Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease

Aim. To assess biomarkers of T2-inflammation in patients with chronic obstructive pulmonary disease (COPD). Materials and methods. We examined 173 adult outpatients (80% male, age 40–89 yrs) with COPD. Lung function tests were assessed by using the Spirograph 2120 (Vitalograph, UK). Blood eosinop...

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Main Authors: Galina R. Sergeeva, Alexander V. Emelyanov, Evgeniya V. Leshenkova, Antonina A. Znakhurenko
Format: Article
Language:Russian
Published: "Consilium Medicum" Publishing house 2025-01-01
Series:Терапевтический архив
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Online Access:https://ter-arkhiv.ru/0040-3660/article/viewFile/646535/193867
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author Galina R. Sergeeva
Alexander V. Emelyanov
Evgeniya V. Leshenkova
Antonina A. Znakhurenko
author_facet Galina R. Sergeeva
Alexander V. Emelyanov
Evgeniya V. Leshenkova
Antonina A. Znakhurenko
author_sort Galina R. Sergeeva
collection DOAJ
description Aim. To assess biomarkers of T2-inflammation in patients with chronic obstructive pulmonary disease (COPD). Materials and methods. We examined 173 adult outpatients (80% male, age 40–89 yrs) with COPD. Lung function tests were assessed by using the Spirograph 2120 (Vitalograph, UK). Blood eosinophils (Eos) were measured by automatic haemoanalyser. Atopic status was determined by serum specific immunoglobulin E to common inhalant allergens. Fractional exhaled nitric oxide (FeNO) was measured by a chemiluminescence analyzer (LR4100, Logan Research, Rochester, UK). Symptoms and quality of life were assessed by using Russian versions of St. George's Respiratory Questionnaire (SGRQ) and COPD Assessment Test. Statistical analyses were performed with Statistica ver. 10.0 (StatSoft, Inc., USA). Results. The most frequent marker was blood Eos ≥150 cell/μl (36%), less often was elevated level FeNO≥20 ppb, allergy was rare (5%) and house dust mites were the common allergen. T2-associated diseases (allergic rhinitis, nasal polyposis, atopic dermatitis) were diagnosed in 7% patients. Conclusion. Forty percent of patients with COPD without concomitant asthma have markers of T2-airway inflammation in a real clinical practice. The most frequent marker was blood Eos≥150 cell/μl that was associated with rate of COPD exacerbation. Frequency of concomitant T2-diseases (allergic rhinitis, nasal polyps, atopic dermatitis) was low.
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spelling doaj-art-2ae4c298f95749bcabc669f7eb004bc12025-08-20T02:14:38Zrus"Consilium Medicum" Publishing houseТерапевтический архив0040-36602309-53422025-01-0197325025610.26442/00403660.2025.03.20314778661Markers of T2-airway inflammation in patients with chronic obstructive pulmonary diseaseGalina R. Sergeeva0https://orcid.org/0000-0003-1544-4336Alexander V. Emelyanov1https://orcid.org/0000-0002-8574-6869Evgeniya V. Leshenkova2https://orcid.org/0000-0003-4616-3166Antonina A. Znakhurenko3https://orcid.org/0009-0008-3218-3397Mechnikov North-Western State Medical UniversityMechnikov North-Western State Medical UniversityMechnikov North-Western State Medical UniversityMechnikov North-Western State Medical UniversityAim. To assess biomarkers of T2-inflammation in patients with chronic obstructive pulmonary disease (COPD). Materials and methods. We examined 173 adult outpatients (80% male, age 40–89 yrs) with COPD. Lung function tests were assessed by using the Spirograph 2120 (Vitalograph, UK). Blood eosinophils (Eos) were measured by automatic haemoanalyser. Atopic status was determined by serum specific immunoglobulin E to common inhalant allergens. Fractional exhaled nitric oxide (FeNO) was measured by a chemiluminescence analyzer (LR4100, Logan Research, Rochester, UK). Symptoms and quality of life were assessed by using Russian versions of St. George's Respiratory Questionnaire (SGRQ) and COPD Assessment Test. Statistical analyses were performed with Statistica ver. 10.0 (StatSoft, Inc., USA). Results. The most frequent marker was blood Eos ≥150 cell/μl (36%), less often was elevated level FeNO≥20 ppb, allergy was rare (5%) and house dust mites were the common allergen. T2-associated diseases (allergic rhinitis, nasal polyposis, atopic dermatitis) were diagnosed in 7% patients. Conclusion. Forty percent of patients with COPD without concomitant asthma have markers of T2-airway inflammation in a real clinical practice. The most frequent marker was blood Eos≥150 cell/μl that was associated with rate of COPD exacerbation. Frequency of concomitant T2-diseases (allergic rhinitis, nasal polyps, atopic dermatitis) was low.https://ter-arkhiv.ru/0040-3660/article/viewFile/646535/193867chronic obstructive pulmonary diseasebiomarkerst2-inflammation
spellingShingle Galina R. Sergeeva
Alexander V. Emelyanov
Evgeniya V. Leshenkova
Antonina A. Znakhurenko
Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
Терапевтический архив
chronic obstructive pulmonary disease
biomarkers
t2-inflammation
title Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
title_full Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
title_fullStr Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
title_full_unstemmed Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
title_short Markers of T2-airway inflammation in patients with chronic obstructive pulmonary disease
title_sort markers of t2 airway inflammation in patients with chronic obstructive pulmonary disease
topic chronic obstructive pulmonary disease
biomarkers
t2-inflammation
url https://ter-arkhiv.ru/0040-3660/article/viewFile/646535/193867
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AT evgeniyavleshenkova markersoft2airwayinflammationinpatientswithchronicobstructivepulmonarydisease
AT antoninaaznakhurenko markersoft2airwayinflammationinpatientswithchronicobstructivepulmonarydisease