HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts

During the 2021/2022 winter season, we isolated highly pathogenic avian influenza (HPAI) H5N1 viruses harbouring an amino acid substitution from Asparagine(N) to Aspartic acid (D) at residue 193 of the hemagglutinin (HA) receptor binding domain (RBD) from migratory birds in South Korea. Herein, we i...

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Main Authors: Seung-Gyu Jang, Young-Il Kim, Mark Anthony B. Casel, Jeong Ho Choi, Ju Ryeon Gil, Rare Rollon, Eun-Ha Kim, Se-Mi Kim, Ho Young Ji, Dong Bin Park, Jungwon Hwang, Jae-Woo Ahn, Myung Hee Kim, Min-Suk Song, Young Ki Choi
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Emerging Microbes and Infections
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Online Access:https://www.tandfonline.com/doi/10.1080/22221751.2024.2302854
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author Seung-Gyu Jang
Young-Il Kim
Mark Anthony B. Casel
Jeong Ho Choi
Ju Ryeon Gil
Rare Rollon
Eun-Ha Kim
Se-Mi Kim
Ho Young Ji
Dong Bin Park
Jungwon Hwang
Jae-Woo Ahn
Myung Hee Kim
Min-Suk Song
Young Ki Choi
author_facet Seung-Gyu Jang
Young-Il Kim
Mark Anthony B. Casel
Jeong Ho Choi
Ju Ryeon Gil
Rare Rollon
Eun-Ha Kim
Se-Mi Kim
Ho Young Ji
Dong Bin Park
Jungwon Hwang
Jae-Woo Ahn
Myung Hee Kim
Min-Suk Song
Young Ki Choi
author_sort Seung-Gyu Jang
collection DOAJ
description During the 2021/2022 winter season, we isolated highly pathogenic avian influenza (HPAI) H5N1 viruses harbouring an amino acid substitution from Asparagine(N) to Aspartic acid (D) at residue 193 of the hemagglutinin (HA) receptor binding domain (RBD) from migratory birds in South Korea. Herein, we investigated the characteristics of the N193D HA-RBD substitution in the A/CommonTeal/Korea/W811/2021[CT/W811] virus by using recombinant viruses engineered via reverse genetics (RG). A receptor affinity assay revealed that the N193D HA-RBD substitution in CT/W811 increases α2,6 sialic acid receptor binding affinity. The rCT/W811-HA193N virus caused rapid lethality with high virus titres in chickens compared with the rCT/W811-HA193D virus, while the rCT/W811-HA193D virus exhibited enhanced virulence in mammalian hosts with multiple tissue tropism. Surprisingly, a ferret-to-ferret transmission assay revealed that rCT/W811-HA193D virus replicates well in the respiratory tract, at a rate about 10 times higher than that of rCT/W811-HA193N, and all rCT/W811-HA193D direct contact ferrets were seroconverted at 10 days post-contact. Further, competition transmission assay of the two viruses revealed that rCT/W811-HA193D has enhanced growth kinetics compared with the rCT/W811-HA193N, eventually becoming the dominant strain in nasal turbinates. Further, rCT/W811-HA193D exhibits high infectivity in primary human bronchial epithelial (HBE) cells, suggesting the potential for human infection. Taken together, the HA-193D containing HPAI H5N1 virus from migratory birds showed enhanced virulence in mammalian hosts, but not in avian hosts, with multi-organ replication and ferret-to-ferret transmission. Thus, this suggests that HA-193D change increases the probability of HPAI H5N1 infection and transmission in humans.
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spelling doaj-art-1d8d058e46454b5eb6f0a2ca051f401a2025-08-20T02:37:32ZengTaylor & Francis GroupEmerging Microbes and Infections2222-17512024-12-0113110.1080/22221751.2024.2302854HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hostsSeung-Gyu Jang0Young-Il Kim1Mark Anthony B. Casel2Jeong Ho Choi3Ju Ryeon Gil4Rare Rollon5Eun-Ha Kim6Se-Mi Kim7Ho Young Ji8Dong Bin Park9Jungwon Hwang10Jae-Woo Ahn11Myung Hee Kim12Min-Suk Song13Young Ki Choi14College of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaMicrobiome Convergence Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Daejeon, Republic of KoreaCenter for Study of Emerging and Re-emerging Viruses, Korea Virus Research Institute, Institute for Basic Science (IBS), Daejeon, Republic of KoreaMicrobiome Convergence Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Daejeon, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaCollege of Medicine and Medical Research Institute, Chungbuk National University, Cheongju, Republic of KoreaDuring the 2021/2022 winter season, we isolated highly pathogenic avian influenza (HPAI) H5N1 viruses harbouring an amino acid substitution from Asparagine(N) to Aspartic acid (D) at residue 193 of the hemagglutinin (HA) receptor binding domain (RBD) from migratory birds in South Korea. Herein, we investigated the characteristics of the N193D HA-RBD substitution in the A/CommonTeal/Korea/W811/2021[CT/W811] virus by using recombinant viruses engineered via reverse genetics (RG). A receptor affinity assay revealed that the N193D HA-RBD substitution in CT/W811 increases α2,6 sialic acid receptor binding affinity. The rCT/W811-HA193N virus caused rapid lethality with high virus titres in chickens compared with the rCT/W811-HA193D virus, while the rCT/W811-HA193D virus exhibited enhanced virulence in mammalian hosts with multiple tissue tropism. Surprisingly, a ferret-to-ferret transmission assay revealed that rCT/W811-HA193D virus replicates well in the respiratory tract, at a rate about 10 times higher than that of rCT/W811-HA193N, and all rCT/W811-HA193D direct contact ferrets were seroconverted at 10 days post-contact. Further, competition transmission assay of the two viruses revealed that rCT/W811-HA193D has enhanced growth kinetics compared with the rCT/W811-HA193N, eventually becoming the dominant strain in nasal turbinates. Further, rCT/W811-HA193D exhibits high infectivity in primary human bronchial epithelial (HBE) cells, suggesting the potential for human infection. Taken together, the HA-193D containing HPAI H5N1 virus from migratory birds showed enhanced virulence in mammalian hosts, but not in avian hosts, with multi-organ replication and ferret-to-ferret transmission. Thus, this suggests that HA-193D change increases the probability of HPAI H5N1 infection and transmission in humans.https://www.tandfonline.com/doi/10.1080/22221751.2024.2302854HPAI H5N8 virusClade 2.3.4.4reassortantpathogenecitySouth KoreaInfluenza infections
spellingShingle Seung-Gyu Jang
Young-Il Kim
Mark Anthony B. Casel
Jeong Ho Choi
Ju Ryeon Gil
Rare Rollon
Eun-Ha Kim
Se-Mi Kim
Ho Young Ji
Dong Bin Park
Jungwon Hwang
Jae-Woo Ahn
Myung Hee Kim
Min-Suk Song
Young Ki Choi
HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
Emerging Microbes and Infections
HPAI H5N8 virus
Clade 2.3.4.4
reassortant
pathogenecity
South Korea
Influenza infections
title HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
title_full HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
title_fullStr HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
title_full_unstemmed HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
title_short HA N193D substitution in the HPAI H5N1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
title_sort ha n193d substitution in the hpai h5n1 virus alters receptor binding affinity and enhances virulence in mammalian hosts
topic HPAI H5N8 virus
Clade 2.3.4.4
reassortant
pathogenecity
South Korea
Influenza infections
url https://www.tandfonline.com/doi/10.1080/22221751.2024.2302854
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