A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein

Abstract Respiratory syncytial virus (RSV) is the leading cause of infant hospitalization. All current RSV therapeutics, including antibody prophylaxis and adult vaccination, target the RSV fusion glycoprotein (RSV-F). The seven neutralization sites on RSV-F are highly conserved and infrequently mut...

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Main Authors: Ahmed K. Oraby, Aleksandra Stojic, Farah Elawar, Leanne M. Bilawchuk, Ryley D. McClelland, Kaci Erwin, Madison J. Granoski, Cameron D. Griffiths, Justin D. Frederick, Elena Arutyunova, M. Joanne Lemieux, Frederick. G. West, Octavio Ramilo, Asuncion Mejias, Jason S. McLellan, David J. Marchant
Format: Article
Language:English
Published: Nature Portfolio 2025-04-01
Series:npj Viruses
Online Access:https://doi.org/10.1038/s44298-025-00119-8
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author Ahmed K. Oraby
Aleksandra Stojic
Farah Elawar
Leanne M. Bilawchuk
Ryley D. McClelland
Kaci Erwin
Madison J. Granoski
Cameron D. Griffiths
Justin D. Frederick
Elena Arutyunova
M. Joanne Lemieux
Frederick. G. West
Octavio Ramilo
Asuncion Mejias
Jason S. McLellan
David J. Marchant
author_facet Ahmed K. Oraby
Aleksandra Stojic
Farah Elawar
Leanne M. Bilawchuk
Ryley D. McClelland
Kaci Erwin
Madison J. Granoski
Cameron D. Griffiths
Justin D. Frederick
Elena Arutyunova
M. Joanne Lemieux
Frederick. G. West
Octavio Ramilo
Asuncion Mejias
Jason S. McLellan
David J. Marchant
author_sort Ahmed K. Oraby
collection DOAJ
description Abstract Respiratory syncytial virus (RSV) is the leading cause of infant hospitalization. All current RSV therapeutics, including antibody prophylaxis and adult vaccination, target the RSV fusion glycoprotein (RSV-F). The seven neutralization sites on RSV-F are highly conserved and infrequently mutate. Here, we show that a single amino acid mutation at position 305 in RSV-F significantly alters antigenic recognition of RSV-F binding sites and reduces the susceptibility of RSV to neutralizing antibodies. In an in vitro evolution assay, we show that RSV-F L305I occurs in a majority of RSV quasi-species. Computational modeling predicted that the L305I mutation altered the epitope landscape of RSV-F, resulting in changes to neutralizing antibody sensitivity and affinity towards the RSV-F glycoprotein. Screening of published RSV-F sequences revealed that position 305 in RSV-F was conserved with a leucine and isoleucine in RSV-A and RSV-B subtypes respectively. Our study suggests that select amino acids in RSV-F may act as ‘conformational switches’ for RSV to evade host serum antibodies. This work has important implications in understanding RSV evolution and resistance as it suggests that mutational resistance to neutralizing antibodies can occur at sites distal to antigenic epitopes, significantly altering antibody sensitivity to viral infection. These unique antigenic landscape changes should be considered in the context of vaccine and therapeutic development in order to better understand viral mechanisms of evasion and resistance.
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spelling doaj-art-25e310fbede84408bf88c73f8ef617802025-08-20T03:14:07ZengNature Portfolionpj Viruses2948-17672025-04-013111410.1038/s44298-025-00119-8A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoproteinAhmed K. Oraby0Aleksandra Stojic1Farah Elawar2Leanne M. Bilawchuk3Ryley D. McClelland4Kaci Erwin5Madison J. Granoski6Cameron D. Griffiths7Justin D. Frederick8Elena Arutyunova9M. Joanne Lemieux10Frederick. G. West11Octavio Ramilo12Asuncion Mejias13Jason S. McLellan14David J. Marchant15Department of Medical Microbiology and ImmunologyDepartment of Medical Microbiology and ImmunologyDepartment of Medical Microbiology and ImmunologyDepartment of Medical Microbiology and ImmunologyDepartment of Medical Microbiology and ImmunologyDepartment of Molecular Biosciences, The University of Texas at AustinDepartment of Medical Microbiology and ImmunologyDepartment of Biomedical Engineering, University of VirginiaDepartment of Medical Microbiology and ImmunologyDepartment of Biochemistry, University of AlbertaDepartment of Biochemistry, University of AlbertaDepartment of Chemistry, University of AlbertaCenter for Vaccines and Immunity, The Abigail Wexner Research Institute at Nationwide Children’s Hospital, The Ohio State University College of MedicineCenter for Vaccines and Immunity, The Abigail Wexner Research Institute at Nationwide Children’s Hospital, The Ohio State University College of MedicineDepartment of Molecular Biosciences, The University of Texas at AustinDepartment of Medical Microbiology and ImmunologyAbstract Respiratory syncytial virus (RSV) is the leading cause of infant hospitalization. All current RSV therapeutics, including antibody prophylaxis and adult vaccination, target the RSV fusion glycoprotein (RSV-F). The seven neutralization sites on RSV-F are highly conserved and infrequently mutate. Here, we show that a single amino acid mutation at position 305 in RSV-F significantly alters antigenic recognition of RSV-F binding sites and reduces the susceptibility of RSV to neutralizing antibodies. In an in vitro evolution assay, we show that RSV-F L305I occurs in a majority of RSV quasi-species. Computational modeling predicted that the L305I mutation altered the epitope landscape of RSV-F, resulting in changes to neutralizing antibody sensitivity and affinity towards the RSV-F glycoprotein. Screening of published RSV-F sequences revealed that position 305 in RSV-F was conserved with a leucine and isoleucine in RSV-A and RSV-B subtypes respectively. Our study suggests that select amino acids in RSV-F may act as ‘conformational switches’ for RSV to evade host serum antibodies. This work has important implications in understanding RSV evolution and resistance as it suggests that mutational resistance to neutralizing antibodies can occur at sites distal to antigenic epitopes, significantly altering antibody sensitivity to viral infection. These unique antigenic landscape changes should be considered in the context of vaccine and therapeutic development in order to better understand viral mechanisms of evasion and resistance.https://doi.org/10.1038/s44298-025-00119-8
spellingShingle Ahmed K. Oraby
Aleksandra Stojic
Farah Elawar
Leanne M. Bilawchuk
Ryley D. McClelland
Kaci Erwin
Madison J. Granoski
Cameron D. Griffiths
Justin D. Frederick
Elena Arutyunova
M. Joanne Lemieux
Frederick. G. West
Octavio Ramilo
Asuncion Mejias
Jason S. McLellan
David J. Marchant
A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
npj Viruses
title A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
title_full A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
title_fullStr A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
title_full_unstemmed A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
title_short A single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
title_sort single amino acid mutation alters multiple neutralization epitopes in the respiratory syncytial virus fusion glycoprotein
url https://doi.org/10.1038/s44298-025-00119-8
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