Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer

Abstract High mobility group A1 (HMGA1) is a class of non-histone chromosomal protein and is highly expressed in the embryonic period and many tumors. It is involved in multiple hallmarks of tumors and affects the occurrence and progression of tumors. Nowadays, many non-coding RNAs (ncRNAs), such as...

Full description

Saved in:
Bibliographic Details
Main Authors: Lijie Zhang, Xiaomin Zhao, Xianghong Gao, Hao Qin, Feng Chen, Zhijuan Lin
Format: Article
Language:English
Published: BMC 2025-03-01
Series:Cancer Cell International
Subjects:
Online Access:https://doi.org/10.1186/s12935-025-03713-1
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850072227650732032
author Lijie Zhang
Xiaomin Zhao
Xianghong Gao
Hao Qin
Feng Chen
Zhijuan Lin
author_facet Lijie Zhang
Xiaomin Zhao
Xianghong Gao
Hao Qin
Feng Chen
Zhijuan Lin
author_sort Lijie Zhang
collection DOAJ
description Abstract High mobility group A1 (HMGA1) is a class of non-histone chromosomal protein and is highly expressed in the embryonic period and many tumors. It is involved in multiple hallmarks of tumors and affects the occurrence and progression of tumors. Nowadays, many non-coding RNAs (ncRNAs), such as miRNA, lncRNA, and circRNA, have been found to play a crucial role in HMGA1 regulation. Moreover, some ncRNAs are reported to be the downstream effectors of HMGA1. They interact with each other to affect multiple hallmarks of cancer and targeting ncRNAs or HMGA1 may provide potential strategies for cancer therapy. In this review, we give an overview of recent studies describing the crosstalk between oncogenic HMGA1 and ncRNAs. We also point out the impact of this interaction on the biological behavior of tumors and their potential in tumor therapy in order to offer potential implications and directions for both basic science and clinical applications.
format Article
id doaj-art-23d0fbf782c44cd89fd086dc2deff763
institution DOAJ
issn 1475-2867
language English
publishDate 2025-03-01
publisher BMC
record_format Article
series Cancer Cell International
spelling doaj-art-23d0fbf782c44cd89fd086dc2deff7632025-08-20T02:47:07ZengBMCCancer Cell International1475-28672025-03-0125111310.1186/s12935-025-03713-1Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancerLijie Zhang0Xiaomin Zhao1Xianghong Gao2Hao Qin3Feng Chen4Zhijuan Lin5Key Laboratory for Immunology in Universities of Shandong Province, School of Basic Medicine Sciences, Shandong Second Medical UniversityKey Laboratory for Immunology in Universities of Shandong Province, School of Basic Medicine Sciences, Shandong Second Medical UniversityKey Laboratory for Immunology in Universities of Shandong Province, School of Basic Medicine Sciences, Shandong Second Medical UniversityKey Laboratory for Immunology in Universities of Shandong Province, School of Basic Medicine Sciences, Shandong Second Medical UniversityWeifang Hospital of Traditional Chinese Medicine, Shandong Second Medical UniversityKey Laboratory for Immunology in Universities of Shandong Province, School of Basic Medicine Sciences, Shandong Second Medical UniversityAbstract High mobility group A1 (HMGA1) is a class of non-histone chromosomal protein and is highly expressed in the embryonic period and many tumors. It is involved in multiple hallmarks of tumors and affects the occurrence and progression of tumors. Nowadays, many non-coding RNAs (ncRNAs), such as miRNA, lncRNA, and circRNA, have been found to play a crucial role in HMGA1 regulation. Moreover, some ncRNAs are reported to be the downstream effectors of HMGA1. They interact with each other to affect multiple hallmarks of cancer and targeting ncRNAs or HMGA1 may provide potential strategies for cancer therapy. In this review, we give an overview of recent studies describing the crosstalk between oncogenic HMGA1 and ncRNAs. We also point out the impact of this interaction on the biological behavior of tumors and their potential in tumor therapy in order to offer potential implications and directions for both basic science and clinical applications.https://doi.org/10.1186/s12935-025-03713-1LncRNACircRNAHMGA1TumorigenesisTargeted therapy
spellingShingle Lijie Zhang
Xiaomin Zhao
Xianghong Gao
Hao Qin
Feng Chen
Zhijuan Lin
Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
Cancer Cell International
LncRNA
CircRNA
HMGA1
Tumorigenesis
Targeted therapy
title Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
title_full Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
title_fullStr Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
title_full_unstemmed Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
title_short Complicated crosstalk between HMGA and non-coding RNAs modulates hallmarks of cancer
title_sort complicated crosstalk between hmga and non coding rnas modulates hallmarks of cancer
topic LncRNA
CircRNA
HMGA1
Tumorigenesis
Targeted therapy
url https://doi.org/10.1186/s12935-025-03713-1
work_keys_str_mv AT lijiezhang complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer
AT xiaominzhao complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer
AT xianghonggao complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer
AT haoqin complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer
AT fengchen complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer
AT zhijuanlin complicatedcrosstalkbetweenhmgaandnoncodingrnasmodulateshallmarksofcancer