Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent

Background. Endothelial dysfunction occurs in obese children and adolescent and is regarded as a key step in the development of atherosclerosis. Important components for the development of endothelial dysfunction are reduced activity of endothelial nitric oxide synthase (eNOS) and an increase in cho...

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Main Authors: Jenny Wesnigk, Luc Bruyndonckx, Vicky Y. Hoymans, Ann De Guchtenaere, Tina Fischer, Gerhard Schuler, Christiaan J. Vrints, Volker Adams
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Cardiology Research and Practice
Online Access:http://dx.doi.org/10.1155/2016/2820432
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author Jenny Wesnigk
Luc Bruyndonckx
Vicky Y. Hoymans
Ann De Guchtenaere
Tina Fischer
Gerhard Schuler
Christiaan J. Vrints
Volker Adams
author_facet Jenny Wesnigk
Luc Bruyndonckx
Vicky Y. Hoymans
Ann De Guchtenaere
Tina Fischer
Gerhard Schuler
Christiaan J. Vrints
Volker Adams
author_sort Jenny Wesnigk
collection DOAJ
description Background. Endothelial dysfunction occurs in obese children and adolescent and is regarded as a key step in the development of atherosclerosis. Important components for the development of endothelial dysfunction are reduced activity of endothelial nitric oxide synthase (eNOS) and an increase in cholesterol deposition in the vessel wall, due to reduced reverse cholesterol transport (RCT) activity. High density lipoprotein (HDL) exhibits antiatherosclerotic properties including modulation of eNOS activity and cholesterol efflux capacity. Lifestyle intervention programs can modify endothelial dysfunction in obese adolescents, but their impact on HDL-mediated eNOS activation and RCT is unknown so far. Methods. Obese adolescents (15±1 years, BMI > 35 kg/m2) where randomized either to an intervention group (IG, n=8; restricted diet and exercise) or to a usual care group (UC, n=8). At the beginning and after 10 months of treatment HDL-mediated eNOS phosphorylation and cholesterol efflux capacity were evaluated. Results. Ten months of treatment resulted in a substantial weight loss (−31%), an improvement of endothelial function, and an increase in HDL-mediated eNOS-Ser1177 phosphorylation and RCT. A correlation between change in eNOS-Ser1177 phosphorylation or RCT and change in endothelial function was noted. Conclusion. A structured lifestyle intervention program improves antiatherosclerotic HDL functions, thereby positively influencing endothelial function.
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spelling doaj-art-1d55edec7db443ecae276eae145ae0c92025-08-20T02:01:39ZengWileyCardiology Research and Practice2090-80162090-05972016-01-01201610.1155/2016/28204322820432Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese AdolescentJenny Wesnigk0Luc Bruyndonckx1Vicky Y. Hoymans2Ann De Guchtenaere3Tina Fischer4Gerhard Schuler5Christiaan J. Vrints6Volker Adams7University of Leipzig, Heart Center Leipzig, Leipzig, GermanyAntwerp University Hospital, Laboratory of Cellular and Molecular Cardiology, Edegem, BelgiumAntwerp University Hospital, Laboratory of Cellular and Molecular Cardiology, Edegem, BelgiumZeepreventorium, De Haan, BelgiumUniversity of Leipzig, Heart Center Leipzig, Leipzig, GermanyUniversity of Leipzig, Heart Center Leipzig, Leipzig, GermanyAntwerp University Hospital, Laboratory of Cellular and Molecular Cardiology, Edegem, BelgiumUniversity of Leipzig, Heart Center Leipzig, Leipzig, GermanyBackground. Endothelial dysfunction occurs in obese children and adolescent and is regarded as a key step in the development of atherosclerosis. Important components for the development of endothelial dysfunction are reduced activity of endothelial nitric oxide synthase (eNOS) and an increase in cholesterol deposition in the vessel wall, due to reduced reverse cholesterol transport (RCT) activity. High density lipoprotein (HDL) exhibits antiatherosclerotic properties including modulation of eNOS activity and cholesterol efflux capacity. Lifestyle intervention programs can modify endothelial dysfunction in obese adolescents, but their impact on HDL-mediated eNOS activation and RCT is unknown so far. Methods. Obese adolescents (15±1 years, BMI > 35 kg/m2) where randomized either to an intervention group (IG, n=8; restricted diet and exercise) or to a usual care group (UC, n=8). At the beginning and after 10 months of treatment HDL-mediated eNOS phosphorylation and cholesterol efflux capacity were evaluated. Results. Ten months of treatment resulted in a substantial weight loss (−31%), an improvement of endothelial function, and an increase in HDL-mediated eNOS-Ser1177 phosphorylation and RCT. A correlation between change in eNOS-Ser1177 phosphorylation or RCT and change in endothelial function was noted. Conclusion. A structured lifestyle intervention program improves antiatherosclerotic HDL functions, thereby positively influencing endothelial function.http://dx.doi.org/10.1155/2016/2820432
spellingShingle Jenny Wesnigk
Luc Bruyndonckx
Vicky Y. Hoymans
Ann De Guchtenaere
Tina Fischer
Gerhard Schuler
Christiaan J. Vrints
Volker Adams
Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
Cardiology Research and Practice
title Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
title_full Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
title_fullStr Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
title_full_unstemmed Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
title_short Impact of Lifestyle Intervention on HDL-Induced eNOS Activation and Cholesterol Efflux Capacity in Obese Adolescent
title_sort impact of lifestyle intervention on hdl induced enos activation and cholesterol efflux capacity in obese adolescent
url http://dx.doi.org/10.1155/2016/2820432
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