H5 subtype avian influenza virus induces Golgi apparatus stress response via TFE3 pathway to promote virus replication.
During infection, avian influenza virus (AIV) triggers endoplasmic reticulum (ER) stress, a well-established phenomenon in previous research. The Golgi apparatus, situated downstream of the ER and crucial for protein trafficking, may be impacted by AIV infection. However, it remains unclear whether...
Saved in:
| Main Authors: | Yuncong Yin, Xianjin Kan, Xinyu Miao, Yingjie Sun, Sujuan Chen, Tao Qin, Chan Ding, Daxin Peng, Xiufan Liu |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Public Library of Science (PLoS)
2024-12-01
|
| Series: | PLoS Pathogens |
| Online Access: | https://doi.org/10.1371/journal.ppat.1012748 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
PAIVS: prediction of avian influenza virus subtype
by: Hyeon-Chun Park, et al.
Published: (2020-03-01) -
Neutrality, cross-immunity and subtype dominance in avian influenza viruses.
by: Vicki L Brown, et al.
Published: (2014-01-01) -
Virulence and transmission characteristics of clade 2.3.4.4b H5N6 subtype avian influenza viruses possessing different internal gene constellations
by: Wanchen Zhao, et al.
Published: (2023-12-01) -
Critical Role of HAX-1 in Promoting Avian Influenza Virus Replication in Lung Epithelial Cells
by: Xue Li, et al.
Published: (2018-01-01) -
The emergence of new antigen branches of H9N2 avian influenza virus in China due to antigenic drift on hemagglutinin through antibody escape at immunodominant sites
by: Nan Zhang, et al.
Published: (2023-12-01)