PPARα in Obesity: Sex Difference and Estrogen Involvement

Peroxisome proliferator-activated receptor α (PPARα) is a member of the steroid hormone receptor superfamily and is well known to act as the molecular target for lipid-lowering drugs of the fibrate family. At the molecular level, PPARα regulates the transcription of a number of genes critical for li...

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Main Author: Michung Yoon
Format: Article
Language:English
Published: Wiley 2010-01-01
Series:PPAR Research
Online Access:http://dx.doi.org/10.1155/2010/584296
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author Michung Yoon
author_facet Michung Yoon
author_sort Michung Yoon
collection DOAJ
description Peroxisome proliferator-activated receptor α (PPARα) is a member of the steroid hormone receptor superfamily and is well known to act as the molecular target for lipid-lowering drugs of the fibrate family. At the molecular level, PPARα regulates the transcription of a number of genes critical for lipid and lipoprotein metabolism. PPARα activators are further shown to reduce body weight gain and adiposity, at least in part, due to the increase of hepatic fatty acid oxidation and the decrease in levels of circulating triglycerides responsible for adipose cell hypertrophy and hyperplasia. However, these effects of the PPARα ligand fenofibrate on obesity are regulated with sexual dimorphism and seem to be influenced by the presence of functioning ovaries, suggesting the involvement of ovarian steroids in the control of obesity by PPARα. In female ovariectomized mice, 17β-estradiol inhibits the actions of fenofibrate on obesity through its suppressive effects on the expression of PPARα target genes, and these processes may be mediated by inhibiting the coactivator recruitment of PPARα. Thus, it is likely that PPARα functions on obesity may be enhanced in estrogen-deficient states.
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spelling doaj-art-293162de04d34b918db1f34df05616b22025-08-20T02:03:51ZengWileyPPAR Research1687-47571687-47652010-01-01201010.1155/2010/584296584296PPARα in Obesity: Sex Difference and Estrogen InvolvementMichung Yoon0Department of Life Sciences, Mokwon University, Taejon 302-729, Republic of KoreaPeroxisome proliferator-activated receptor α (PPARα) is a member of the steroid hormone receptor superfamily and is well known to act as the molecular target for lipid-lowering drugs of the fibrate family. At the molecular level, PPARα regulates the transcription of a number of genes critical for lipid and lipoprotein metabolism. PPARα activators are further shown to reduce body weight gain and adiposity, at least in part, due to the increase of hepatic fatty acid oxidation and the decrease in levels of circulating triglycerides responsible for adipose cell hypertrophy and hyperplasia. However, these effects of the PPARα ligand fenofibrate on obesity are regulated with sexual dimorphism and seem to be influenced by the presence of functioning ovaries, suggesting the involvement of ovarian steroids in the control of obesity by PPARα. In female ovariectomized mice, 17β-estradiol inhibits the actions of fenofibrate on obesity through its suppressive effects on the expression of PPARα target genes, and these processes may be mediated by inhibiting the coactivator recruitment of PPARα. Thus, it is likely that PPARα functions on obesity may be enhanced in estrogen-deficient states.http://dx.doi.org/10.1155/2010/584296
spellingShingle Michung Yoon
PPARα in Obesity: Sex Difference and Estrogen Involvement
PPAR Research
title PPARα in Obesity: Sex Difference and Estrogen Involvement
title_full PPARα in Obesity: Sex Difference and Estrogen Involvement
title_fullStr PPARα in Obesity: Sex Difference and Estrogen Involvement
title_full_unstemmed PPARα in Obesity: Sex Difference and Estrogen Involvement
title_short PPARα in Obesity: Sex Difference and Estrogen Involvement
title_sort pparα in obesity sex difference and estrogen involvement
url http://dx.doi.org/10.1155/2010/584296
work_keys_str_mv AT michungyoon pparainobesitysexdifferenceandestrogeninvolvement