A microRNA signature that correlates with cognition and is a target against cognitive decline

Abstract While some individuals age without pathological memory impairments, others develop age‐associated cognitive diseases. Since changes in cognitive function develop slowly over time in these patients, they are often diagnosed at an advanced stage of molecular pathology, a time point when causa...

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Main Authors: Md Rezaul Islam, Lalit Kaurani, Tea Berulava, Urs Heilbronner, Monika Budde, Tonatiuh Pena Centeno, Vakthang Elerdashvili, Maria‐Patapia Zafieriou, Eva Benito, Sinem M Sertel, Maria Goldberg, Fanny Senner, Janos L Kalman, Susanne Burkhardt, Anne Sophie Oepen, Mohammad Sadman Sakib, Cemil Kerimoglu, Oliver Wirths, Heike Bickeböller, Claudia Bartels, Frederic Brosseron, Katharina Buerger, Nicoleta‐Carmen Cosma, Klaus Fliessbach, Michael T. Heneka, Daniel Janowitz, Ingo Kilimann, Luca Kleinedam, Christoph Laske, Coraline D Metzger, Matthias H Munk, Robert Perneczky, Oliver Peters, Josef Priller, Boris S. Rauchmann, Nina Roy, Anja Schneider, Annika Spottke, Eike J Spruth, Stefan Teipel, Maike Tscheuschler, Michael Wagner, Jens Wiltfang, Emrah Düzel, Frank Jessen, Delcode Study Group, Silvio O Rizzoli, Wolfram‐Hubertus Zimmermann, Thomas G Schulze, Peter Falkai, Farahnaz Sananbenesi, Andre Fischer
Format: Article
Language:English
Published: Springer Nature 2021-10-01
Series:EMBO Molecular Medicine
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Online Access:https://doi.org/10.15252/emmm.202013659
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author Md Rezaul Islam
Lalit Kaurani
Tea Berulava
Urs Heilbronner
Monika Budde
Tonatiuh Pena Centeno
Vakthang Elerdashvili
Maria‐Patapia Zafieriou
Eva Benito
Sinem M Sertel
Maria Goldberg
Fanny Senner
Janos L Kalman
Susanne Burkhardt
Anne Sophie Oepen
Mohammad Sadman Sakib
Cemil Kerimoglu
Oliver Wirths
Heike Bickeböller
Claudia Bartels
Frederic Brosseron
Katharina Buerger
Nicoleta‐Carmen Cosma
Klaus Fliessbach
Michael T. Heneka
Daniel Janowitz
Ingo Kilimann
Luca Kleinedam
Christoph Laske
Coraline D Metzger
Matthias H Munk
Robert Perneczky
Oliver Peters
Josef Priller
Boris S. Rauchmann
Nina Roy
Anja Schneider
Annika Spottke
Eike J Spruth
Stefan Teipel
Maike Tscheuschler
Michael Wagner
Jens Wiltfang
Emrah Düzel
Frank Jessen
Delcode Study Group
Silvio O Rizzoli
Wolfram‐Hubertus Zimmermann
Thomas G Schulze
Peter Falkai
Farahnaz Sananbenesi
Andre Fischer
author_facet Md Rezaul Islam
Lalit Kaurani
Tea Berulava
Urs Heilbronner
Monika Budde
Tonatiuh Pena Centeno
Vakthang Elerdashvili
Maria‐Patapia Zafieriou
Eva Benito
Sinem M Sertel
Maria Goldberg
Fanny Senner
Janos L Kalman
Susanne Burkhardt
Anne Sophie Oepen
Mohammad Sadman Sakib
Cemil Kerimoglu
Oliver Wirths
Heike Bickeböller
Claudia Bartels
Frederic Brosseron
Katharina Buerger
Nicoleta‐Carmen Cosma
Klaus Fliessbach
Michael T. Heneka
Daniel Janowitz
Ingo Kilimann
Luca Kleinedam
Christoph Laske
Coraline D Metzger
Matthias H Munk
Robert Perneczky
Oliver Peters
Josef Priller
Boris S. Rauchmann
Nina Roy
Anja Schneider
Annika Spottke
Eike J Spruth
Stefan Teipel
Maike Tscheuschler
Michael Wagner
Jens Wiltfang
Emrah Düzel
Frank Jessen
Delcode Study Group
Silvio O Rizzoli
Wolfram‐Hubertus Zimmermann
Thomas G Schulze
Peter Falkai
Farahnaz Sananbenesi
Andre Fischer
author_sort Md Rezaul Islam
collection DOAJ
description Abstract While some individuals age without pathological memory impairments, others develop age‐associated cognitive diseases. Since changes in cognitive function develop slowly over time in these patients, they are often diagnosed at an advanced stage of molecular pathology, a time point when causative treatments fail. Thus, there is great need for the identification of inexpensive and minimal invasive approaches that could be used for screening with the aim to identify individuals at risk for cognitive decline that can then undergo further diagnostics and eventually stratified therapies. In this study, we use an integrative approach combining the analysis of human data and mechanistic studies in model systems to identify a circulating 3‐microRNA signature that reflects key processes linked to neural homeostasis and inform about cognitive status. We furthermore provide evidence that expression changes in this signature represent multiple mechanisms deregulated in the aging and diseased brain and are a suitable target for RNA therapeutics.
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spelling doaj-art-1e2ab5f4a61e40db8cbade9a85f5dc942025-08-20T03:06:00ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842021-10-01131112810.15252/emmm.202013659A microRNA signature that correlates with cognition and is a target against cognitive declineMd Rezaul Islam0Lalit Kaurani1Tea Berulava2Urs Heilbronner3Monika Budde4Tonatiuh Pena Centeno5Vakthang Elerdashvili6Maria‐Patapia Zafieriou7Eva Benito8Sinem M Sertel9Maria Goldberg10Fanny Senner11Janos L Kalman12Susanne Burkhardt13Anne Sophie Oepen14Mohammad Sadman Sakib15Cemil Kerimoglu16Oliver Wirths17Heike Bickeböller18Claudia Bartels19Frederic Brosseron20Katharina Buerger21Nicoleta‐Carmen Cosma22Klaus Fliessbach23Michael T. Heneka24Daniel Janowitz25Ingo Kilimann26Luca Kleinedam27Christoph Laske28Coraline D Metzger29Matthias H Munk30Robert Perneczky31Oliver Peters32Josef Priller33Boris S. Rauchmann34Nina Roy35Anja Schneider36Annika Spottke37Eike J Spruth38Stefan Teipel39Maike Tscheuschler40Michael Wagner41Jens Wiltfang42Emrah Düzel43Frank Jessen44Delcode Study GroupSilvio O Rizzoli45Wolfram‐Hubertus Zimmermann46Thomas G Schulze47Peter Falkai48Farahnaz Sananbenesi49Andre Fischer50Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesInstitute of Psychiatric Phenomics and Genomics (IPPG), University Hospital, Ludwig‐Maximilians‐University MunichInstitute of Psychiatric Phenomics and Genomics (IPPG), University Hospital, Ludwig‐Maximilians‐University MunichDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesInstitute of Pharmacology and Toxicology, University Medical Center GöttingenDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment of Neuro‐ and Sensory Physiology, University Medical Center GöttingenDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesInstitute of Psychiatric Phenomics and Genomics (IPPG), University Hospital, Ludwig‐Maximilians‐University MunichInstitute of Psychiatric Phenomics and Genomics (IPPG), University Hospital, Ludwig‐Maximilians‐University MunichDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesDepartment for Psychiatry and Psychotherapy, University Medical Center GöttingenDepartment of Genetic Epidemiology, University Medical Center GöttingenDepartment for Psychiatry and Psychotherapy, University Medical Center GöttingenGerman Center for Neurodegenerative DiseasesGerman Center for Neurodegenerative Diseases (DZNE, Munich)Department of Psychiatry and Psychotherapy, Charité – Universitätsmedizin BerlinGerman Center for Neurodegenerative DiseasesGerman Center for Neurodegenerative DiseasesInstitute for Stroke and Dementia Research (ISD), University Hospital, LMU MunichGerman Center for Neurodegenerative Diseases (DZNE)German Center for Neurodegenerative DiseasesDepartment of Psychosomatic Medicine, Rostock University Medical CenterGerman Center for Neurodegenerative Diseases (DZNE)Section for Dementia Research, Hertie Institute for Clinical Brain Research and Department of Psychiatry and Psychotherapy, University of TübingenDepartment of Psychiatry and Psychotherapy, Ludwig‐Maximilians‐University MunichDepartment of Psychiatry and Psychotherapy, Charité – Universitätsmedizin BerlinGerman Center for Neurodegenerative Diseases (DZNE)Department of Psychiatry and Psychotherapy, Ludwig‐Maximilians‐University MunichGerman Center for Neurodegenerative DiseasesGerman Center for Neurodegenerative DiseasesGerman Center for Neurodegenerative DiseasesGerman Center for Neurodegenerative Diseases (DZNE)German Center for Neurodegenerative Diseases (DZNE)Department of Psychiatry, Medical Faculty, University of CologneGerman Center for Neurodegenerative DiseasesDepartment for Psychiatry and Psychotherapy, University Medical Center GöttingenGerman Center for Neurodegenerative Diseases (DZNE)German Center for Neurodegenerative DiseasesDepartment of Neuro‐ and Sensory Physiology, University Medical Center GöttingenInstitute of Pharmacology and Toxicology, University Medical Center GöttingenDepartment for Psychiatry and Psychotherapy, University Medical Center GöttingenDepartment of Psychiatry and Psychotherapy, Ludwig‐Maximilians‐University MunichDepartment for Psychiatry and Psychotherapy, University Medical Center GöttingenDepartment for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative DiseasesAbstract While some individuals age without pathological memory impairments, others develop age‐associated cognitive diseases. Since changes in cognitive function develop slowly over time in these patients, they are often diagnosed at an advanced stage of molecular pathology, a time point when causative treatments fail. Thus, there is great need for the identification of inexpensive and minimal invasive approaches that could be used for screening with the aim to identify individuals at risk for cognitive decline that can then undergo further diagnostics and eventually stratified therapies. In this study, we use an integrative approach combining the analysis of human data and mechanistic studies in model systems to identify a circulating 3‐microRNA signature that reflects key processes linked to neural homeostasis and inform about cognitive status. We furthermore provide evidence that expression changes in this signature represent multiple mechanisms deregulated in the aging and diseased brain and are a suitable target for RNA therapeutics.https://doi.org/10.15252/emmm.202013659Alzheimerbiomarkercognitive impairmentmicroRNARNA therapeutics
spellingShingle Md Rezaul Islam
Lalit Kaurani
Tea Berulava
Urs Heilbronner
Monika Budde
Tonatiuh Pena Centeno
Vakthang Elerdashvili
Maria‐Patapia Zafieriou
Eva Benito
Sinem M Sertel
Maria Goldberg
Fanny Senner
Janos L Kalman
Susanne Burkhardt
Anne Sophie Oepen
Mohammad Sadman Sakib
Cemil Kerimoglu
Oliver Wirths
Heike Bickeböller
Claudia Bartels
Frederic Brosseron
Katharina Buerger
Nicoleta‐Carmen Cosma
Klaus Fliessbach
Michael T. Heneka
Daniel Janowitz
Ingo Kilimann
Luca Kleinedam
Christoph Laske
Coraline D Metzger
Matthias H Munk
Robert Perneczky
Oliver Peters
Josef Priller
Boris S. Rauchmann
Nina Roy
Anja Schneider
Annika Spottke
Eike J Spruth
Stefan Teipel
Maike Tscheuschler
Michael Wagner
Jens Wiltfang
Emrah Düzel
Frank Jessen
Delcode Study Group
Silvio O Rizzoli
Wolfram‐Hubertus Zimmermann
Thomas G Schulze
Peter Falkai
Farahnaz Sananbenesi
Andre Fischer
A microRNA signature that correlates with cognition and is a target against cognitive decline
EMBO Molecular Medicine
Alzheimer
biomarker
cognitive impairment
microRNA
RNA therapeutics
title A microRNA signature that correlates with cognition and is a target against cognitive decline
title_full A microRNA signature that correlates with cognition and is a target against cognitive decline
title_fullStr A microRNA signature that correlates with cognition and is a target against cognitive decline
title_full_unstemmed A microRNA signature that correlates with cognition and is a target against cognitive decline
title_short A microRNA signature that correlates with cognition and is a target against cognitive decline
title_sort microrna signature that correlates with cognition and is a target against cognitive decline
topic Alzheimer
biomarker
cognitive impairment
microRNA
RNA therapeutics
url https://doi.org/10.15252/emmm.202013659
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