Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4

Abstract Early dissemination refers to the process by which cancer cells spread to distant organs at an early stage of the disease, often before the primary tumor is clinically detectable. Experimental studies have demonstrated that Her2 promotes early dissemination of breast cancer by inhibiting th...

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Main Authors: Guanwen Wang, Ping Wen, Ting Xue, Yuxin Huang, Qing Shao, Ningning Zhang, Fanli Qu, Jing Wang, Nan Wang, Xiaohua Zeng
Format: Article
Language:English
Published: BMC 2024-12-01
Series:Cell Communication and Signaling
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Online Access:https://doi.org/10.1186/s12964-024-02000-2
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author Guanwen Wang
Ping Wen
Ting Xue
Yuxin Huang
Qing Shao
Ningning Zhang
Fanli Qu
Jing Wang
Nan Wang
Xiaohua Zeng
author_facet Guanwen Wang
Ping Wen
Ting Xue
Yuxin Huang
Qing Shao
Ningning Zhang
Fanli Qu
Jing Wang
Nan Wang
Xiaohua Zeng
author_sort Guanwen Wang
collection DOAJ
description Abstract Early dissemination refers to the process by which cancer cells spread to distant organs at an early stage of the disease, often before the primary tumor is clinically detectable. Experimental studies have demonstrated that Her2 promotes early dissemination of breast cancer by inhibiting the p38 signaling pathway. However, the precise mechanism by which Her2 suppresses the activation of p38 signaling in early-stage cancer cells (ECCs) remains unclear. Here, we report that MAP3K4, an upstream kinase of p38, is downregulated in Her2 + ductal carcinoma in situ (DCIS) cells and tissues, which is required for Her2-induced early dissemination of DCIS cells by regulating the activation of the p38 signaling cascade. Furthermore, Her2 suppresses the transcription of MAP3K4 by downregulating the expression of HOXB13, a crucial transcription factor contributing to MAP3K4 expression in DCIS cells. Together, these findings unveil a novel downstream regulatory mechanism through which Her2 inhibits the activation of p38 signaling and facilitates early dissemination of breast cancer, offering insights into the development of effective diagnostic methods and targeted therapies for inhibiting the early dissemination of Her2 + breast cancer.
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institution DOAJ
issn 1478-811X
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publishDate 2024-12-01
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series Cell Communication and Signaling
spelling doaj-art-25accc8f4605486cb40ea6f16d6c1b1a2025-08-20T02:39:41ZengBMCCell Communication and Signaling1478-811X2024-12-0122111610.1186/s12964-024-02000-2Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4Guanwen Wang0Ping Wen1Ting Xue2Yuxin Huang3Qing Shao4Ningning Zhang5Fanli Qu6Jing Wang7Nan Wang8Xiaohua Zeng9Department of Breast Cancer Center, Chongqing University Cancer HospitalSchool of Medicine, Chongqing University Cancer Hospital, Chongqing UniversitySchool of Medicine, Chongqing University Cancer Hospital, Chongqing UniversitySchool of Medicine, Chongqing University Cancer Hospital, Chongqing UniversityDepartment of Breast Cancer Center, Chongqing University Cancer HospitalDepartment of Breast Cancer Center, Chongqing University Cancer HospitalDepartment of Breast Cancer Center, Chongqing University Cancer HospitalInner Mongolia Autonomous Region People’s HospitalThe Second Surgical Department of Breast Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin Medical University Cancer Institute & Hospital, National Clinical Research Center for CancerDepartment of Breast Cancer Center, Chongqing University Cancer HospitalAbstract Early dissemination refers to the process by which cancer cells spread to distant organs at an early stage of the disease, often before the primary tumor is clinically detectable. Experimental studies have demonstrated that Her2 promotes early dissemination of breast cancer by inhibiting the p38 signaling pathway. However, the precise mechanism by which Her2 suppresses the activation of p38 signaling in early-stage cancer cells (ECCs) remains unclear. Here, we report that MAP3K4, an upstream kinase of p38, is downregulated in Her2 + ductal carcinoma in situ (DCIS) cells and tissues, which is required for Her2-induced early dissemination of DCIS cells by regulating the activation of the p38 signaling cascade. Furthermore, Her2 suppresses the transcription of MAP3K4 by downregulating the expression of HOXB13, a crucial transcription factor contributing to MAP3K4 expression in DCIS cells. Together, these findings unveil a novel downstream regulatory mechanism through which Her2 inhibits the activation of p38 signaling and facilitates early dissemination of breast cancer, offering insights into the development of effective diagnostic methods and targeted therapies for inhibiting the early dissemination of Her2 + breast cancer.https://doi.org/10.1186/s12964-024-02000-2Breast cancerHer2Early disseminationp38MAP3K4HOXB13
spellingShingle Guanwen Wang
Ping Wen
Ting Xue
Yuxin Huang
Qing Shao
Ningning Zhang
Fanli Qu
Jing Wang
Nan Wang
Xiaohua Zeng
Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
Cell Communication and Signaling
Breast cancer
Her2
Early dissemination
p38
MAP3K4
HOXB13
title Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
title_full Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
title_fullStr Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
title_full_unstemmed Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
title_short Her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of MAP3K4
title_sort her2 promotes early dissemination of breast cancer by inhibiting the p38 pathway through the downregulation of map3k4
topic Breast cancer
Her2
Early dissemination
p38
MAP3K4
HOXB13
url https://doi.org/10.1186/s12964-024-02000-2
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