Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients

Breast cancer was considered as a kind of prone breast tumors with the complicated pathological mechanisms and diverse clinical classifications. In the clinical treatments of HER2-positive tumor patients, HER2 monoclonal antibodies, such as Herceptin, have shown well-defined therapeutic effects. Nev...

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Main Authors: Yu Song, Qiao-Chen Geng, Wen-Jing An, Fu-Cheng Zhang, Ran Jiang, Rui-Sheng Zhao, Zhi-Jian Deng, Heng Li
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Molecular & Cellular Oncology
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Online Access:https://www.tandfonline.com/doi/10.1080/23723556.2024.2309715
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author Yu Song
Qiao-Chen Geng
Wen-Jing An
Fu-Cheng Zhang
Ran Jiang
Rui-Sheng Zhao
Zhi-Jian Deng
Heng Li
author_facet Yu Song
Qiao-Chen Geng
Wen-Jing An
Fu-Cheng Zhang
Ran Jiang
Rui-Sheng Zhao
Zhi-Jian Deng
Heng Li
author_sort Yu Song
collection DOAJ
description Breast cancer was considered as a kind of prone breast tumors with the complicated pathological mechanisms and diverse clinical classifications. In the clinical treatments of HER2-positive tumor patients, HER2 monoclonal antibodies, such as Herceptin, have shown well-defined therapeutic effects. Nevertheless, due to the heterogeneity of breast cancers, drug resistance inevitably appeared during the application of Herceptin. In order to fully understand the immune tolerance status of the tumor microenvironment in the population of sensitive and insensitive patients, this study carried out a series of studies through Luminex cytokines assay, clinicopathological analysis, immunofluorescence, and PCR. The results confirmed that in clinical samples sensitive to Herceptin, there were a large number of macrophages, and the protein expression levels and in situ expression of macrophage-related chemokines and inflammatory mediators are significantly higher than drug-resistant tumor samples. Further studies found that T cell function has a low correlation with tumor growth, and there are obvious obstacles in the process of peripheral blood immune cells entering the tumor microenvironment. In summary, this study provided clues for understanding the clinical drug resistance of HER2 monoclonal antibody and the clinical rational use of drugs and combination drugs.
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publishDate 2024-12-01
publisher Taylor & Francis Group
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series Molecular & Cellular Oncology
spelling doaj-art-26eb539acf5e45d59c9da63d01489be62025-08-20T01:58:48ZengTaylor & Francis GroupMolecular & Cellular Oncology2372-35562024-12-0111110.1080/23723556.2024.2309715Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patientsYu Song0Qiao-Chen Geng1Wen-Jing An2Fu-Cheng Zhang3Ran Jiang4Rui-Sheng Zhao5Zhi-Jian Deng6Heng Li7College of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaCollege of Pharmacy, Xinxiang Medical University, Xinxiang, ChinaYong Loo Lin School of Medicine, National University of Singapore, Singapore, SingaporeBreast cancer was considered as a kind of prone breast tumors with the complicated pathological mechanisms and diverse clinical classifications. In the clinical treatments of HER2-positive tumor patients, HER2 monoclonal antibodies, such as Herceptin, have shown well-defined therapeutic effects. Nevertheless, due to the heterogeneity of breast cancers, drug resistance inevitably appeared during the application of Herceptin. In order to fully understand the immune tolerance status of the tumor microenvironment in the population of sensitive and insensitive patients, this study carried out a series of studies through Luminex cytokines assay, clinicopathological analysis, immunofluorescence, and PCR. The results confirmed that in clinical samples sensitive to Herceptin, there were a large number of macrophages, and the protein expression levels and in situ expression of macrophage-related chemokines and inflammatory mediators are significantly higher than drug-resistant tumor samples. Further studies found that T cell function has a low correlation with tumor growth, and there are obvious obstacles in the process of peripheral blood immune cells entering the tumor microenvironment. In summary, this study provided clues for understanding the clinical drug resistance of HER2 monoclonal antibody and the clinical rational use of drugs and combination drugs.https://www.tandfonline.com/doi/10.1080/23723556.2024.2309715Herceptinbreast cancermacrophageschemotaxis
spellingShingle Yu Song
Qiao-Chen Geng
Wen-Jing An
Fu-Cheng Zhang
Ran Jiang
Rui-Sheng Zhao
Zhi-Jian Deng
Heng Li
Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
Molecular & Cellular Oncology
Herceptin
breast cancer
macrophages
chemotaxis
title Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
title_full Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
title_fullStr Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
title_full_unstemmed Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
title_short Hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in HER2-positive breast cancer patients
title_sort hypofunction of macrophage chemotaxis contributes to defective efficacy of herceptin in her2 positive breast cancer patients
topic Herceptin
breast cancer
macrophages
chemotaxis
url https://www.tandfonline.com/doi/10.1080/23723556.2024.2309715
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