Histone Lysine Methylation in Diabetic Nephropathy

Diabetic nephropathy (DN) belongs to debilitating microvascular complications of diabetes and is the leading cause of end-stage renal diseases worldwide. Furthermore, outcomes from the DCCT/EDIC study showed that DN often persists and progresses despite intensive glucose control in many diabetes pat...

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Main Authors: Guang-dong Sun, Wen-peng Cui, Qiao-yan Guo, Li-ning Miao
Format: Article
Language:English
Published: Wiley 2014-01-01
Series:Journal of Diabetes Research
Online Access:http://dx.doi.org/10.1155/2014/654148
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author Guang-dong Sun
Wen-peng Cui
Qiao-yan Guo
Li-ning Miao
author_facet Guang-dong Sun
Wen-peng Cui
Qiao-yan Guo
Li-ning Miao
author_sort Guang-dong Sun
collection DOAJ
description Diabetic nephropathy (DN) belongs to debilitating microvascular complications of diabetes and is the leading cause of end-stage renal diseases worldwide. Furthermore, outcomes from the DCCT/EDIC study showed that DN often persists and progresses despite intensive glucose control in many diabetes patients, possibly as a result of prior episode of hyperglycemia, which is called “metabolic memory.” The underlying mechanisms responsible for the development and progression of DN remain poorly understood. Activation of multiple signaling pathways and key transcription factors can lead to aberrant expression of DN-related pathologic genes in target renal cells. Increasing evidence suggests that epigenetic mechanisms in chromatin such as DNA methylation, histone acetylation, and methylation can influence the pathophysiology of DN and metabolic memory. Exciting researches from cell culture and experimental animals have shown that key histone methylation patterns and the related histone methyltransferases and histone demethylases can play important roles in the regulation of inflammatory and profibrotic genes in renal cells under diabetic conditions. Because histone methylation is dynamic and potentially reversible, it can provide a window of opportunity for the development of much-needed novel therapeutic potential for DN in the future. In this minireview, we discuss recent advances in the field of histone methylation and its roles in the pathogenesis and progression of DN.
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spelling doaj-art-1decba9bb55545a9a850ff91d0873d892025-08-20T02:01:40ZengWileyJournal of Diabetes Research2314-67452314-67532014-01-01201410.1155/2014/654148654148Histone Lysine Methylation in Diabetic NephropathyGuang-dong Sun0Wen-peng Cui1Qiao-yan Guo2Li-ning Miao3Department of Nephrology, Second Hospital of Jilin University, Changchun 130041, ChinaDepartment of Nephrology, Second Hospital of Jilin University, Changchun 130041, ChinaDepartment of Nephrology, Second Hospital of Jilin University, Changchun 130041, ChinaDepartment of Nephrology, Second Hospital of Jilin University, Changchun 130041, ChinaDiabetic nephropathy (DN) belongs to debilitating microvascular complications of diabetes and is the leading cause of end-stage renal diseases worldwide. Furthermore, outcomes from the DCCT/EDIC study showed that DN often persists and progresses despite intensive glucose control in many diabetes patients, possibly as a result of prior episode of hyperglycemia, which is called “metabolic memory.” The underlying mechanisms responsible for the development and progression of DN remain poorly understood. Activation of multiple signaling pathways and key transcription factors can lead to aberrant expression of DN-related pathologic genes in target renal cells. Increasing evidence suggests that epigenetic mechanisms in chromatin such as DNA methylation, histone acetylation, and methylation can influence the pathophysiology of DN and metabolic memory. Exciting researches from cell culture and experimental animals have shown that key histone methylation patterns and the related histone methyltransferases and histone demethylases can play important roles in the regulation of inflammatory and profibrotic genes in renal cells under diabetic conditions. Because histone methylation is dynamic and potentially reversible, it can provide a window of opportunity for the development of much-needed novel therapeutic potential for DN in the future. In this minireview, we discuss recent advances in the field of histone methylation and its roles in the pathogenesis and progression of DN.http://dx.doi.org/10.1155/2014/654148
spellingShingle Guang-dong Sun
Wen-peng Cui
Qiao-yan Guo
Li-ning Miao
Histone Lysine Methylation in Diabetic Nephropathy
Journal of Diabetes Research
title Histone Lysine Methylation in Diabetic Nephropathy
title_full Histone Lysine Methylation in Diabetic Nephropathy
title_fullStr Histone Lysine Methylation in Diabetic Nephropathy
title_full_unstemmed Histone Lysine Methylation in Diabetic Nephropathy
title_short Histone Lysine Methylation in Diabetic Nephropathy
title_sort histone lysine methylation in diabetic nephropathy
url http://dx.doi.org/10.1155/2014/654148
work_keys_str_mv AT guangdongsun histonelysinemethylationindiabeticnephropathy
AT wenpengcui histonelysinemethylationindiabeticnephropathy
AT qiaoyanguo histonelysinemethylationindiabeticnephropathy
AT liningmiao histonelysinemethylationindiabeticnephropathy