer stemness. Mechanistical studies indicated that PRDM1 directly bound to the promoter region of Myc, a critical contributor of cancer stem cells (CSCs), and thus enhanced Myc transcription activity. Conclusions: Taken together, this study indicates that PRDM1 could enhance gastric cancer stemness by increasing the transcriptional activity.http://www.sciencedirect.com/science/article/pii/S1936523325001743PRDM1Gastric cancerStemnessMycTranscriptional factor
spellingShingle Hai Qin
Manqin Yuan
Yaqin Yuan
Xue Liu
Siye Yi
PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
Translational Oncology
PRDM1
Gastric cancer
Stemness
Myc
Transcriptional factor
title PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
title_full PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
title_fullStr PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
title_full_unstemmed PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
title_short PRDM1 promotes the stemness of gastric cancer cells by enhancing the transactivation of Myc
title_sort prdm1 promotes the stemness of gastric cancer cells by enhancing the transactivation of myc
topic PRDM1
Gastric cancer
Stemness
Myc
Transcriptional factor
url http://www.sciencedirect.com/science/article/pii/S1936523325001743
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