Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells

Abstract Dysregulated expression of circular RNAs (circRNAs) has been implicated in the initiation and progression of various diseases, including cancer. In this study, we investigated the role of hsa_circ_0005571 in modulating the biological characteristics of colorectal cancer (CRC) cells. Initial...

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Main Authors: Haipeng Ge, Jiaxuan Zhang, Ran Tao, Li Ding, Song Jiang, Jiale Shen, Longjun Bian, Jun Qin
Format: Article
Language:English
Published: Nature Portfolio 2025-08-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-16450-2
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author Haipeng Ge
Jiaxuan Zhang
Ran Tao
Li Ding
Song Jiang
Jiale Shen
Longjun Bian
Jun Qin
author_facet Haipeng Ge
Jiaxuan Zhang
Ran Tao
Li Ding
Song Jiang
Jiale Shen
Longjun Bian
Jun Qin
author_sort Haipeng Ge
collection DOAJ
description Abstract Dysregulated expression of circular RNAs (circRNAs) has been implicated in the initiation and progression of various diseases, including cancer. In this study, we investigated the role of hsa_circ_0005571 in modulating the biological characteristics of colorectal cancer (CRC) cells. Initially, the circRNA expression profiles of CRC tissues and corresponding normal tissues were analyzed using bioinformatics to identify differentially expressed circRNAs. Subsequently, quantitative real-time PCR (qRT‒PCR) was used to validate the expression levels of hsa_circ_0005571 in both CRC tissues and cell lines. In vitro assays, including the Cell Counting Kit-8 (CCK-8), colony formation, 5-ethynyl-2′-deoxyuridine (EdU) incorporation, wound healing, and Transwell invasion assays, were employed to assess the effects of hsa_circ_0005571 on CRC cell proliferation and invasion. Additionally, tumorigenic potential was examined through in vivo tumorigenesis experiments in nude mice. Bioinformatic predictions and experimental evidence indicated that hsa_circ_0005571 may function as a sponge for miR-520f-3p, which was found to be downregulated in CRC cells. Moreover, LRP8 was predicted and confirmed as a direct downstream target of miR-520f-3p, with both the mRNA and protein levels of LRP8 significantly elevated in CRC cells. Overall, our results suggest that hsa_circ_0005571 enhances CRC proliferation and invasion by modulating the miR-520f-3p/LRP8 axis. Furthermore, elevated hsa_circ_0005571 expression in CRC tissue samples and cell lines relative to that in normal controls was positively correlated with metastasis (p = 0.029) and advanced clinical stage (p = 0.006). Survival analyses revealed that CRC patients with high levels of hsa_circ_0005571 expression had significantly lower overall survival rates than did those with low expression (p < 0.05). Functional assays confirmed that hsa_circ_0005571 facilitates CRC cell proliferation and migration, whereas its knockdown results in reduced migration, proliferation, and tumorigenic potential in vivo. Finally, Western blot analyses demonstrated that LRP8 expression varied in accordance with the levels of hsa_circ_0005571 and miR-520f-3p, further confirming the involvement of the hsa_circ_0005571/miR-520f-3p/LRP8 regulatory axis in CRC. Our study suggested that hsa_circRNA_0005571 promotes proliferation and migration through the miR-520f-3p/LRP8 axis in CRC. Consequently, hsa_circ_0005571 may represent a promising novel gene target for the diagnosis and treatment of CRC.
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spelling doaj-art-2adc1daadb9b4a0aaa926cb30b2827ac2025-08-24T11:27:36ZengNature PortfolioScientific Reports2045-23222025-08-0115111210.1038/s41598-025-16450-2Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cellsHaipeng Ge0Jiaxuan Zhang1Ran Tao2Li Ding3Song Jiang4Jiale Shen5Longjun Bian6Jun Qin7Department of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of General Surgery, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityDepartment of Trauma Center, Affiliated Hospital of Nantong UniversityAbstract Dysregulated expression of circular RNAs (circRNAs) has been implicated in the initiation and progression of various diseases, including cancer. In this study, we investigated the role of hsa_circ_0005571 in modulating the biological characteristics of colorectal cancer (CRC) cells. Initially, the circRNA expression profiles of CRC tissues and corresponding normal tissues were analyzed using bioinformatics to identify differentially expressed circRNAs. Subsequently, quantitative real-time PCR (qRT‒PCR) was used to validate the expression levels of hsa_circ_0005571 in both CRC tissues and cell lines. In vitro assays, including the Cell Counting Kit-8 (CCK-8), colony formation, 5-ethynyl-2′-deoxyuridine (EdU) incorporation, wound healing, and Transwell invasion assays, were employed to assess the effects of hsa_circ_0005571 on CRC cell proliferation and invasion. Additionally, tumorigenic potential was examined through in vivo tumorigenesis experiments in nude mice. Bioinformatic predictions and experimental evidence indicated that hsa_circ_0005571 may function as a sponge for miR-520f-3p, which was found to be downregulated in CRC cells. Moreover, LRP8 was predicted and confirmed as a direct downstream target of miR-520f-3p, with both the mRNA and protein levels of LRP8 significantly elevated in CRC cells. Overall, our results suggest that hsa_circ_0005571 enhances CRC proliferation and invasion by modulating the miR-520f-3p/LRP8 axis. Furthermore, elevated hsa_circ_0005571 expression in CRC tissue samples and cell lines relative to that in normal controls was positively correlated with metastasis (p = 0.029) and advanced clinical stage (p = 0.006). Survival analyses revealed that CRC patients with high levels of hsa_circ_0005571 expression had significantly lower overall survival rates than did those with low expression (p < 0.05). Functional assays confirmed that hsa_circ_0005571 facilitates CRC cell proliferation and migration, whereas its knockdown results in reduced migration, proliferation, and tumorigenic potential in vivo. Finally, Western blot analyses demonstrated that LRP8 expression varied in accordance with the levels of hsa_circ_0005571 and miR-520f-3p, further confirming the involvement of the hsa_circ_0005571/miR-520f-3p/LRP8 regulatory axis in CRC. Our study suggested that hsa_circRNA_0005571 promotes proliferation and migration through the miR-520f-3p/LRP8 axis in CRC. Consequently, hsa_circ_0005571 may represent a promising novel gene target for the diagnosis and treatment of CRC.https://doi.org/10.1038/s41598-025-16450-2Colorectal cancercircRNAhsa_circ_0005571ProliferationMigration
spellingShingle Haipeng Ge
Jiaxuan Zhang
Ran Tao
Li Ding
Song Jiang
Jiale Shen
Longjun Bian
Jun Qin
Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
Scientific Reports
Colorectal cancer
circRNA
hsa_circ_0005571
Proliferation
Migration
title Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
title_full Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
title_fullStr Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
title_full_unstemmed Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
title_short Hsa_circ_0005571 promotes the proliferation and invasion of colorectal cancer cells
title_sort hsa circ 0005571 promotes the proliferation and invasion of colorectal cancer cells
topic Colorectal cancer
circRNA
hsa_circ_0005571
Proliferation
Migration
url https://doi.org/10.1038/s41598-025-16450-2
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