Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva

We investigate the role of homeostatic mechanisms involved in acute, postprandial nutrient metabolism and nutrient-induced systemic inflammation in CKD presence and progression in Metabolic dysfunction-associated steatohepatitis (MASH). We assessed postprandial incretins (GLP-1 and GIP), intestinotr...

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Main Authors: Giovanni Musso, Mella Alberto, Filippo Mariano, Maurizio Cassader, Franco De Michieli, Antonella Riva, Giovanna Petrangolini, Stefano Togni, Silvia Pinach, Roberto Gambino
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Gut Microbes
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Online Access:https://www.tandfonline.com/doi/10.1080/19490976.2024.2424907
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author Giovanni Musso
Mella Alberto
Filippo Mariano
Maurizio Cassader
Franco De Michieli
Antonella Riva
Giovanna Petrangolini
Stefano Togni
Silvia Pinach
Roberto Gambino
author_facet Giovanni Musso
Mella Alberto
Filippo Mariano
Maurizio Cassader
Franco De Michieli
Antonella Riva
Giovanna Petrangolini
Stefano Togni
Silvia Pinach
Roberto Gambino
author_sort Giovanni Musso
collection DOAJ
description We investigate the role of homeostatic mechanisms involved in acute, postprandial nutrient metabolism and nutrient-induced systemic inflammation in CKD presence and progression in Metabolic dysfunction-associated steatohepatitis (MASH). We assessed postprandial incretins (GLP-1 and GIP), intestinotropic hormone GLP-2, endotoxin LPS, Zonulin (a marker of intestinal permeability), hepatokines, adipokines and NF-kB activation in circulating MNCs during a meal tolerance test in 52 biopsy proven MASH patients randomized to curcumin Meriva or placebo and 26 matched controls. At baseline, MASH-CKD had a lower GLP-2 response and a 2-fold higher postprandial LPS and NF-kB activation in MNCs than MASH patients without CKD, but similar remaining postprandial or fasting parameters. Postprandial IAUC GLP-2 predicted the presence of CKD in MASH (OR = 0.43, 95%CI:0.32-0.80, p = 0.008) independently of liver histology and traditional risk factors. After 72 weeks, changes in IAUC GLP-2 independently predicted the presence of CKD (OR = 0.49, 95%CI:0.21-0.73, p = 0.010) and eGFR changes [β(SE) = 0.510(0.007, p = 0.006] at end-of-treatment, In MASH, an impaired GLP-2 response to meals is associated with intestinal barrier dysfunction, endotoxemia and NF-kB-mediated systemic inflammation and may promote renal dysfunction and CKD. These data provide the rationale for evaluating GLP-2 analogues in MASH-related CKD.
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spelling doaj-art-27aee9611363417ea5dc3b9b2ee08c802025-08-20T02:18:05ZengTaylor & Francis GroupGut Microbes1949-09761949-09842024-12-0116110.1080/19490976.2024.2424907Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin merivaGiovanni Musso0Mella Alberto1Filippo Mariano2Maurizio Cassader3Franco De Michieli4Antonella Riva5Giovanna Petrangolini6Stefano Togni7Silvia Pinach8Roberto Gambino9MECAU Department San Luigi Gonzaga Hospital, Turin, ItalyDepartment of Nephrology, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyDepartment of Nephrology, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyDepartment of Medical Sciences, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyDepartment of Medical Sciences, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyR & D Indena S.p.A, Milan, ItalyR & D Indena S.p.A, Milan, ItalyR & D Indena S.p.A, Milan, ItalyDepartment of Medical Sciences, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyDepartment of Medical Sciences, Città della Salute e della Scienza Hospital, University of Turin, Turin, ItalyWe investigate the role of homeostatic mechanisms involved in acute, postprandial nutrient metabolism and nutrient-induced systemic inflammation in CKD presence and progression in Metabolic dysfunction-associated steatohepatitis (MASH). We assessed postprandial incretins (GLP-1 and GIP), intestinotropic hormone GLP-2, endotoxin LPS, Zonulin (a marker of intestinal permeability), hepatokines, adipokines and NF-kB activation in circulating MNCs during a meal tolerance test in 52 biopsy proven MASH patients randomized to curcumin Meriva or placebo and 26 matched controls. At baseline, MASH-CKD had a lower GLP-2 response and a 2-fold higher postprandial LPS and NF-kB activation in MNCs than MASH patients without CKD, but similar remaining postprandial or fasting parameters. Postprandial IAUC GLP-2 predicted the presence of CKD in MASH (OR = 0.43, 95%CI:0.32-0.80, p = 0.008) independently of liver histology and traditional risk factors. After 72 weeks, changes in IAUC GLP-2 independently predicted the presence of CKD (OR = 0.49, 95%CI:0.21-0.73, p = 0.010) and eGFR changes [β(SE) = 0.510(0.007, p = 0.006] at end-of-treatment, In MASH, an impaired GLP-2 response to meals is associated with intestinal barrier dysfunction, endotoxemia and NF-kB-mediated systemic inflammation and may promote renal dysfunction and CKD. These data provide the rationale for evaluating GLP-2 analogues in MASH-related CKD.https://www.tandfonline.com/doi/10.1080/19490976.2024.2424907SteatohepatitisNF-kBpostprandialeGFRalbuminuriachemokine
spellingShingle Giovanni Musso
Mella Alberto
Filippo Mariano
Maurizio Cassader
Franco De Michieli
Antonella Riva
Giovanna Petrangolini
Stefano Togni
Silvia Pinach
Roberto Gambino
Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
Gut Microbes
Steatohepatitis
NF-kB
postprandial
eGFR
albuminuria
chemokine
title Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
title_full Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
title_fullStr Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
title_full_unstemmed Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
title_short Impaired postprandial GLP-2 response enhances endotoxemia, systemic inflammation, and kidney injury in metabolic dysfunction-associated steatohepatitis (MASH): effect of phospholipid curcumin meriva
title_sort impaired postprandial glp 2 response enhances endotoxemia systemic inflammation and kidney injury in metabolic dysfunction associated steatohepatitis mash effect of phospholipid curcumin meriva
topic Steatohepatitis
NF-kB
postprandial
eGFR
albuminuria
chemokine
url https://www.tandfonline.com/doi/10.1080/19490976.2024.2424907
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