KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression

Objective: Periodontal ligament stem cells (PDLSCs) are emerging as a promising source for periodontal regeneration and to manage periodontitis. This study aims to investigate the roles of Krüppel-like factor 2 (KLF2) and nuclear factor erythroid 2-related factor (Nrf2) in mediating osteogenic diffe...

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Main Authors: Jiujiu Chen, Lichao Deng
Format: Article
Language:English
Published: Elsevier 2025-06-01
Series:International Dental Journal
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Online Access:http://www.sciencedirect.com/science/article/pii/S0020653925000607
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author Jiujiu Chen
Lichao Deng
author_facet Jiujiu Chen
Lichao Deng
author_sort Jiujiu Chen
collection DOAJ
description Objective: Periodontal ligament stem cells (PDLSCs) are emerging as a promising source for periodontal regeneration and to manage periodontitis. This study aims to investigate the roles of Krüppel-like factor 2 (KLF2) and nuclear factor erythroid 2-related factor (Nrf2) in mediating osteogenic differentiation of human PDLSCs (hPDLSCs) in the context of lipopolysaccharide (LPS) stimulation. Methods: The osteogenic differentiation potential of hPDLSCs isolated from human premolar root samples were examined by alkaline phosphatase (ALP) staining and ALP activity assay, Alizarin red S staining and quantitative analysis of mineralised matrix. Intracellular reactive oxygen species (ROS) production and glutathione (GSH) concentration were assessed to reflect oxidative stress. Results: KLF2 overexpression influenced Nrf2-regulated transcription, leading to significant increases in GSH concentration, ALP activity, mineralised matrix formation, and RUNX2 expression in LPS-stimulated hPDLSCs, as well as significant reductions in ROS production and cell apoptosis. The subsequent Nrf2 knockdown impaired the protective effect of KLF2 on hPDLSCs against LPS stimulation. Conclusion: The findings of the study demonstrate KLF2 overexpression has the ability to promote the osteogenic differentiation of hPDLSCs by promoting Nrf2 activation, suggesting that KLF2 mediating Nrf2 could be a promising target to facilitate the efficacy of PDLSC-based bone regeneration in periodontitis.
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spelling doaj-art-1bd7a71944274a68872bdfd96ecf49922025-08-20T01:49:08ZengElsevierInternational Dental Journal0020-65392025-06-017531554156310.1016/j.identj.2025.02.019KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 ExpressionJiujiu Chen0Lichao Deng1Pediatric Dentistry, Wenzhou Dental Hospital, Wenzhou, Zhejiang, ChinaEndodontic Department, School & Hospital of Stomatology Wenzhou Medical University, Wenzhou, Zhejiang, China; Corresponding author. Endodontic Department, School & Hospital of Stomatology Wenzhou Medical University, No. 1288 Longyao Avenue, Longwan District, Wenzhou, Zhejiang 325000, China.Objective: Periodontal ligament stem cells (PDLSCs) are emerging as a promising source for periodontal regeneration and to manage periodontitis. This study aims to investigate the roles of Krüppel-like factor 2 (KLF2) and nuclear factor erythroid 2-related factor (Nrf2) in mediating osteogenic differentiation of human PDLSCs (hPDLSCs) in the context of lipopolysaccharide (LPS) stimulation. Methods: The osteogenic differentiation potential of hPDLSCs isolated from human premolar root samples were examined by alkaline phosphatase (ALP) staining and ALP activity assay, Alizarin red S staining and quantitative analysis of mineralised matrix. Intracellular reactive oxygen species (ROS) production and glutathione (GSH) concentration were assessed to reflect oxidative stress. Results: KLF2 overexpression influenced Nrf2-regulated transcription, leading to significant increases in GSH concentration, ALP activity, mineralised matrix formation, and RUNX2 expression in LPS-stimulated hPDLSCs, as well as significant reductions in ROS production and cell apoptosis. The subsequent Nrf2 knockdown impaired the protective effect of KLF2 on hPDLSCs against LPS stimulation. Conclusion: The findings of the study demonstrate KLF2 overexpression has the ability to promote the osteogenic differentiation of hPDLSCs by promoting Nrf2 activation, suggesting that KLF2 mediating Nrf2 could be a promising target to facilitate the efficacy of PDLSC-based bone regeneration in periodontitis.http://www.sciencedirect.com/science/article/pii/S0020653925000607Periodontitistissue engineeringregenerationinflammation
spellingShingle Jiujiu Chen
Lichao Deng
KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
International Dental Journal
Periodontitis
tissue engineering
regeneration
inflammation
title KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
title_full KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
title_fullStr KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
title_full_unstemmed KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
title_short KLF2 Promotes Osteogenic Differentiation of Human Periodontal Ligament Stem Cells by Regulating Nrf2 Expression
title_sort klf2 promotes osteogenic differentiation of human periodontal ligament stem cells by regulating nrf2 expression
topic Periodontitis
tissue engineering
regeneration
inflammation
url http://www.sciencedirect.com/science/article/pii/S0020653925000607
work_keys_str_mv AT jiujiuchen klf2promotesosteogenicdifferentiationofhumanperiodontalligamentstemcellsbyregulatingnrf2expression
AT lichaodeng klf2promotesosteogenicdifferentiationofhumanperiodontalligamentstemcellsbyregulatingnrf2expression