Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma

The complexity of the pathogenetic mechanisms of the development of chronic inflammation in asthma determines its heterogeneity and insufficient treatment effectiveness. Nuclear transcription factors, which include peroxisome proliferator-activated receptors, that is, PPARs, play an important role i...

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Main Authors: Oxana Yu. Kytikova, Juliy M. Perelman, Tatyana P. Novgorodtseva, Yulia K. Denisenko, Viktor P. Kolosov, Marina V. Antonyuk, Tatyana A. Gvozdenko
Format: Article
Language:English
Published: Wiley 2020-01-01
Series:PPAR Research
Online Access:http://dx.doi.org/10.1155/2020/8906968
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author Oxana Yu. Kytikova
Juliy M. Perelman
Tatyana P. Novgorodtseva
Yulia K. Denisenko
Viktor P. Kolosov
Marina V. Antonyuk
Tatyana A. Gvozdenko
author_facet Oxana Yu. Kytikova
Juliy M. Perelman
Tatyana P. Novgorodtseva
Yulia K. Denisenko
Viktor P. Kolosov
Marina V. Antonyuk
Tatyana A. Gvozdenko
author_sort Oxana Yu. Kytikova
collection DOAJ
description The complexity of the pathogenetic mechanisms of the development of chronic inflammation in asthma determines its heterogeneity and insufficient treatment effectiveness. Nuclear transcription factors, which include peroxisome proliferator-activated receptors, that is, PPARs, play an important role in the regulation of initiation and resolution of the inflammatory process. The ability of PPARs to modulate not only lipid homeostasis but also the activity of the inflammatory response makes them an important pathogenetic target in asthma therapy. At present, special attention is focused on natural (polyunsaturated fatty acids (PUFAs), endocannabinoids, and eicosanoids) and synthetic (fibrates, thiazolidinediones) PPAR ligands and the study of signaling mechanisms involved in the implementation of their anti-inflammatory effects in asthma. This review summarizes current views on the structure and function of PPARs, as well as their participation in the pathogenesis of chronic inflammation in asthma. The potential use of PPAR ligands as therapeutic agents for treating asthma is under discussion.
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institution Kabale University
issn 1687-4757
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language English
publishDate 2020-01-01
publisher Wiley
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series PPAR Research
spelling doaj-art-22b043aa86794485bc0f70ce593c46b02025-08-20T03:54:48ZengWileyPPAR Research1687-47571687-47652020-01-01202010.1155/2020/89069688906968Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in AsthmaOxana Yu. Kytikova0Juliy M. Perelman1Tatyana P. Novgorodtseva2Yulia K. Denisenko3Viktor P. Kolosov4Marina V. Antonyuk5Tatyana A. Gvozdenko6Vladivostok Branch of Far Eastern Scientific Centre of Physiology and Pathology of Respiration, Institute of Medical Climatology and Rehabilitative Treatment, Vladivostok, RussiaFar Eastern Scientific Center of Physiology and Pathology of Respiration, Russian Academy of Sciences, Blagoveshchensk, RussiaVladivostok Branch of Far Eastern Scientific Centre of Physiology and Pathology of Respiration, Institute of Medical Climatology and Rehabilitative Treatment, Vladivostok, RussiaVladivostok Branch of Far Eastern Scientific Centre of Physiology and Pathology of Respiration, Institute of Medical Climatology and Rehabilitative Treatment, Vladivostok, RussiaFar Eastern Scientific Center of Physiology and Pathology of Respiration, Russian Academy of Sciences, Blagoveshchensk, RussiaVladivostok Branch of Far Eastern Scientific Centre of Physiology and Pathology of Respiration, Institute of Medical Climatology and Rehabilitative Treatment, Vladivostok, RussiaVladivostok Branch of Far Eastern Scientific Centre of Physiology and Pathology of Respiration, Institute of Medical Climatology and Rehabilitative Treatment, Vladivostok, RussiaThe complexity of the pathogenetic mechanisms of the development of chronic inflammation in asthma determines its heterogeneity and insufficient treatment effectiveness. Nuclear transcription factors, which include peroxisome proliferator-activated receptors, that is, PPARs, play an important role in the regulation of initiation and resolution of the inflammatory process. The ability of PPARs to modulate not only lipid homeostasis but also the activity of the inflammatory response makes them an important pathogenetic target in asthma therapy. At present, special attention is focused on natural (polyunsaturated fatty acids (PUFAs), endocannabinoids, and eicosanoids) and synthetic (fibrates, thiazolidinediones) PPAR ligands and the study of signaling mechanisms involved in the implementation of their anti-inflammatory effects in asthma. This review summarizes current views on the structure and function of PPARs, as well as their participation in the pathogenesis of chronic inflammation in asthma. The potential use of PPAR ligands as therapeutic agents for treating asthma is under discussion.http://dx.doi.org/10.1155/2020/8906968
spellingShingle Oxana Yu. Kytikova
Juliy M. Perelman
Tatyana P. Novgorodtseva
Yulia K. Denisenko
Viktor P. Kolosov
Marina V. Antonyuk
Tatyana A. Gvozdenko
Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
PPAR Research
title Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
title_full Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
title_fullStr Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
title_full_unstemmed Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
title_short Peroxisome Proliferator-Activated Receptors as a Therapeutic Target in Asthma
title_sort peroxisome proliferator activated receptors as a therapeutic target in asthma
url http://dx.doi.org/10.1155/2020/8906968
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AT yuliakdenisenko peroxisomeproliferatoractivatedreceptorsasatherapeutictargetinasthma
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