Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions
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| Main Authors: | , , , , , , , |
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| Format: | Article |
| Language: | English |
| Published: |
Wiley
2014-07-01
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| Series: | Molecular Genetics & Genomic Medicine |
| Online Access: | https://doi.org/10.1002/mgg3.79 |
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| author | Neil E. Bowles Cammon B. Arrington Keiichi Hirono Tsuneyuki Nakamura Long Ngo Yin Shen Wee Fukiko Ichida John H. Weis |
| author_facet | Neil E. Bowles Cammon B. Arrington Keiichi Hirono Tsuneyuki Nakamura Long Ngo Yin Shen Wee Fukiko Ichida John H. Weis |
| author_sort | Neil E. Bowles |
| collection | DOAJ |
| format | Article |
| id | doaj-art-295ed0ecfa2046649289f892c1a3c0ab |
| institution | OA Journals |
| issn | 2324-9269 |
| language | English |
| publishDate | 2014-07-01 |
| publisher | Wiley |
| record_format | Article |
| series | Molecular Genetics & Genomic Medicine |
| spelling | doaj-art-295ed0ecfa2046649289f892c1a3c0ab2025-08-20T01:55:12ZengWileyMolecular Genetics & Genomic Medicine2324-92692014-07-012435636110.1002/mgg3.79Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesionsNeil E. Bowles0Cammon B. Arrington1Keiichi Hirono2Tsuneyuki Nakamura3Long Ngo4Yin Shen Wee5Fukiko Ichida6John H. Weis7Division of Cardiology Department of Pediatrics University of Utah School of Medicine Salt Lake City UtahDivision of Cardiology Department of Pediatrics University of Utah School of Medicine Salt Lake City UtahDepartment of Pediatrics Faculty of Medicine University of Toyama Toyama JapanDepartment of Pediatrics Kanazawa Medical University Kanazawa JapanDepartment of Medicine Harvard Medical School Beth Israel Deaconess Medical Center Brookline MassachusettsDepartment of Pathology University of Utah School of Medicine Salt Lake City UtahDepartment of Pediatrics Faculty of Medicine University of Toyama Toyama JapanDepartment of Pathology University of Utah School of Medicine Salt Lake City Utahhttps://doi.org/10.1002/mgg3.79 |
| spellingShingle | Neil E. Bowles Cammon B. Arrington Keiichi Hirono Tsuneyuki Nakamura Long Ngo Yin Shen Wee Fukiko Ichida John H. Weis Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions Molecular Genetics & Genomic Medicine |
| title | Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions |
| title_full | Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions |
| title_fullStr | Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions |
| title_full_unstemmed | Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions |
| title_short | Kawasaki disease patients homozygous for the rs12252‐C variant of interferon‐induced transmembrane protein‐3 are significantly more likely to develop coronary artery lesions |
| title_sort | kawasaki disease patients homozygous for the rs12252 c variant of interferon induced transmembrane protein 3 are significantly more likely to develop coronary artery lesions |
| url | https://doi.org/10.1002/mgg3.79 |
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